1,774 results match your criteria Nature Reviews Nephrology[Journal]


Measuring fibrosis without biopsies.

Authors:
Monica Wang

Nat Rev Nephrol 2019 Apr 18. Epub 2019 Apr 18.

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0153-4DOI Listing

Immune-based combination therapy for metastatic kidney cancer.

Nat Rev Nephrol 2019 Apr 16. Epub 2019 Apr 16.

Division of Translational Oncology, IRCCS Istituti Clinici Scientifici Maugeri, Pavia, Italy.

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http://dx.doi.org/10.1038/s41581-019-0149-0DOI Listing

Unique metabolic traits identify CCPAP.

Authors:
Monica Wang

Nat Rev Nephrol 2019 Apr 15. Epub 2019 Apr 15.

Nature Reviews Nephrology, .

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http://www.nature.com/articles/s41581-019-0151-6
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http://dx.doi.org/10.1038/s41581-019-0151-6DOI Listing
April 2019
1 Read

Structure and function of polycystins: insights into polycystic kidney disease.

Nat Rev Nephrol 2019 Apr 4. Epub 2019 Apr 4.

Université Côte d'Azur, Centre National de la Recherche Scientifique, Institut National de la Santé et de la Recherche Médicale, Institut de Pharmacologie Moléculaire et Cellulaire, Labex ICST, Valbonne, France.

Mutations in the polycystins PC1 or PC2 cause autosomal dominant polycystic kidney disease (ADPKD), which is characterized by the formation of fluid-filled renal cysts that disrupt renal architecture and function, ultimately leading to kidney failure in the majority of patients. Although the genetic basis of ADPKD is now well established, the physiological function of polycystins remains obscure and a matter of intense debate. The structural determination of both the homomeric PC2 and heteromeric PC1-PC2 complexes, as well as the electrophysiological characterization of PC2 in the primary cilium of renal epithelial cells, provided new valuable insights into the mechanisms of ADPKD pathogenesis. Read More

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http://www.nature.com/articles/s41581-019-0143-6
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http://dx.doi.org/10.1038/s41581-019-0143-6DOI Listing
April 2019
6 Reads

VE-PTP phosphatase: a new target for diabetic kidney disease.

Authors:
Susan J Allison

Nat Rev Nephrol 2019 Apr 1. Epub 2019 Apr 1.

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0147-2DOI Listing

A novel SGLT1-mediated mechanism of hyperglycaemia-induced hyperfiltration.

Authors:
Ellen F Carney

Nat Rev Nephrol 2019 Apr 1. Epub 2019 Apr 1.

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0146-3DOI Listing
April 2019
1 Read

Gut microbial short-chain fatty acids and the risk of diabetes.

Nat Rev Nephrol 2019 Mar 27. Epub 2019 Mar 27.

Division of Nephrology, Department of Medicine, University of California, Irvine, CA, USA.

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http://www.nature.com/articles/s41581-019-0142-7
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http://dx.doi.org/10.1038/s41581-019-0142-7DOI Listing
March 2019
5 Reads

The adjuvant treatment of kidney cancer: a multidisciplinary outlook.

Nat Rev Nephrol 2019 Mar 26. Epub 2019 Mar 26.

Department of Urology, Netherlands Cancer Institute, Amsterdam, Netherlands.

Approximately 70% of cases of kidney cancer are localized or locally advanced at diagnosis. Among patients who undergo surgery for these cancers, 30-35% will eventually develop potentially fatal metachronous distant metastases. Effective adjuvant treatments are urgently needed to reduce the risk of recurrence of kidney cancer and of dying of metastatic disease. Read More

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http://dx.doi.org/10.1038/s41581-019-0131-xDOI Listing
March 2019
2 Reads

Repurposing CAR T cells to treat SLE.

Authors:
Monica Wang

Nat Rev Nephrol 2019 Mar 25. Epub 2019 Mar 25.

Nature Reviews Nephrology, .

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http://www.nature.com/articles/s41581-019-0144-5
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March 2019
5 Reads

Introducing routine genetic testing for patients with CKD.

Nat Rev Nephrol 2019 Mar 22. Epub 2019 Mar 22.

Azrieli Faculty of Medicine, Bar-Ilan University, Safed, Israel.

