68 results match your criteria Apraxia of Lid Opening


Oxybuprocaine for apraxia of lid opening in Parkinson's disease: A placebo-controlled pilot trial.

Mov Disord 2018 Mar 23;33(3):494-495. Epub 2017 Nov 23.

Clinical Research Center and Department of Neurology, National Hospital Organization Utano Hospital, Kyoto, Japan.

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http://dx.doi.org/10.1002/mds.27243DOI Listing
March 2018
10 Reads

Botulinum toxin for treating unilateral apraxia of eyelid opening in a patient with congenital myotonia.

Arq Bras Oftalmol 2017 Sep-Oct;80(5):330-331

Department of Ophthalmology, Rio Hortega University Hospital, Dulzaina St, Valladolid, Spain.

A 37-year-old female presented with severe apraxia of lid opening (ALO) affecting the right upper lid associated with Becker congenital myotonia (MC). The patient had a history of right upper lid ptosis for 25 years that was exacerbated over the previous month with severe incapacity to open her right eye. No other associated neurological or ophthalmic symptoms were observed. Read More

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http://dx.doi.org/10.5935/0004-2749.20170081DOI Listing
December 2017
13 Reads
0.440 Impact Factor

I-FP-CIT SPECT imaging in blepharospasm.

Rev Neurol (Paris) 2017 Dec 16;173(10):645-649. Epub 2017 Jun 16.

Neurology, CHU Caremeau, place du Professeur-Debré, 30000 Nîmes, France.

Introduction: Blepharospasm is a focal dystonia characterized by involuntary cocontraction of the eyelid protractors, causing spasmodic closure of the eyelids. Apraxia of eyelid opening is caused by an inability to initiate lid opening without paralytic abnormality. Some studies suggest that patients with either pure blepharospasm or blepharospasm associated with apraxia of eyelid opening are more prone to developing Parkinson's disease. Read More

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https://linkinghub.elsevier.com/retrieve/pii/S00353787163006
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http://dx.doi.org/10.1016/j.neurol.2017.05.006DOI Listing
December 2017
7 Reads

Frontalis suspension surgery to treat patients with blepharospasm and eyelid opening apraxia: long-term results.

J Neural Transm (Vienna) 2017 02 5;124(2):253-257. Epub 2016 Nov 5.

Department of Ear-, Nose and Throat, Universitätsmedizin Göttingen (UMG), Göttingen, Germany.

Blepharospasm may be accompanied by eyelid opening apraxia (EOA) reducing the efficacy of botulinum toxin (BT) therapy. The frontalis suspension operation (FSO) is then the only effective treatment option available. We want to report the first long-term results with FSO. Read More

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http://dx.doi.org/10.1007/s00702-016-1641-3DOI Listing
February 2017
18 Reads

Bilateral ptosis: Lesion in the oculomotor nuclei or supranuclear lesion?

NeuroRehabilitation 2015 ;36(3):323-7

Département des Neurosciences cliniques, Unité de Neuro-Réhabilitation Aigüe, Centre Hospitalier Universitaire Vaudois, Lausanne, Switzerland.

Background: Bilateral ptosis is a very interesting clinical challenge for doctors because of the multiple possible localizations of a lesion which can lead to this neurological sign.

Objectives: Through this case report, we aim to determine the difference between an apraxia of lid opening (ALO) with difficulty in initiating the act of lid elevation, in spite of adequate understanding, motor control and cranial nerve pathways, and a bilateral ptosis with a lesion in the oculomotor nucleus or blepharospasm.

Methods: The case report of a 50-year-old patient presenting bilateral ptosis and multiple ischemic lesions in the brainstem and bilateral frontal lobe lesions after the emergency removal of a large frontal tumor. Read More

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http://www.medra.org/servlet/aliasResolver?alias=iospress&am
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http://dx.doi.org/10.3233/NRE-151220DOI Listing
May 2016
7 Reads

Frontalis suspension surgery to treat patients with essential blepharospasm and apraxia of eyelid opening-technique and results.

Head Face Med 2014 Oct 22;10:44. Epub 2014 Oct 22.

ENT-Department, University of Göttingen Medical Center, Göttingen, Germany.

Introduction: We describe the results of 15 patients suffering from essential blepharospasm with apraxia of eyelid opening who underwent frontalis suspension surgery.

Material And Methods: Patients with apraxia of eyelid opening and unresponsive to botulinum toxin injections were studied. Bilateral frontalis suspension surgery was performed (sling operation) using polytetrafluoroethylene (Gore-Tex®) sutures. Read More

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http://www.head-face-med.com/content/pdf/1746-160X-10-44.pdf
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http://www.head-face-med.com/content/10/1/44
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http://dx.doi.org/10.1186/1746-160X-10-44DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4213496PMC
October 2014
51 Reads

Combination of blepharospasm and apraxia of eyelid opening: a condition resistant to treatment.

Acta Neurol Belg 2012 Mar 24;112(1):95-6. Epub 2012 Jan 24.

