474 results match your criteria Nature Reviews Gastroenterology & Hepatology[Journal]


Bacterial strains modulate CD8 T cell function and cancer immunity.

Authors:
Hugh Thomas

Nat Rev Gastroenterol Hepatol 2019 Feb 7. Epub 2019 Feb 7.

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-019-0120-3DOI Listing
February 2019

ACLF monocyte dysfunction.

Authors:
Clemens Thoma

Nat Rev Gastroenterol Hepatol 2019 Feb 5. Epub 2019 Feb 5.

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-019-0119-9DOI Listing
February 2019

Regulatory T cells in cancer immunosuppression - implications for anticancer therapy.

Nat Rev Clin Oncol 2019 Jan 31. Epub 2019 Jan 31.

Division of Cancer Immunology, Research Institute/Exploratory Oncology Research and Clinical Trial Centre (EPOC), National Cancer Centre, Tokyo/Chiba, Japan.

Regulatory T (T) cells, an immunosuppressive subset of CD4 T cells characterized by the expression of the master transcription factor forkhead box protein P3 (FOXP3), are a component of the immune system with essential roles in maintaining self-tolerance. In addition, T cells can suppress anticancer immunity, thereby hindering protective immunosurveillance of neoplasia and hampering effective antitumour immune responses in tumour-bearing hosts, thus promoting tumour development and progression. Identification of the factors that are specifically expressed in T cells and/or that influence T cell homeostasis and function is important to understanding cancer pathogenesis and to identifying therapeutic targets. Read More

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http://dx.doi.org/10.1038/s41571-019-0175-7DOI Listing
January 2019

Mutation and clonal selection in the ageing oesophagus.

Authors:
Hugh Thomas

Nat Rev Gastroenterol Hepatol 2019 Jan 31. Epub 2019 Jan 31.

Nature Reviews Gastroenterology & Hepatology, .

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http://www.nature.com/articles/s41575-019-0117-y
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http://dx.doi.org/10.1038/s41575-019-0117-yDOI Listing
January 2019
2 Reads

Gut microbiota-mediated liver inflammation in PSC.

Authors:
Iain Dickson

Nat Rev Gastroenterol Hepatol 2019 Jan 31. Epub 2019 Jan 31.

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-019-0116-zDOI Listing
January 2019

Reduction of visceral adipose tissue mass requires IL-6 signalling.

Authors:
Clemens Thoma

Nat Rev Gastroenterol Hepatol 2019 Jan 23. Epub 2019 Jan 23.

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-019-0114-1DOI Listing
January 2019
1 Read

Panel of 10 serum metabolites shows promise in the non-invasive detection of advanced fibrosis.

Authors:
Clemens Thoma

Nat Rev Gastroenterol Hepatol 2019 Jan 23. Epub 2019 Jan 23.

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-019-0113-2DOI Listing
January 2019
1 Read

Modified FOLFIRINOX superior to gemcitabine in resected pancreatic ductal adenocarcinoma.

Authors:
Clemens Thoma

Nat Rev Gastroenterol Hepatol 2019 Jan 23. Epub 2019 Jan 23.

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-019-0112-3DOI Listing
January 2019
1 Read

Big data in IBD: a look into the future.

Nat Rev Gastroenterol Hepatol 2019 Jan 18. Epub 2019 Jan 18.

INSERM U954 and Department of Hepatogastroenterology, Nancy University Hospital, Université de Lorraine, Vandoeuvre-lès-Nancy, Nancy, France.

Big data methodologies, made possible with the increasing generation and availability of digital data and enhanced analytical capabilities, have produced new insights to improve outcomes in many disciplines. Application of big data in the health-care sector is in its early stages, although the potential for leveraging underutilized data to gain a better understanding of disease and improve quality of care is enormous. Owing to the intrinsic characteristics of inflammatory bowel disease (IBD) and the management dilemmas that it imposes, the implementation of big data research strategies not only can complement current research efforts but also could represent the only way to disentangle the complexity of the disease. Read More

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http://dx.doi.org/10.1038/s41575-019-0102-5DOI Listing
January 2019
2 Reads

Better end points needed in primary sclerosing cholangitis trials.

