10 results match your criteria reperfusion rvf

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Terminal T-wave inversion predicts reperfusion tachyarrhythmias in STEMI.

J Electrocardiol 2022 Jan 5;71:28-31. Epub 2022 Jan 5.

Department of Cardiology, Clinical Sciences, Lund University, Lund, Sweden; Arrhythmia Clinic, Skåne University Hospital, 22185 Lund, Sweden.

Introduction: A reliable electrocardiographic predictor of ventricular fibrillation (VF) in patients with ST elevation myocardial infarction (STEMI) is lacking so far. Previous experimental/simulation study suggested a terminal T-wave inversion (TTWI) in ischemia-related ECG leads corresponding to anterior infarct localization as an independent predictor of reperfusion VF (rVF). This T-wave characteristic has never been tested as a rVF predictor in clinical settings. Read More

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January 2022

Prolonged T-T interval is associated with ventricular fibrillation during reperfusion in ST-elevation myocardial infarction.

Int J Cardiol 2019 04 4;280:80-83. Epub 2019 Jan 4.

Department of Cardiology, Clinical Sciences, Lund University, SE-22185 Lund, Sweden.

Aim: Ventricular fibrillation (VF) during reperfusion in ST-elevation myocardial infarction (STEMI) is associated with increased in-hospital mortality. Dispersion of ventricular repolarization contributes to ventricular vulnerability during ischemia. T-T interval was proposed as a ventricular repolarization dispersion marker, however its value for prediction of reperfusion VF remains uncertain. Read More

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Predictors of ventricular fibrillation at reperfusion in patients with acute ST-elevation myocardial infarction treated by primary percutaneous coronary intervention.

Am J Cardiol 2015 Feb 29;115(4):417-22. Epub 2014 Nov 29.

Department of Cardiology, Lund University, Lund, Sweden.

Ventricular fibrillation (VF) during reperfusion (rVF) in ST-segment elevation myocardial infarction (STEMI) is an infrequent but serious event that complicates coronary interventions. The aim of this study was to analyze clinical predictors of rVF in an unselected population of patients with STEMI treated with percutaneous coronary intervention (PCI). Consecutive patients with STEMI admitted to a tertiary care hospital for primary PCI from 2007 to 2012 were retrospectively assessed for the presence of rVF. Read More

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February 2015

Primary reperfusion in acute right ventricular infarction: An observational study.

World J Cardiol 2014 Jan;6(1):14-22

Eulo Lupi-Herrera, The American British Cowdray Medical Center I.A.P., Mexico City 14080, Mexico.

Aim: To investigate the impact of primary reperfusion therapy (RT) on early and late mortality in acute right ventricular infarction (RVI).

Methods: RVI patients (n = 679) were prospectively classified as without right ventricular failure (RVF) (class A, n = 425, 64%), with RVF (class B, n = 158, 24%) or with cardiogenic shock (CS) (class C, n = 96, 12%). Of the 679 patients, 148 (21. Read More

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January 2014

Amiodarone for the prevention of reperfusion ventricular fibrillation.

J Cardiothorac Vasc Anesth 2010 Apr 2;24(2):239-43. Epub 2009 Oct 2.

Department of Anesthesiology and Critical Care, Sri Venkateswara Institute of Medical Sciences, Andhra Pradesh, India.

Objective: The purpose of this study was to evaluate the efficacy of prophylactic single-dose amiodarone administered through the pump circuit before releasing the aortic cross-clamp (ACC) in preventing the occurrence of reperfusion ventricular fibrillation (RVF).

Design: A prospective, randomized double-blind, placebo-controlled efficacy study.

Setting: A tertiary level teaching hospital. Read More

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[Pharmacological preconditioning with sildenafil of warm ischemic kidneys].

Actas Urol Esp 2008 Jan;32(1):67-74

Servicios de Urología, Hospital General Universitario Gregorio Marañón, Madrid.

Objectives: To evaluate the preconditioning effect of sildenafil administered preoperatively in kidneys subjected to a period of warm ischemia (WI), hypothermic perfusion (HP) or cold storage (CS) and finally, autotransplant (AT).

Material And Method: We studied 6 groups of autotransplanted kidneys: no-WI-inmediate AT (Group A); 45 min of WI + immediate AT (Group B); 45 min of WI + 60 min of HP + autotransplant (Group C); 45 min of WI + 60 min of CS + autotransplant (Group D); 100 mg of oral sildenafil preoperatively + 45 min of WI + autotransplant (Group E); 100 mg of oral sildenafil preoperatively + 45 min of WI+60 min of HP + autotransplant (Group F). Belzer solution was used for HP; UW-Viaspan for CS. Read More

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January 2008

Does the antiarrhythmic effect of ischemic preconditioning in rats involve the L-arginine nitric oxide pathway?

J Cardiovasc Pharmacol 1995 Apr;25(4):524-30

Department of Cardiovascular and Pulmonary Pharmacology, Janssen Research Foundation, Beerse, Belgium.

Ischemic preconditioning (PC) has been shown to limit ischemia- and reperfusion-induced arrhythmias. We wished to determine whether the antiarrhythmic effect of PC would be affected by inhibition of the L-arginine nitric oxide (NO) pathway in anesthetized rats. Ischemia and reperfusion were produced by occlusion and release of a snare around the left coronary artery in all rats. Read More

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Electrophysiological mechanisms for postcardioplegia reperfusion ventricular fibrillation.

Circulation 1994 Nov;90(5 Pt 2):II293-8

Department of Surgery, University of Alabama at Birmingham 35294.

Background: Reperfusion arrhythmias that follow regional ischemia at normothermia have been studied extensively and are considered to be a manifestation of ischemia-reperfusion injury. In contrast, reperfusion arrhythmias that occur following hypothermic cardioplegic arrest have received little attention from investigators. This study defines the electrophysiological mechanisms for postcardioplegia reperfusion ventricular fibrillation (RVF). Read More

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November 1994

The mechanism of protective effect of diltiazem on reperfusion-induced arrhythmias in isolated rat heart.

Jpn Circ J 1990 Jan;54(1):117-25

Research Institute of Angiocardiology, Faculty of Medicine, Kyushu University, Fukuoka, Japan.

This investigation was undertaken to define the mechanism by which diltiazem protects against life-threatening, reperfusion-induced arrhythmias. Using an isolated retrogressively perfused rat heart preparation with transient coronary artery occlusion, we compared the effects of diltiazem in its active form (d-cis) to its stereo-isomer (1-cis). Pre-ischemic administration of d-diltiazem (5 x 10(-8), 5 x 10(-7), 5 x 10(-6) M) caused a dose-dependent reduction in ventricular arrhythmias upon reperfusion following 10 min of regional ischemia. Read More

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January 1990

Alterations in the distribution of high-energy phosphates during ischemia in a canine model of reperfusion-induced ventricular fibrillation.

Am Heart J 1985 Sep;110(3):590-4

High-energy phosphate metabolites of the canine heart were analyzed before coronary artery occlusion and after 15 minutes of ischemia, and the results were then correlated with the occurrence of ventricular fibrillation upon reperfusion (RVF). Animals which developed VF upon reperfusion after 15 minutes of ischemia had lower levels of creatine phosphate and endocardial adenosine triphosphate (ATP), and increased accumulation of the catabolites of ATP metabolism, inosine and hypoxanthine. Animals which developed RVF also had lower levels of regional myocardial blood flow in the center of the ischemic zone during the period of coronary occlusion. Read More

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September 1985
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