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http://dx.doi.org/10.1038/s41581-019-0140-9DOI Listing

Epigenetics and epigenomics in diabetic kidney disease and metabolic memory.

Nat Rev Nephrol 2019 Mar 20. Epub 2019 Mar 20.

Department of Diabetes Complications and Metabolism, Diabetes Metabolism Research Institute, Beckman Research Institute of City of Hope, Duarte, CA, USA.

The development and progression of diabetic kidney disease (DKD), a highly prevalent complication of diabetes mellitus, are influenced by both genetic and environmental factors. DKD is an important contributor to the morbidity of patients with diabetes mellitus, indicating a clear need for an improved understanding of disease aetiology to inform the development of more efficacious treatments. DKD is characterized by an accumulation of extracellular matrix, hypertrophy and fibrosis in kidney glomerular and tubular cells. Read More

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http://www.nature.com/articles/s41581-019-0135-6
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March 2019
6 Reads

Not your usual parietal cell.

Authors:
Monica Wang

Nat Rev Nephrol 2019 Mar 18. Epub 2019 Mar 18.

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0141-8DOI Listing

Glycosylation in health and disease.

Nat Rev Nephrol 2019 Mar 11. Epub 2019 Mar 11.

Department of Microbiology, University of Alabama at Birmingham, Birmingham, AL, USA.

The glycome describes the complete repertoire of glycoconjugates composed of carbohydrate chains, or glycans, that are covalently linked to lipid or protein molecules. Glycoconjugates are formed through a process called glycosylation and can differ in their glycan sequences, the connections between them and their length. Glycoconjugate synthesis is a dynamic process that depends on the local milieu of enzymes, sugar precursors and organelle structures as well as the cell types involved and cellular signals. Read More

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http://www.nature.com/articles/s41581-019-0129-4
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March 2019
8 Reads

Evidence-based therapy for undocumented immigrants with ESRD.

Nat Rev Nephrol 2019 Mar 8. Epub 2019 Mar 8.

Department of Medicine, Division of Nephrology, University of Washington School of Medicine, Seattle, WA, USA.

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http://dx.doi.org/10.1038/s41581-019-0137-4DOI Listing

Tubuloids for modelling kidney disease.

Authors:
Susan J Allison

Nat Rev Nephrol 2019 Mar 5. Epub 2019 Mar 5.

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0138-3DOI Listing

A materials approach for kidney organoids.

Authors:
Susan J Allison

Nat Rev Nephrol 2019 Mar 5. Epub 2019 Mar 5.

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0139-2DOI Listing

Nanosensors enable early detection of acute T cell-mediated rejection of transplants.

Authors:
Susan J Allison

Nat Rev Nephrol 2019 May;15(5):253

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0134-7DOI Listing

ABCF1 switches off inflammation in sepsis.

Authors:
Ellen F Carney

Nat Rev Nephrol 2019 May;15(5):255

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0136-5DOI Listing

Macrophages support kidney and vasculature assembly in development.

Authors:
Susan J Allison

Nat Rev Nephrol 2019 Feb 27. Epub 2019 Feb 27.

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0133-8DOI Listing
February 2019

Ubiquitylation of PTEN drives fibrosis in diabetic kidney disease.

Authors:
Susan J Allison

Nat Rev Nephrol 2019 May;15(5):254

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0130-yDOI Listing

Immune mechanisms of salt-sensitive hypertension and renal end-organ damage.

Authors:
David L Mattson

Nat Rev Nephrol 2019 May;15(5):290-300

Department of Physiology, Medical College of Wisconsin, Milwaukee, WI, USA.

Immune mechanisms have been recognized to have a role in the pathogenesis of hypertension, vascular disease and kidney damage in humans and animals for many decades. Contemporary advances in experimentation have permitted a deeper understanding of the mechanisms by which inflammation and immunity participate in cardiovascular disease, and multiple observations have demonstrated strong correlations between the discoveries made in animals and those made in patients with hypertension. Of note, striking phenotypic similarities have been observed in the infiltration of immune cells in the kidney and the development of end-organ damage in patients and animal models with sodium-sensitive hypertension. Read More

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http://www.nature.com/articles/s41581-019-0121-z
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May 2019
8 Reads

Succinate homeostasis protects against lithogenesis and hypertension.