Nat Rev Gastroenterol Hepatol 2019 Jan 17. Epub 2019 Jan 17.

Division of Gastroenterology and Center for Autoimmune Liver Diseases, San Gerardo Hospital, Department of Medicine and Surgery, University of Milano-Bicocca, Monza, Italy.

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http://dx.doi.org/10.1038/s41575-019-0110-5DOI Listing
January 2019
2 Reads

Alcohol, liver disease and the gut microbiota.

Authors:
Jasmohan S Bajaj

Nat Rev Gastroenterol Hepatol 2019 Jan 14. Epub 2019 Jan 14.

Division of Gastroenterology, Hepatology and Nutrition, Virginia Commonwealth University and McGuire VA Medical Center, Richmond, VA, USA.

Alcoholic liver disease, which ranges from mild disease to alcoholic hepatitis and cirrhosis, is a leading cause of morbidity and mortality worldwide. Alcohol intake can lead to changes in gut microbiota composition, even before liver disease development. These alterations worsen with advancing disease and could be complicit in disease progression. Read More

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http://dx.doi.org/10.1038/s41575-018-0099-1DOI Listing
January 2019
5 Reads

Preventing brain damage in necrotizing enterocolitis.

Authors:
Clemens Thoma

Nat Rev Gastroenterol Hepatol 2019 Feb;16(2):75

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-019-0107-0DOI Listing
February 2019
1 Read

Prucalopride before surgery alleviates postoperative ileus.

Authors:
Hugh Thomas

Nat Rev Gastroenterol Hepatol 2019 Feb;16(2):76

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-019-0106-1DOI Listing
February 2019
1 Read

Coeliac disease.

Nat Rev Dis Primers 2019 Jan 10;5(1). Epub 2019 Jan 10.

Department of Internal Medicine, Tampere University Hospital and Faculty of Medicine and Life Sciences, University of Tampere, Tampere, Finland.

Coeliac disease is an immune-mediated enteropathy against dietary gluten present in wheat, rye and barley and is one of the most common lifelong food-related disorders worldwide. Coeliac disease is also considered to be a systemic disorder characterized by a variable combination of gluten-related signs and symptoms and disease-specific antibodies in addition to enteropathy. The ingestion of gluten leads to the generation of harmful gluten peptides, which, in predisposed individuals, can induce adaptive and innate immune responses. Read More

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http://www.nature.com/articles/s41572-018-0054-z
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January 2019
6 Reads

Fungal dysbiosis associated with colorectal cancer.

Authors:
Iain Dickson

Nat Rev Gastroenterol Hepatol 2019 Feb;16(2):76

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-019-0105-2DOI Listing
February 2019
1 Read

Probiotics fail to improve preschool gastroenteritis.

Authors:
Iain Dickson

Nat Rev Gastroenterol Hepatol 2019 Feb;16(2):76

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-019-0104-3DOI Listing
February 2019
1 Read

No risk reduction of colorectal adenoma with aspirin or eicosapentaenoic acid.

Authors:
Iain Dickson

Nat Rev Gastroenterol Hepatol 2019 Feb;16(2):76

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-019-0103-4DOI Listing
February 2019
1 Read

Novel connections and precision approaches.

Nat Rev Immunol 2019 Feb;19(2):75-76

Joan and Sanford I. Weill Department of Medicine, Division of Gastroenterology, Department of Microbiology and Immunology, and the Jill Roberts Institute for Research in Inflammatory Bowel Disease, Weill Cornell Medicine, Cornell University, New York, NY, USA.

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http://www.nature.com/articles/s41577-018-0114-3
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http://dx.doi.org/10.1038/s41577-018-0114-3DOI Listing
February 2019
5 Reads

A spotlight on pathogenesis, interactions and novel therapeutic options in NAFLD.

Nat Rev Gastroenterol Hepatol 2019 Feb;16(2):80-82

Division of Gastroenterology, Department of Medical Sciences, University of Turin, Turin, Italy.

