3 results match your criteria Glomerulonephritis Nonstreptococcal Associated With Infection

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Postinfectious glomerulonephritis.

Authors:
Neeraja Kambham

Adv Anat Pathol 2012 Sep;19(5):338-47

Department of Pathology, Stanford University Medical Center, Stanford, CA, USA.

Postinfectious glomerulonephritis (PIGN) is an immunologically mediated glomerular injury triggered by an infection. Poststreptococcal glomerulonephritis (PSGN) is a classic example of PIGN with diffuse proliferative and exudative glomerular histology, dominant C3 staining and subepithelial "humps." Only the nephritogenic streptococcal infections cause PSGN and susceptibility to develop PSGN depends on both host and microbial factors. Read More

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http://dx.doi.org/10.1097/PAP.0b013e31826663d9DOI Listing
September 2012
19 Reads

Acute glomerulonephritis.

Authors:
R N Srivastava

Indian J Pediatr 1999 Mar-Apr;66(2):199-205

Apollo Indraprastha Hospital, New Delhi.

Acute glomerulonephritis (AGN) manifests with abrupt onset of hematuria, facial edema, hypertension and impairment of renal function. The commonest form of AGN in developing countries is that following a beta hemolytic streptococcal infection where the glomerular injury is mediated by deposition of immune complexes. In the usual patient with moderately severe poststreptococcal AGN (PSAGN) the above-mentioned features are present However, gross or microscopic hematuria may be the only abnormality. Read More

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July 2000
14 Reads

Asymptomatic glomerulonephritis after nonstreptococcal upper respiratory infections.

Ann Intern Med 1979 Nov;91(5):697-702

Two hundred forty previously healthy military personnel with nonstreptococcal upper respiratory infections were prospectively studied to define the incidence and clinicopathologic characteristics of possible virus-associated glomerulonephritis. Nine patients without preceding streptococcal infection had erythrocyte casts on urinalysis and glomerulonephritis on biopsy. Of these nine, four had a reduction in total hemolytic complement and five had serologic evidence of infection with adenovirus, influenza A, or influenza B. Read More

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November 1979
12 Reads
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