Authors:
Ellen F Carney

Nat Rev Nephrol 2019 May;15(5):255

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0132-9DOI Listing

Immunity, microbiota and kidney disease.

Nat Rev Nephrol 2019 May;15(5):263-274

Department of Immunobiology, Yale University School of Medicine, New Haven, CT, USA.

The recognition that intestinal microbiota exert profound effects on human health has led to major advances in our understanding of disease processes. Studies over the past 20 years have shown that host components, including components of the host immune system, shape the microbial community. Pathogenic alterations in commensal microorganisms contribute to disease manifestations that are generally considered to be noncommunicable, such as inflammatory bowel disease, diabetes mellitus and liver disease, through a variety of mechanisms, including effects on host immunity. Read More

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http://dx.doi.org/10.1038/s41581-019-0118-7DOI Listing
May 2019
9 Reads

Pre-eclampsia: pathogenesis, novel diagnostics and therapies.

Nat Rev Nephrol 2019 May;15(5):275-289

Departments of Medicine and Biomedical Sciences, Cedars-Sinai Medical Center, Los Angeles, CA, USA.

Pre-eclampsia is a complication of pregnancy that is associated with substantial maternal and fetal morbidity and mortality. The disease presents with new-onset hypertension and often proteinuria in the mother, which can progress to multi-organ dysfunction, including hepatic, renal and cerebral disease, if the fetus and placenta are not delivered. Maternal endothelial dysfunction due to circulating factors of fetal origin from the placenta is a hallmark of pre-eclampsia. Read More

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http://dx.doi.org/10.1038/s41581-019-0119-6DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC6472952PMC

PIVOTAL trial: iron loading improves clinical outcomes.

Authors:
Daniel W Coyne

Nat Rev Nephrol 2019 May;15(5):260-261

Washington University School of Medicine, Division of Nephrology, St Louis, MO, USA.

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http://www.nature.com/articles/s41581-019-0128-5
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May 2019
11 Reads

Modelling diabetic vasculopathy with human vessel organoids.

Authors:
Ryuji Morizane

Nat Rev Nephrol 2019 May;15(5):258-260

Renal Division, Brigham and Women's Hospital, Boston, MA, USA.

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http://dx.doi.org/10.1038/s41581-019-0125-8DOI Listing

Growing mouse kidneys in rats.

Nat Rev Nephrol 2019 May;15(5):255

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0127-6DOI Listing

Fluid flow enhances vascularization and maturation of kidney organoids.

Authors:
Susan J Allison

Nat Rev Nephrol 2019 May;15(5):254

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0126-7DOI Listing

Cyclin G1 drives maladaptive renal repair.

Authors:
Monica Wang

Nat Rev Nephrol 2019 Apr;15(4):191

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0124-9DOI Listing

Albuminuria: a target for clinical trials in kidney disease?

Nat Rev Nephrol 2019 May;15(5):257-258

Geffen School of Medicine, UCLA, Los Angeles, CA, USA.

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http://dx.doi.org/10.1038/s41581-019-0123-xDOI Listing

Some nephron progenitors fail to commit.

Authors:
Ellen F Carney

Nat Rev Nephrol 2019 Apr;15(4):192

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0122-yDOI Listing

Cellular signalling by primary cilia in development, organ function and disease.

Nat Rev Nephrol 2019 Apr;15(4):199-219

Department of Biology, Section of Cell Biology and Physiology, University of Copenhagen, Copenhagen, Denmark.

Primary cilia project in a single copy from the surface of most vertebrate cell types; they detect and transmit extracellular cues to regulate diverse cellular processes during development and to maintain tissue homeostasis. The sensory capacity of primary cilia relies on the coordinated trafficking and temporal localization of specific receptors and associated signal transduction modules in the cilium. The canonical Hedgehog (HH) pathway, for example, is a bona fide ciliary signalling system that regulates cell fate and self-renewal in development and tissue homeostasis. Read More

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http://www.nature.com/articles/s41581-019-0116-9
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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC6426138PMC
April 2019
32 Reads

Uraemic syndrome of chronic kidney disease: altered remote sensing and signalling.

Nat Rev Nephrol 2019 May;15(5):301-316

Department of Pediatrics, University of California San Diego, La Jolla, CA, USA.

