2,250 results match your criteria Nature Reviews Molecular Cell Biology [Journal]


Publisher Correction: A call for a better understanding of causation in cell biology.

Nat Rev Mol Cell Biol 2019 Apr 18. Epub 2019 Apr 18.

Allen Discovery Center at Tufts University, Medford, MA, USA.

In the above article, the name of the first author was spelled incorrectly. This has been corrected in the HTML and PDF versions of the article. Read More

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http://www.nature.com/articles/s41580-019-0130-6
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http://dx.doi.org/10.1038/s41580-019-0130-6DOI Listing
April 2019
2 Reads

Quality and quantity control of gene expression by nonsense-mediated mRNA decay.

Nat Rev Mol Cell Biol 2019 Apr 16. Epub 2019 Apr 16.

Department of Biochemistry and Biophysics, School of Medicine and Dentistry, University of Rochester, Rochester, NY, USA.

Nonsense-mediated mRNA decay (NMD) is one of the best characterized and most evolutionarily conserved cellular quality control mechanisms. Although NMD was first found to target one-third of mutated, disease-causing mRNAs, it is now known to also target ~10% of unmutated mammalian mRNAs to facilitate appropriate cellular responses - adaptation, differentiation or death - to environmental changes. Mutations in NMD genes in humans are associated with intellectual disability and cancer. Read More

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http://www.nature.com/articles/s41580-019-0126-2
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http://dx.doi.org/10.1038/s41580-019-0126-2DOI Listing
April 2019
1 Read

Metabolic regulation of cell growth and proliferation.

Nat Rev Mol Cell Biol 2019 Apr 11. Epub 2019 Apr 11.

Department of Cancer Biology and Genetics, Memorial Sloan Kettering Cancer Center, New York, NY, USA.

Cellular metabolism is at the foundation of all biological activities. The catabolic processes that support cellular bioenergetics and survival have been well studied. By contrast, how cells alter their metabolism to support anabolic biomass accumulation is less well understood. Read More

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http://dx.doi.org/10.1038/s41580-019-0123-5DOI Listing

A call for a better understanding of causation in cell biology.

Nat Rev Mol Cell Biol 2019 Apr 8. Epub 2019 Apr 8.

Allen Discovery Center at Tufts University, Medford, MA, USA.

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http://dx.doi.org/10.1038/s41580-019-0127-1DOI Listing
April 2019
1 Read

Cellular senescence causes ageing.

Authors:
Jesús Gil

Nat Rev Mol Cell Biol 2019 Apr 8. Epub 2019 Apr 8.

MRC London Institute of Medical Sciences (LMS) and Institute of Clinical Sciences (ICS), Imperial College London, London, UK.

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http://dx.doi.org/10.1038/s41580-019-0128-0DOI Listing

Establishing and regulating the composition of cilia for signal transduction.

Nat Rev Mol Cell Biol 2019 Apr 4. Epub 2019 Apr 4.

Center of Human and Molecular Biology (ZHMB), Medical Biochemistry and Molecular Biology, Saarland University, Homburg, Germany.

The primary cilium is a hair-like surface-exposed organelle of the eukaryotic cell that decodes a variety of signals - such as odorants, light and Hedgehog morphogens - by altering the local concentrations and activities of signalling proteins. Signalling within the cilium is conveyed through a diverse array of second messengers, including conventional signalling molecules (such as cAMP) and some unusual intermediates (such as sterols). Diffusion barriers at the ciliary base establish the unique composition of this signalling compartment, and cilia adapt their proteome to signalling demands through regulated protein trafficking. Read More

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http://dx.doi.org/10.1038/s41580-019-0116-4DOI Listing
April 2019
1 Read

Histone serotonylation boosts neuronal transcription.

Nat Rev Mol Cell Biol 2019 Apr 3. Epub 2019 Apr 3.

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0124-4DOI Listing

Mechanisms and functions of ribosome-associated protein quality control.

Nat Rev Mol Cell Biol 2019 Apr 2. Epub 2019 Apr 2.

Center for Molecular Biology of Heidelberg University (ZMBH), Heidelberg, Germany.