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http://dx.doi.org/10.1038/s41575-018-0094-6DOI Listing
February 2019
1 Read

T cells tune intestinal stem cell fate.

Authors:
Clemens Thoma

Nat Rev Gastroenterol Hepatol 2019 Jan;16(1)

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-018-0096-4DOI Listing
January 2019
1 Read

Food perception primes the liver for metabolic adaptation.

Authors:
Iain Dickson

Nat Rev Gastroenterol Hepatol 2019 Jan;16(1)

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-018-0098-2DOI Listing
January 2019
1 Read

Fatigue in IBD: epidemiology, pathophysiology and management.

Nat Rev Gastroenterol Hepatol 2018 Dec 10. Epub 2018 Dec 10.

Division of Gastroenterology, Massachusetts General Hospital, Boston, MA, USA.

Fatigue is an important clinical problem in patients with IBD, affecting nearly 50% of patients in clinical remission and > 80% of those with active disease. The resulting decrease in quality of life and impaired work productivity and functioning contribute markedly to the societal costs of fatigue. However, despite the burden and effects of fatigue, little is known about its aetiology and pathophysiology, which impairs our ability to effectively treat this symptom. Read More

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http://www.nature.com/articles/s41575-018-0091-9
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December 2018
7 Reads

Updates on hepatic homeostasis and the many tiers of hepatobiliary repair.

Nat Rev Gastroenterol Hepatol 2019 Feb;16(2):84-86

Division of Experimental Pathology, Department of Pathology, University of Pittsburgh School of Medicine, Pittsburgh, PA, USA.

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http://dx.doi.org/10.1038/s41575-018-0090-xDOI Listing
February 2019
1 Read

FMT for drug-induced colitis.

Authors:
Hugh Thomas

Nat Rev Gastroenterol Hepatol 2019 Jan;16(1)

Nature Reviews Gastroenterology & Hepatology, .

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http://www.nature.com/articles/s41575-018-0092-8
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January 2019
13 Reads

Global epidemiology and holistic prevention of pancreatitis.

Nat Rev Gastroenterol Hepatol 2018 Nov 27. Epub 2018 Nov 27.

Division of Gastroenterology & Hepatology, University of Pittsburgh Medical Center, Pittsburgh, PA, USA.

Knowledge of pancreatitis in the 20th century was shaped predominantly by animal data and clinical trials. Several large general population-based cohort studies and comprehensive systematic literature reviews in the 21st century have had a major effect on our understanding of pancreatitis and its sequelae. This Review provides precise and up-to-date data on the burden of acute pancreatitis, chronic pancreatitis and post-pancreatitis diabetes mellitus. Read More

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http://www.nature.com/articles/s41575-018-0087-5
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November 2018
23 Reads

The role of macrophages in nonalcoholic fatty liver disease and nonalcoholic steatohepatitis.

Nat Rev Gastroenterol Hepatol 2018 Nov 27. Epub 2018 Nov 27.

Department of Hepatology and Gastroenterology, Aarhus University Hospital, Aarhus, Denmark.

Nonalcoholic fatty liver disease (NAFLD) and its inflammatory and often progressive subtype nonalcoholic steatohepatitis (NASH) are becoming the leading cause of liver-related morbidity and mortality worldwide, and a primary indication for liver transplantation. The pathophysiology of NASH is multifactorial and not yet completely understood; however, innate immunity is a major contributing factor in which liver-resident macrophages (Kupffer cells) and recruited macrophages play a central part in disease progression. In this Review, we assess the evidence for macrophage involvement in the development of steatosis, inflammation and fibrosis in NASH. Read More

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http://dx.doi.org/10.1038/s41575-018-0082-xDOI Listing
November 2018
2 Reads

IL-12, IL-23 and IL-17 in IBD: immunobiology and therapeutic targeting.

Nat Rev Gastroenterol Hepatol 2018 Nov 26. Epub 2018 Nov 26.