Parkinson's Clinic of Eastern Toronto and Movement Disorders Centre, 404-2863 Ellesmere Rd, Toronto, ON M1E5E9, Canada.

Blepharospasm is seen in many cases of Parkinsonism including progressive supranuclear palsy. These patients usually respond well to botulinum toxin, however some patients subsequently fail to respond to even higher doses of botulinum toxin after an initial good response. They should not be considered failure of treatment with botulinum toxin, as a significant number of these patients have underlying apraxia of eyelid opening in addition to blepharospasm, which may be the cause of failure to respond to botulinum toxin. Read More

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http://link.springer.com/content/pdf/10.1007/s13760-012-0019
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http://link.springer.com/10.1007/s13760-012-0019-z
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http://dx.doi.org/10.1007/s13760-012-0019-zDOI Listing
March 2012
3 Reads

Effects of varying subthalamic nucleus stimulation on apraxia of lid opening in Parkinson's disease.

J Neurol 2012 Sep 17;259(9):1944-50. Epub 2012 Feb 17.

Département de Neurologie, Centre Hospitalier Universitaire de Grenoble, Grenoble, France.

Apraxia of lid opening (ALO) is a non-paralytic inability to open the eyes or sustain lid elevation at will. The exact pathophysiological mechanisms underlying the syndrome are still unknown. ALO has been reported in patients with Parkinson's disease (PD) after subthalamic nucleus (STN) deep brain stimulation (DBS), suggesting a possible involvement of the basal ganglia. Read More

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http://dx.doi.org/10.1007/s00415-012-6447-0DOI Listing
September 2012
2 Reads

A case of Parkinson's disease with apraxia of lid opening and freezing of gait.

Authors:
Zhen-Fu Wang

Chin Med J (Engl) 2011 Nov;124(21):3600

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November 2011
3 Reads

[Clinical characteristics and treatment of dystonia].

Authors:
Takahiro Mezaki

Rinsho Shinkeigaku 2011 Jul;51(7):465-70

Department of Neurology, Sakakibara Hakuho Hospital.

Dystonia is defined as a syndrome of sustained muscle contractions, frequently causing twisting and repetitive movements, or abnormal postures. Its diagnosis is based on clinical characteristics. In dystonia, the pattern of abnormal posture or movement tends to be constant during the short term even if its severity fluctuates. Read More

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July 2011
2 Reads

Normal and abnormal lid function.

Authors:
Janet C Rucker

Handb Clin Neurol 2011 ;102:403-24

Departments of Neurology and Ophthalmology, The Mount Sinai Medical Center, New York, NY 10029, USA.

This chapter on lid function is comprised of two primary sections, the first on normal eyelid anatomy, neurological innervation, and physiology, and the second on abnormal eyelid function in disease states. The eyelids serve several important ocular functions, the primary objectives of which are protection of the anterior globe from injury and maintenance of the ocular tear film. Typical eyelid behaviors to perform these functions include blinking (voluntary, spontaneous, or reflexive), voluntary eye closure (gentle or forced), partial lid lowering during squinting, normal lid retraction during emotional states such as surprise or fear (startle reflex), and coordination of lid movements with vertical eye movements for maximal eye protection. Read More

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http://dx.doi.org/10.1016/B978-0-444-52903-9.00021-2DOI Listing
August 2011
2 Reads

The pre-supplementary and primary motor areas generate rhythm for voluntary eye opening and closing movements.

Tohoku J Exp Med 2010 Oct;222(2):97-104

Department of Ophthalmology and Visual Science, Tokyo Medical and Dental University Graduate School, Tokyo, Japan.

Blinking and opening/closing of the eyelid are considered to be different movements with independent control mechanisms. Apraxia of lid opening (ALO) is a clinical syndrome in which patients experience difficulty in opening their eyes voluntarily. Our previous study with fluorodeoxyglucose and positron emission tomography (PET) has suggested that functional impairments in the supplementary motor area (SMA) and the anterior cingulate gyrus may be involved in the pathophysiology of ALO. Read More

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October 2010
3 Reads

Visual signs and symptoms of progressive supranuclear palsy.

Clin Exp Optom 2011 Mar;94(2):150-60

Vision Sciences, Aston University, Birmingham, United Kingdom.

Progressive supranuclear palsy is a rare, degenerative brain disorder and the second most common syndrome in which the patient exhibits 'parkinsonism', that is, a variety of symptoms involving problems with movement. General symptoms include difficulties with gait and balance; the patient walking clumsily and often falling backwards. The syndrome can be difficult to diagnose and visual signs and symptoms can help to separate it from closely related movement disorders such as Parkinson's disease, multiple system atrophy, dementia with Lewy bodies and corticobasal degeneration. Read More

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http://dx.doi.org/10.1111/j.1444-0938.2010.00504.xDOI Listing
March 2011
3 Reads

Involuntary eyelid closure after STN-DBS: evidence for different pathophysiological entities.

J Neurol Neurosurg Psychiatry 2010 Sep 20;81(9):1002-7. Epub 2010 Jun 20.