The stalling of ribosomes during protein synthesis results in the production of truncated polypeptides that can have deleterious effects on cells and therefore must be eliminated. In eukaryotes, this function is carried out by a dedicated surveillance mechanism known as ribosome-associated protein quality control (RQC). The E3 ubiquitin ligase Ltn1 (listerin in mammals) plays a key part in RQC by targeting the aberrant nascent polypeptides for proteasomal degradation. Read More

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http://dx.doi.org/10.1038/s41580-019-0118-2DOI Listing
April 2019
2 Reads

On making bones or fat.

Authors:
Kim Baumann

Nat Rev Mol Cell Biol 2019 Mar 28. Epub 2019 Mar 28.

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0125-3DOI Listing

Phase separation tunes signal transduction.

Authors:
Paulina Strzyz

Nat Rev Mol Cell Biol 2019 Mar 27. Epub 2019 Mar 27.

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0121-7DOI Listing
March 2019
1 Read

Hypoxia makes its mark on histones.

Authors:
Paulina Strzyz

Nat Rev Mol Cell Biol 2019 Mar 27. Epub 2019 Mar 27.

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0122-6DOI Listing
March 2019
1 Read

Blocking autophagy to starve pancreatic cancer.

Nat Rev Mol Cell Biol 2019 Mar 26. Epub 2019 Mar 26.

Lineberger Comprehensive Cancer Center, University of North Carolina at Chapel Hill, Chapel Hill, NC, USA.

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http://dx.doi.org/10.1038/s41580-019-0120-8DOI Listing

The role of transcription in shaping the spatial organization of the genome.

Nat Rev Mol Cell Biol 2019 Mar 18. Epub 2019 Mar 18.

European Molecular Biology Laboratory (EMBL), Genome Biology Unit, Heidelberg, Germany.

The spatial organization of the genome into compartments and topologically associated domains can have an important role in the regulation of gene expression. But could gene expression conversely regulate genome organization? Here, we review recent studies that assessed the requirement of transcription and/or the transcription machinery for the establishment or maintenance of genome topology. The results reveal different requirements at different genomic scales. Read More

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http://www.nature.com/articles/s41580-019-0114-6
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http://dx.doi.org/10.1038/s41580-019-0114-6DOI Listing
March 2019
4 Reads

Under-replicated DNA gets a second chance.

Authors:
Anne Mirabella

Nat Rev Mol Cell Biol 2019 Mar 18. Epub 2019 Mar 18.

Associate Editor, Nature Communications, .

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http://dx.doi.org/10.1038/s41580-019-0119-1DOI Listing

STINGing revelations.

Authors:
Paulina Strzyz

Nat Rev Mol Cell Biol 2019 Mar 14. Epub 2019 Mar 14.

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0117-3DOI Listing
March 2019
1 Read

Erratic loci relationships.

Nat Rev Mol Cell Biol 2019 Apr;20(4):196-197

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0115-5DOI Listing

The multiple mechanisms that regulate p53 activity and cell fate.

Nat Rev Mol Cell Biol 2019 Apr;20(4):199-210

Department of Systems Biology, Harvard Medical School, Boston, MA, USA.

The tumour suppressor p53 has a central role in the response to cellular stress. Activated p53 transcriptionally regulates hundreds of genes that are involved in multiple biological processes, including in DNA damage repair, cell cycle arrest, apoptosis and senescence. In the context of DNA damage, p53 is thought to be a decision-making transcription factor that selectively activates genes as part of specific gene expression programmes to determine cellular outcomes. Read More

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http://www.nature.com/articles/s41580-019-0110-x
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http://dx.doi.org/10.1038/s41580-019-0110-xDOI Listing
April 2019
1 Read

Author Correction: Telomeres in cancer: tumour suppression and genome instability.

Nat Rev Mol Cell Biol 2019 Apr;20(4):259

Laboratory for Cell Biology and Genetics, The Rockefeller University, 1230 York Avenue, New York, New York, 10065, USA.