Department of Gastroenterology, Addenbrooke's Hospital, Cambridge University Hospitals NHS Foundation Trust, Cambridge, UK.

IL-12 and IL-23 are closely related cytokines with important roles in the regulation of tissue inflammation. Converging evidence from studies in mice, human observational studies and population genetics supports the importance of these cytokines in the regulation of mucosal inflammation in the gut in particular. Ustekinumab, a therapeutic antibody targeting both cytokines is now widely licensed for the treatment of Crohn's disease, including in Europe, the USA, Canada and Japan, whilst agents targeting IL-23 specifically are in late-phase clinical trials. Read More

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http://www.nature.com/articles/s41575-018-0084-8
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http://dx.doi.org/10.1038/s41575-018-0084-8DOI Listing
November 2018
5 Reads

New approaches along the IBD course: diet, tight control and stem cells.

Nat Rev Gastroenterol Hepatol 2019 Feb;16(2):82-84

Division of Gastroenterology and Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, MA, USA.

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http://dx.doi.org/10.1038/s41575-018-0088-4DOI Listing
February 2019
17 Reads

Wnt-β-catenin signalling in liver development, health and disease.

Nat Rev Gastroenterol Hepatol 2019 Feb;16(2):121-136

Department of Liver and Gastrointestinal Diseases, Biodonostia Health Research Institute - Donostia University Hospital - University of the Basque Country (UPV/EHU), San Sebastian, Spain.

The canonical Wnt-β-catenin pathway is a complex, evolutionarily conserved signalling mechanism that regulates fundamental physiological and pathological processes. Wnt-β-catenin signalling tightly controls embryogenesis, including hepatobiliary development, maturation and zonation. In the mature healthy liver, the Wnt-β-catenin pathway is mostly inactive but can become re-activated during cell renewal and/or regenerative processes, as well as in certain pathological conditions, diseases, pre-malignant conditions and cancer. Read More

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http://www.nature.com/articles/s41575-018-0075-9
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http://dx.doi.org/10.1038/s41575-018-0075-9DOI Listing
February 2019
11 Reads

Control and dysregulation of redox signalling in the gastrointestinal tract.

Nat Rev Gastroenterol Hepatol 2019 Feb;16(2):106-120

Division of Gastroenterology and Hepatology, Department of Medicine and the Mucosal Inflammation Program, University of Colorado School of Medicine, Aurora, CO, USA.

Redox signalling in the gastrointestinal mucosa is held in an intricate balance. Potent microbicidal mechanisms can be used by infiltrating immune cells, such as neutrophils, to protect compromised mucosae from microbial infection through the generation of reactive oxygen species. Unchecked, collateral damage to the surrounding tissue from neutrophil-derived reactive oxygen species can be detrimental; thus, maintenance and restitution of a breached intestinal mucosal barrier are paramount to host survival. Read More

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http://www.nature.com/articles/s41575-018-0079-5
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http://dx.doi.org/10.1038/s41575-018-0079-5DOI Listing
February 2019
23 Reads

HIV and the liver.

Nat Rev Gastroenterol Hepatol 2019 Jan;16(1):1-2

Division of Liver Diseases, Icahn School of Medicine at Mount Sinai, New York City, NY, USA.

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http://dx.doi.org/10.1038/s41575-018-0085-7DOI Listing
January 2019
1 Read

Gastroparesis.

Nat Rev Dis Primers 2018 Nov 1;4(1):41. Epub 2018 Nov 1.

Department of Digestive Diseases, Policlinico S. Orsola-Malpighi, University of Bologna, Bologna, Italy.

Gastroparesis is a disorder characterized by delayed gastric emptying of solid food in the absence of a mechanical obstruction of the stomach, resulting in the cardinal symptoms of early satiety, postprandial fullness, nausea, vomiting, belching and bloating. Gastroparesis is now recognized as part of a broader spectrum of gastric neuromuscular dysfunction that includes impaired gastric accommodation. The overlap between upper gastrointestinal symptoms makes the distinction between gastroparesis and other disorders, such as functional dyspepsia, challenging. Read More

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http://dx.doi.org/10.1038/s41572-018-0038-zDOI Listing
November 2018
1 Read

Mechanosensing regulates liver growth.