Uraemic syndrome (also known as uremic syndrome) in patients with advanced chronic kidney disease involves the accumulation in plasma of small-molecule uraemic solutes and uraemic toxins (also known as uremic toxins), dysfunction of multiple organs and dysbiosis of the gut microbiota. As such, uraemic syndrome can be viewed as a disease of perturbed inter-organ and inter-organism (host-microbiota) communication. Multiple biological pathways are affected, including those controlled by solute carrier (SLC) and ATP-binding cassette (ABC) transporters and drug-metabolizing enzymes, many of which are also involved in drug absorption, distribution, metabolism and elimination (ADME). Read More

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http://dx.doi.org/10.1038/s41581-019-0111-1DOI Listing
May 2019
7 Reads

Kidney disease in the Global Burden of Disease Study 2017.

Nat Rev Nephrol 2019 Apr;15(4):193-194

School of Primary Care and Population Sciences, Faculty of Medicine, University of Southampton, Southampton, UK.

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http://www.nature.com/articles/s41581-019-0120-0
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April 2019
4 Reads

The genetic changes of Wilms tumour.

Nat Rev Nephrol 2019 Apr;15(4):240-251

Wellcome Sanger Institute, Cambridge, UK.

Wilms tumour is the most common renal malignancy of childhood. The disease is curable in the majority of cases, albeit at considerable cost in terms of late treatment-related effects in some children. However, one in ten children with Wilms tumour will die of their disease despite modern treatment approaches. Read More

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http://dx.doi.org/10.1038/s41581-019-0112-0DOI Listing
April 2019
1 Read

Macrophages: versatile players in renal inflammation and fibrosis.

Nat Rev Nephrol 2019 Mar 28;15(3):144-158. Epub 2019 Jan 28.

Department of Medicine & Therapeutics, Li Ka Shing Institute of Health Sciences, and Lui Che Woo Institute of Innovative Medicine, The Chinese University of Hong Kong, Shatin, Hong Kong, China.

Macrophages have important roles in immune surveillance and in the maintenance of kidney homeostasis; their response to renal injury varies enormously depending on the nature and duration of the insult. Macrophages can adopt a variety of phenotypes: at one extreme, M1 pro-inflammatory cells contribute to infection clearance but can also promote renal injury; at the other extreme, M2 anti-inflammatory cells have a reparative phenotype and can contribute to the resolution phase of the response to injury. In addition, bone marrow monocytes can differentiate into myeloid-derived suppressor cells that can regulate T cell immunity in the kidney. Read More

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http://dx.doi.org/10.1038/s41581-019-0110-2DOI Listing
March 2019
1 Read

C3 glomerulopathy - understanding a rare complement-driven renal disease.

Nat Rev Nephrol 2019 Mar;15(3):129-143

Molecular Otolaryngology and Renal Research Laboratories and the Departments of Internal Medicine and Pediatrics (Divisions of Nephrology), Carver College of Medicine, University of Iowa, Iowa City, IA, USA.

The C3 glomerulopathies are a group of rare kidney diseases characterized by complement dysregulation occurring in the fluid phase and in the glomerular microenvironment, which results in prominent complement C3 deposition in kidney biopsy samples. The two major subgroups of C3 glomerulopathy - dense deposit disease (DDD) and C3 glomerulonephritis (C3GN) - have overlapping clinical and pathological features suggestive of a disease continuum. Dysregulation of the complement alternative pathway is fundamental to the manifestations of C3 glomerulopathy, although terminal pathway dysregulation is also common. Read More

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http://www.nature.com/articles/s41581-018-0107-2
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March 2019
24 Reads
8.542 Impact Factor

Nitric oxide protects against AKI by reprogramming metabolism.

Nat Rev Nephrol 2019 Apr;15(4):195-196

Service and Laboratory of Nephrology, Department of Medicine, University and University Hospital of Geneva, Geneva, Switzerland.

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http://www.nature.com/articles/s41581-019-0113-z
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April 2019
1 Read

The genetic architecture of blood pressure.

Authors:
Ellen F Carney

Nat Rev Nephrol 2019 Apr;15(4):192

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0117-8DOI Listing
April 2019
2 Reads

DNA methylation links intrauterine stress with abnormal nephrogenesis.

Authors:
Samir S El-Dahr

Nat Rev Nephrol 2019 Apr;15(4):196-197

Department of Pediatrics, Tulane University School of Medicine, New Orleans, LA, USA.