In the original Fig. 2a, telomeres are erroneously depicted having blunt ends following resection and CST-mediated fill-in. Instead, telomeres retain 3' overhangs, as depicted below. Read More

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http://dx.doi.org/10.1038/s41580-019-0113-7DOI Listing

Identification of bioactive metabolites using activity metabolomics.

Nat Rev Mol Cell Biol 2019 Feb 27. Epub 2019 Feb 27.

The Scripps Research Institute, Center for Metabolomics and Mass Spectrometry, La Jolla, CA, USA.

The metabolome, the collection of small-molecule chemical entities involved in metabolism, has traditionally been studied with the aim of identifying biomarkers in the diagnosis and prediction of disease. However, the value of metabolome analysis (metabolomics) has been redefined from a simple biomarker identification tool to a technology for the discovery of active drivers of biological processes. It is now clear that the metabolome affects cellular physiology through modulation of other 'omics' levels, including the genome, epigenome, transcriptome and proteome. Read More

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http://dx.doi.org/10.1038/s41580-019-0108-4DOI Listing
February 2019
2 Reads

Publisher Correction: Breaking the chains: deubiquitylating enzyme specificity begets function.

Nat Rev Mol Cell Biol 2019 Feb 19. Epub 2019 Feb 19.

Ubiquitin Signalling Division, The Walter and Eliza Hall Institute of Medical Research, Melbourne, Australia.

Figure 2 of the article as originally published contained a graphic editing error, whereby the publisher's redrawn figure wrongly indicated the presence of a Drosophila melanogaster orthologue of ZUP1. This has been corrected in the HTML and PDF versions of the manuscript. Read More

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http://dx.doi.org/10.1038/s41580-019-0112-8DOI Listing
February 2019

The unusual SASPects.

Authors:
Paulina Strzyz

Nat Rev Mol Cell Biol 2019 Apr;20(4):195

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0111-9DOI Listing
April 2019
1 Read

May the force be with your lipids.

Authors:
Grant Otto

Nat Rev Mol Cell Biol 2019 Apr;20(4):196-197

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0109-3DOI Listing

Haematopoietic stem cell activity and interactions with the niche.

Nat Rev Mol Cell Biol 2019 Feb 11. Epub 2019 Feb 11.

Ruth L. and David S. Gottesman Institute for Stem Cell and Regenerative Medicine Research, Albert Einstein College of Medicine, Bronx, New York, NY, USA.

The haematopoietic stem cell (HSC) microenvironment in the bone marrow, termed the niche, ensures haematopoietic homeostasis by controlling the proliferation, self-renewal, differentiation and migration of HSCs and progenitor cells at steady state and in response to emergencies and injury. Improved methods for HSC isolation, driven by advances in single-cell and molecular technologies, have led to a better understanding of their behaviour, heterogeneity and lineage fate and of the niche cells and signals that regulate their function. Niche regulatory signals can be in the form of cell-bound or secreted factors and other local physical cues. Read More

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http://www.nature.com/articles/s41580-019-0103-9
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http://dx.doi.org/10.1038/s41580-019-0103-9DOI Listing
February 2019
15 Reads

Breaking the chains: deubiquitylating enzyme specificity begets function.

Nat Rev Mol Cell Biol 2019 Feb 7. Epub 2019 Feb 7.

Ubiquitin Signalling Division, The Walter and Eliza Hall Institute of Medical Research, Melbourne, Australia.

The deubiquitylating enzymes (DUBs, also known as deubiquitylases or deubiquitinases) maintain the dynamic state of the cellular ubiquitome by releasing conjugated ubiquitin from proteins. In light of the many cellular functions of ubiquitin, DUBs occupy key roles in almost all aspects of cell behaviour. Many DUBs show selectivity for particular ubiquitin linkage types or positions within ubiquitin chains. Read More

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http://dx.doi.org/10.1038/s41580-019-0099-1DOI Listing
February 2019

Detouring the roadblocks in gene expression.

Nat Rev Mol Cell Biol 2019 Apr;20(4):197

Max Delbruck Center for Molecular Medicine, Berlin Institute for Medical Systems Biology, Berlin, Germany.