Authors:
Hugh Thomas

Nat Rev Gastroenterol Hepatol 2018 Dec;15(12):718

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-018-0083-9DOI Listing
December 2018
1 Read

Enterocyte zonation along the intestinal villus reveals a division of labour.

Authors:
Iain Dickson

Nat Rev Gastroenterol Hepatol 2018 Dec;15(12):717

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-018-0080-zDOI Listing
December 2018
1 Read

Use of hepatitis C viraemic organs in kidney transplantation: a need to hit the pause button?

Nat Rev Gastroenterol Hepatol 2019 Jan;16(1):5-6

Department of Medicine, Division of Gastroenterology/Hepatology, University of California San Francisco, San Francisco, CA, USA.

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http://dx.doi.org/10.1038/s41575-018-0071-0DOI Listing
January 2019
1 Read

Decoding hepatocyte ploidy.

Authors:
Clemens Thoma

Nat Rev Gastroenterol Hepatol 2018 Nov;15(11):656

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-018-0076-8DOI Listing
November 2018
1 Read

A gut-brain neural connection for rapid nutrient sensing.

Authors:
Iain Dickson

Nat Rev Gastroenterol Hepatol 2018 Nov;15(11):655

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-018-0077-7DOI Listing
November 2018
1 Read

Author Correction: Hepatorenal syndrome.

Nat Rev Dis Primers 2018 Oct 15;4(1):33. Epub 2018 Oct 15.

Division of Gastroenterology and Hepatology, Mayo Clinic, Rochester, MN, USA.

The original version of this article omitted an initial from the name of contributing author Patrick S. Kamath, who was listed as Patrick Kamath. The article has now been corrected. Read More

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http://dx.doi.org/10.1038/s41572-018-0035-2DOI Listing
October 2018
6 Reads

Intestinal homeostasis is reliant on self-maintaining macrophages.

Authors:
Hugh Thomas

Nat Rev Gastroenterol Hepatol 2018 Nov;15(11):656-657

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-018-0074-xDOI Listing
November 2018
3 Reads

Personalized response prediction.

Authors:
Clemens Thoma

Nat Rev Gastroenterol Hepatol 2018 Nov;15(11):657

Nature Reviews Gastroenterology & Hepatology, .

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http://www.nature.com/articles/s41575-018-0072-z
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http://dx.doi.org/10.1038/s41575-018-0072-zDOI Listing
November 2018
7 Reads

You are what you eat: diet, health and the gut microbiota.

Nat Rev Gastroenterol Hepatol 2019 Jan;16(1):35-56

Immunology Department, Weizmann Institute of Science, Rehovot, Israel.

Since the renaissance of microbiome research in the past decade, much insight has accumulated in comprehending forces shaping the architecture and functionality of resident microorganisms in the human gut. Of the multiple host-endogenous and host-exogenous factors involved, diet emerges as a pivotal determinant of gut microbiota community structure and function. By introducing dietary signals into the nexus between the host and its microbiota, nutrition sustains homeostasis or contributes to disease susceptibility. Read More

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http://dx.doi.org/10.1038/s41575-018-0061-2DOI Listing
January 2019
1 Read

Apoptosis and necroptosis in the liver: a matter of life and death.

Nat Rev Gastroenterol Hepatol 2018 Dec;15(12):738-752

Department of Medicine III, Division of Gastroenterology, Hepatology and Hepatobiliary Oncology, University Hospital Rheinisch-Westfälische Technische Hochschule (RWTH) Aachen, Aachen, Germany.

Cell death represents a basic biological paradigm that governs outcomes and long-term sequelae in almost every hepatic disease condition. Acute liver failure is characterized by massive loss of parenchymal cells but is usually followed by restitution ad integrum. By contrast, cell death in chronic liver diseases often occurs at a lesser extent but leads to long-term alterations in organ architecture and function, contributing to chronic hepatocyte turnover, the recruitment of immune cells and activation of hepatic stellate cells. Read More

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http://www.nature.com/articles/s41575-018-0065-y
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December 2018
4 Reads

Chemoprevention of Barrett's oesophagus: a step closer with PPIs and aspirin.