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http://dx.doi.org/10.1038/s41581-019-0114-yDOI Listing
April 2019
1 Read

CNVs in CAKUT.

Authors:
Susan J Allison

Nat Rev Nephrol 2019 Apr;15(4):192

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-019-0115-xDOI Listing
April 2019
1 Read

Evolving concepts in the pathogenesis of uraemic cardiomyopathy.

Nat Rev Nephrol 2019 Mar;15(3):159-175

Joan C. Edwards School of Medicine, Marshall University, Huntington, WV, USA.

The term uraemic cardiomyopathy refers to the cardiac abnormalities that are seen in patients with chronic kidney disease (CKD). Historically, this term was used to describe a severe cardiomyopathy that was associated with end-stage renal disease and characterized by severe functional abnormalities that could be reversed following renal transplantation. In a modern context, uraemic cardiomyopathy describes the clinical phenotype of cardiac disease that accompanies CKD and is perhaps best characterized as diastolic dysfunction seen in conjunction with left ventricular hypertrophy and fibrosis. Read More

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http://www.nature.com/articles/s41581-018-0101-8
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March 2019
14 Reads

Epigenetic regulation in AKI and kidney repair: mechanisms and therapeutic implications.

Nat Rev Nephrol 2019 Apr;15(4):220-239

Department of Cellular Biology and Anatomy, Medical College of Georgia, Augusta University and Charlie Norwood VA Medical Center, Augusta, GA, USA.

Acute kidney injury (AKI) is a major public health concern associated with high morbidity and mortality. Despite decades of research, the pathogenesis of AKI remains incompletely understood and effective therapies are lacking. An increasing body of evidence suggests a role for epigenetic regulation in the process of AKI and kidney repair, involving remarkable changes in histone modifications, DNA methylation and the expression of various non-coding RNAs. Read More

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http://dx.doi.org/10.1038/s41581-018-0103-6DOI Listing
April 2019
2 Reads

Local solutions save young lives.

Authors:
Simon J Davies

Nat Rev Nephrol 2019 Mar;15(3):127-128

Faculty of Medicine and Health Sciences, Keele University, Keele, Staffordshire, UK.

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http://dx.doi.org/10.1038/s41581-018-0109-0DOI Listing
March 2019
5 Reads
8.542 Impact Factor

Endothelium structure and function in kidney health and disease.

Nat Rev Nephrol 2019 Feb;15(2):87-108

INSERM, UMR_S 1138, Centre de Recherche des Cordeliers, F-75006, Paris, France.

The kidney harbours different types of endothelia, each with specific structural and functional characteristics. The glomerular endothelium, which is highly fenestrated and covered by a rich glycocalyx, participates in the sieving properties of the glomerular filtration barrier and in the maintenance of podocyte structure. The microvascular endothelium in peritubular capillaries, which is also fenestrated, transports reabsorbed components and participates in epithelial cell function. Read More

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http://dx.doi.org/10.1038/s41581-018-0098-zDOI Listing
February 2019
3 Reads

Progress in the understanding of polycystic kidney disease.

Nat Rev Nephrol 2019 Feb;15(2):70-72

Division of Nephrology and Hypertension, Mayo Clinic College of Medicine, Rochester, MN, USA.

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http://dx.doi.org/10.1038/s41581-018-0108-1DOI Listing
February 2019
9 Reads

Sodium transport in diabetes: two sides to the coin.

Nat Rev Nephrol 2019 Mar;15(3):125-126

Division of Nephrology, Department of Medicine, University of Toronto, Toronto, Ontario, Canada.

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http://www.nature.com/articles/s41581-018-0106-3
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March 2019
3 Reads

Single-cell genomics and gene editing: implications for nephrology.

Nat Rev Nephrol 2019 Feb;15(2):63-64

Division of Nephrology, Department of Medicine, Washington University in Saint Louis School of Medicine, St. Louis, MO, USA.

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http://dx.doi.org/10.1038/s41581-018-0094-3DOI Listing
February 2019
1 Read

Sepsis halts renal protein translation.

Authors:
Monica Wang

Nat Rev Nephrol 2019 Mar;15(3):124

Nature Reviews Nephrology, .

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http://dx.doi.org/10.1038/s41581-018-0104-5DOI Listing
March 2019
1 Read