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http://dx.doi.org/10.1038/s41580-019-0107-5DOI Listing

The proteostasis network and its decline in ageing.

Nat Rev Mol Cell Biol 2019 Feb 7. Epub 2019 Feb 7.

Max Planck Institute of Biochemistry, Department of Cellular Biochemistry, Martinsried, Germany.

Ageing is a major risk factor for the development of many diseases, prominently including neurodegenerative disorders such as Alzheimer disease and Parkinson disease. A hallmark of many age-related diseases is the dysfunction in protein homeostasis (proteostasis), leading to the accumulation of protein aggregates. In healthy cells, a complex proteostasis network, comprising molecular chaperones and proteolytic machineries and their regulators, operates to ensure the maintenance of proteostasis. Read More

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http://dx.doi.org/10.1038/s41580-019-0101-yDOI Listing
February 2019
3 Reads

Publisher Correction: Regulation of microRNA biogenesis and its crosstalk with other cellular pathways.

Nat Rev Mol Cell Biol 2019 Feb 6. Epub 2019 Feb 6.

Biochemistry Center Regensburg (BZR), Laboratory for RNA Biology, University of Regensburg, Regensburg, Germany.

The legend of Figure 2 neglected to acknowledge that part b was adapted with permission from ref., Elsevier and that part d, third panel from the left was reproduced from ref., Springer Nature Limited. Read More

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http://dx.doi.org/10.1038/s41580-019-0106-6DOI Listing
February 2019

Forcing through barriers.

Authors:
Paulina Strzyz

Nat Rev Mol Cell Biol 2019 Mar;20(3):136

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0104-8DOI Listing

Telomere crisis activates autophagic death.

Nat Rev Mol Cell Biol 2019 Mar;20(3):133

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0105-7DOI Listing

How cells keep scale.

Authors:
Paulina Strzyz

Nat Rev Mol Cell Biol 2019 Mar;20(3):136

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0102-xDOI Listing

Spatial proteomics: a powerful discovery tool for cell biology.

Nat Rev Mol Cell Biol 2019 Jan 18. Epub 2019 Jan 18.

Max Planck Institute of Biochemistry, Department of Proteomics and Signal Transduction, Martinsried, Germany.

Protein subcellular localization is tightly controlled and intimately linked to protein function in health and disease. Capturing the spatial proteome - that is, the localizations of proteins and their dynamics at the subcellular level - is therefore essential for a complete understanding of cell biology. Owing to substantial advances in microscopy, mass spectrometry and machine learning applications for data analysis, the field is now mature for proteome-wide investigations of spatial cellular regulation. Read More

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http://www.nature.com/articles/s41580-018-0094-y
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http://dx.doi.org/10.1038/s41580-018-0094-yDOI Listing
January 2019
22 Reads

Regulation of apoptosis in health and disease: the balancing act of BCL-2 family proteins.

Nat Rev Mol Cell Biol 2019 Mar;20(3):175-193

John B. Little Center for Radiation Sciences, Harvard T. H. Chan School of Public Health, Boston, MA, USA.

The loss of vital cells within healthy tissues contributes to the development, progression and treatment outcomes of many human disorders, including neurological and infectious diseases as well as environmental and medical toxicities. Conversely, the abnormal survival and accumulation of damaged or superfluous cells drive prominent human pathologies such as cancers and autoimmune diseases. Apoptosis is an evolutionarily conserved cell death pathway that is responsible for the programmed culling of cells during normal eukaryotic development and maintenance of organismal homeostasis. Read More

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http://dx.doi.org/10.1038/s41580-018-0089-8DOI Listing
March 2019
1 Read

Enhancers and promoters regulate burst kinetics.

Authors:
Grant Otto

Nat Rev Mol Cell Biol 2019 Mar;20(3):134-135

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-019-0100-zDOI Listing
March 2019
1 Read

Crosstalk between metabolism and circadian clocks.

Nat Rev Mol Cell Biol 2019 Apr;20(4):227-241

Department of Biomolecular Sciences, Weizmann Institute of Science, Rehovot, Israel.