Nat Rev Clin Oncol 2018 Dec;15(12):728-730

Section of Gastroenterology and Hepatology, Department of Medicine, Baylor College of Medicine, Houston, TX, USA.

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http://dx.doi.org/10.1038/s41571-018-0096-xDOI Listing
December 2018
1 Read

Senescence prevents regeneration after acute liver injury.

Authors:
Hugh Thomas

Nat Rev Gastroenterol Hepatol 2018 Oct;15(10):582

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-018-0068-8DOI Listing
October 2018
1 Read

Hepatorenal syndrome.

Nat Rev Dis Primers 2018 Sep 13;4(1):23. Epub 2018 Sep 13.

Division of Gastroenterology and Hepatology, Mayo Clinic, Rochester, MN, USA.

Hepatorenal syndrome (HRS) is a form of kidney function impairment that characteristically occurs in cirrhosis. Recent changes in terminology have led to acute HRS being referred to as acute kidney injury (AKI)-HRS and chronic HRS as chronic kidney disease (CKD)-HRS. AKI-HRS is characterized by a severe impairment of kidney function owing to vasoconstriction of the renal arteries in the absence of substantial abnormalities in kidney histology. Read More

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http://dx.doi.org/10.1038/s41572-018-0022-7DOI Listing
September 2018
7 Reads

Exploring human gastric cancers through organoid culture.

Authors:
Hugh Thomas

Nat Rev Gastroenterol Hepatol 2018 Oct;15(10):581

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-018-0066-xDOI Listing
October 2018
2 Reads

Wilson disease.

Nat Rev Dis Primers 2018 Sep 6;4(1):21. Epub 2018 Sep 6.

Section of Digestive Diseases and Transplantation and Immunology, Department of Medicine and Surgery, Yale University School of Medicine, New Haven, CT, USA.

Wilson disease (WD) is a potentially treatable, inherited disorder of copper metabolism that is characterized by the pathological accumulation of copper. WD is caused by mutations in ATP7B, which encodes a transmembrane copper-transporting ATPase, leading to impaired copper homeostasis and copper overload in the liver, brain and other organs. The clinical course of WD can vary in the type and severity of symptoms, but progressive liver disease is a common feature. Read More

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http://dx.doi.org/10.1038/s41572-018-0018-3DOI Listing
September 2018
3 Reads

Endocannabinoids counterbalance intestinal inflammation.

Authors:
Iain Dickson

Nat Rev Gastroenterol Hepatol 2018 Nov;15(11):656-657

Nature Reviews Gastroenterology & Hepatology, .

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http://dx.doi.org/10.1038/s41575-018-0067-9DOI Listing
November 2018
1 Read

Gut microbiome, microbial metabolites and the development of NAFLD.

Nat Rev Gastroenterol Hepatol 2018 Dec;15(12):719-720

NAFLD Research Center, Department of Medicine, La Jolla, CA, USA.

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http://dx.doi.org/10.1038/s41575-018-0058-xDOI Listing
December 2018
1 Read

Publisher Correction: Alcoholic liver disease.

Nat Rev Dis Primers 2018 Aug 28;4(1):18. Epub 2018 Aug 28.

University of Southern California Keck School of Medicine and Greater Los Angeles VA Healthcare System, Los Angeles, CA, USA.

The originally published article contained an error in figure 8 part a, in which cut off values for F3 fibrosis, cirrhosis, and screening for oesophageal varices and HCC were incorrectly presented as <8 kPa, <12.5 kPa and <20 kPa, respectively. These cut off values have been corrected in the HTML and PDF versions of the manuscript to >8 kPa for F3 fibrosis, >12. Read More

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http://dx.doi.org/10.1038/s41572-018-0021-8DOI Listing
August 2018
15 Reads