Humans, like all mammals, partition their daily behaviour into activity (wakefulness) and rest (sleep) phases that differ largely in their metabolic requirements. The circadian clock evolved as an autonomous timekeeping system that aligns behavioural patterns with the solar day and supports the body functions by anticipating and coordinating the required metabolic programmes. The key component of this synchronization is a master clock in the brain, which responds to light-darkness cues from the environment. Read More

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http://dx.doi.org/10.1038/s41580-018-0096-9DOI Listing
April 2019
2 Reads

Mitochondrial proteins: from biogenesis to functional networks.

Nat Rev Mol Cell Biol 2019 Jan 9. Epub 2019 Jan 9.

Institute of Biochemistry and Molecular Biology, ZBMZ, Faculty of Medicine, University of Freiburg, Freiburg, Germany.

Mitochondria are essential for the viability of eukaryotic cells as they perform crucial functions in bioenergetics, metabolism and signalling and have been associated with numerous diseases. Recent functional and proteomic studies have revealed the remarkable complexity of mitochondrial protein organization. Protein machineries with diverse functions such as protein translocation, respiration, metabolite transport, protein quality control and the control of membrane architecture interact with each other in dynamic networks. Read More

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http://dx.doi.org/10.1038/s41580-018-0092-0DOI Listing
January 2019
2 Reads

Adipogenesis and metabolic health.

Nat Rev Mol Cell Biol 2019 Apr;20(4):242-258

Touchstone Diabetes Center, Department of Internal Medicine, The University of Texas Southwestern Medical Center, Dallas, TX, USA.

Obesity is characterized by increased adipose tissue mass and has been associated with a strong predisposition towards metabolic diseases and cancer. Thus, it constitutes a public health issue of major proportion. The expansion of adipose depots can be driven either by the increase in adipocyte size (hypertrophy) or by the formation of new adipocytes from precursor differentiation in the process of adipogenesis (hyperplasia). Read More

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http://dx.doi.org/10.1038/s41580-018-0093-zDOI Listing
April 2019
7 Reads

Lipid give and take.

Authors:
Grant Otto

Nat Rev Mol Cell Biol 2019 Mar;20(3):134-135

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-018-0098-7DOI Listing
March 2019
1 Read

Freedom of expression in methylated regions.

Nat Rev Mol Cell Biol 2019 Feb;20(2):66-67

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-018-0097-8DOI Listing
February 2019
2 Reads

Mitochondria unite.

Authors:
Paulina Strzyz

Nat Rev Mol Cell Biol 2019 Feb;20(2):65

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-018-0095-xDOI Listing
February 2019
1 Read

Hippo-YAP/TAZ signalling in organ regeneration and regenerative medicine.

Nat Rev Mol Cell Biol 2019 Apr;20(4):211-226

VIB Center for Cancer Biology, and KU Leuven Department of Oncology, University of Leuven, Leuven, Belgium.

The Hippo pathway and its downstream effectors, the transcriptional co-activators Yes-associated protein (YAP) and transcriptional co-activator with PDZ-binding motif (TAZ), regulate organ growth and cell plasticity during animal development and regeneration. Remarkably, experimental activation of YAP/TAZ in the mouse can promote regeneration in organs with poor or compromised regenerative capacity, such as the adult heart and the liver and intestine of old or diseased mice. However, therapeutic YAP/TAZ activation may cause serious side effects. Read More

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http://dx.doi.org/10.1038/s41580-018-0086-yDOI Listing
April 2019
1 Read

Dynamics and functions of lipid droplets.

Nat Rev Mol Cell Biol 2019 Mar;20(3):137-155

Sir William Dunn School of Pathology, University of Oxford, Oxford, UK.

Lipid droplets are storage organelles at the centre of lipid and energy homeostasis. They have a unique architecture consisting of a hydrophobic core of neutral lipids, which is enclosed by a phospholipid monolayer that is decorated by a specific set of proteins. Originating from the endoplasmic reticulum, lipid droplets can associate with most other cellular organelles through membrane contact sites. Read More

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http://dx.doi.org/10.1038/s41580-018-0085-zDOI Listing
March 2019
1 Read

6mA in stress tolerance across generations.

Authors:
Grant Otto

Nat Rev Mol Cell Biol 2019 Feb;20(2):66-67

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-018-0091-1DOI Listing
February 2019
1 Read

Guardians of the oocyte methylome.

Authors:
Kim Baumann

Nat Rev Mol Cell Biol 2019 Jan;20(1):2-3

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-018-0090-2DOI Listing
January 2019
5 Reads

A MAD way to regulate mitosis.

Authors:
David Barford

Nat Rev Mol Cell Biol 2019 Mar;20(3):135

MRC Laboratory of Molecular Biology, Cambridge, UK.

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http://dx.doi.org/10.1038/s41580-018-0084-0DOI Listing
March 2019
2 Reads

Sorting it out at the Golgi.

Authors:
Paulina Strzyz

Nat Rev Mol Cell Biol 2019 Jan;20(1):2-3

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-018-0087-xDOI Listing
January 2019
1 Read

Protein trafficking through TIGER domains.

Authors:
Grant Otto

Nat Rev Mol Cell Biol 2019 Jan;20(1)

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-018-0088-9DOI Listing
January 2019
2 Reads

Ribosome assembly coming into focus.

Nat Rev Mol Cell Biol 2019 Feb;20(2):116-131

Department of Biological Sciences, Carnegie Mellon University, Pittsburgh, PA, USA.

In the past 25 years, genetic and biochemical analyses of ribosome assembly in yeast have identified most of the factors that participate in this complex pathway and have generated models for the mechanisms driving the assembly. More recently, the publication of numerous cryo-electron microscopy structures of yeast ribosome assembly intermediates has provided near-atomic resolution snapshots of ribosome precursor particles. Satisfyingly, these structural data support the genetic and biochemical models and provide additional mechanistic insight into ribosome assembly. Read More

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http://www.nature.com/articles/s41580-018-0078-y
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February 2019
3 Reads

Functions and mechanisms of non-histone protein acetylation.

Nat Rev Mol Cell Biol 2019 Mar;20(3):156-174

The Novo Nordisk Foundation Center for Protein Research, Faculty of Health Sciences, University of Copenhagen, Copenhagen, Denmark.

Nε-lysine acetylation was discovered more than half a century ago as a post-translational modification of histones and has been extensively studied in the context of transcription regulation. In the past decade, proteomic analyses have revealed that non-histone proteins are frequently acetylated and constitute a major portion of the acetylome in mammalian cells. Indeed, non-histone protein acetylation is involved in key cellular processes relevant to physiology and disease, such as gene transcription, DNA damage repair, cell division, signal transduction, protein folding, autophagy and metabolism. Read More

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http://dx.doi.org/10.1038/s41580-018-0081-3DOI Listing
March 2019
71 Reads

New insights into the mechanisms of epithelial-mesenchymal transition and implications for cancer.

Nat Rev Mol Cell Biol 2019 Feb;20(2):69-84

Whitehead Institute for Biomedical Research, Cambridge, MA, USA.

Epithelial-mesenchymal transition (EMT) is a cellular programme that is known to be crucial for embryogenesis, wound healing and malignant progression. During EMT, cell-cell and cell-extracellular matrix interactions are remodelled, which leads to the detachment of epithelial cells from each other and the underlying basement membrane, and a new transcriptional programme is activated to promote the mesenchymal fate. In the context of neoplasias, EMT confers on cancer cells increased tumour-initiating and metastatic potential and a greater resistance to elimination by several therapeutic regimens. Read More

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http://dx.doi.org/10.1038/s41580-018-0080-4DOI Listing
February 2019
13 Reads

Neurodegenerative polyglutamylation.

Authors:
Paulina Strzyz

Nat Rev Mol Cell Biol 2019 Jan;20(1)

Nature Reviews Molecular Cell Biology, .

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http://dx.doi.org/10.1038/s41580-018-0083-1DOI Listing
January 2019
2 Reads