Center of Neurology, Hertie Institute for Clinical Brain Research, and German Center for Neurodegenerative Diseases (DZNE), University of Tübingen, Tübingen, Germany.

Objective: Involuntary eyelid closure (IEC) may occur after deep brain stimulation of the subthalamic nucleus (STN-DBS) in Parkinson's disease (PD) and is often categorised as apraxia of lid opening (ALO), albeit the appropriateness of this term is under debate. To gain insight into the hitherto undefined pathophysiology of IEC after STN-DBS, we performed a comprehensive clinical and electrophysiological characterisation of lid function in a total of six PD patients.

Methods: The study was carried out in six PD patients who developed IEC after STN-DBS. Read More

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http://dx.doi.org/10.1136/jnnp.2009.196691DOI Listing
September 2010
2 Reads

Apraxia of lid opening mimicking ptosis in compound heterozygosity for A467T and W748S POLG1 mutations.

Mov Disord 2008 Jul;23(9):1286-8

Department of Neurology, University of Bonn, Bonn, Germany.

Patients harboring A467T and W748S POLG1 mutations present with a broad variety of neurological phenotypes, including cerebellar ataxia, progressive external ophthalmoplegia (PEO), myoclonus, epilepsy, and peripheral neuropathy. With exception of ataxia and myoclonus, movement disorders are not typical features of POLG1 associated disorders. We report on two affected siblings compound heterozygous for A467T and W748S mutations, one suffering from choreoathetosis and apraxia of lid opening due to focal eyelid dystonia that mimicked progression of ptosis, resulting in functional blindness. Read More

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http://doi.wiley.com/10.1002/mds.22135
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http://dx.doi.org/10.1002/mds.22135DOI Listing
July 2008
31 Reads

Upper eyelid myectomy in blepharospasm with associated apraxia of lid opening.

Am J Ophthalmol 2008 Mar 11;145(3):541-547. Epub 2008 Jan 11.

Moran Eye Center, University of Utah, Salt Lake City, Utah 84102, USA.

Purpose: To assess the impact of upper eyelid myectomy surgery on blepharospasm with associated apraxia of lid opening (ALO), dry eye, photophobia, and daily functioning in patients who are refractory to botulinum toxin treatment.

Design: Noncomparative, consecutive, interventional case series.

Methods: A retrospective chart review was performed to identify 100 consecutive patients beginning on January 1, 2000, who underwent upper eyelid myectomy surgery for blepharospasm and fulfilled the inclusion criteria. Read More

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http://dx.doi.org/10.1016/j.ajo.2007.10.017DOI Listing
March 2008
2 Reads

Exacerbation of blepharospasm associated with craniocervical dystonia after placement of bilateral globus pallidus internus deep brain stimulator.

Mov Disord 2008 Feb;23(3):454-6

Center for Facial Appearances, Salt Lake City, Utah 84102, USA.

To report a case of exacerbation of blepharospasm after bilateral globus pallidus internus (GPi) deep brain stimulator (DBS) placement. A 69-year-old male presented after bilateral GPi DBS placement for blepharospasm and craniocervical dystonia with worsening eyelid spasms and associated apraxia of lid opening (ALO). Numerous attempts to adjust DBS parameters were ineffective. Read More

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http://doi.wiley.com/10.1002/mds.21889
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http://dx.doi.org/10.1002/mds.21889DOI Listing
February 2008
4 Reads

Apraxia of lid opening.

Br J Ophthalmol 2007 Jul;91(7):854

Eye Department, Worthing Hospital, Worthing BN11 2DH, UK.

We describe eyelid movement abnormalities in an 80-year-old man with apraxia of lid opening (ALO), resulting from involuntary levator palpebrae inhibition (ILPI) and pretarsal orbicularis oculi (OO) contraction. He was unable to open his lids at will following closure. Attempted eye opening resulted in forceful contraction of the frontalis muscle, backward thrusting of the head and lengthened lid closure. Read More

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http://bjo.bmj.com/cgi/doi/10.1136/bjo.2007.124040
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http://dx.doi.org/10.1136/bjo.2007.124040DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1955626PMC
July 2007
9 Reads

Dystonia redefined as central non-paretic loss of control of muscle action: a concept including inability to activate muscles required for a specific movement, or 'negative dystonia'.

Authors:
Takahiro Mezaki

Med Hypotheses 2007 4;69(6):1309-12. Epub 2007 Jun 4.

Department of Neurology, Sakakibara Hakuho Hospital, 5630 Sakakibara-cho, Tsu City, Mie 514-1251, Japan.

Dystonia is defined as a syndrome of sustained muscle contractions, frequently causing twisting and repetitive movements, or abnormal postures. Although this definition comprises an essential feature of dystonia, the clinical observation indicates that there is an additional aspect of dystonia; failure to adequately activate muscles required for specific movement, exemplified by the lack of contractions of the levator palpebrae superioris muscles in apraxia of lid opening, as well as by inability to activate appropriate muscles in cervical dystonia or in the paretic form of writer's cramp, and possibly by dropped head syndrome or camptocormia seen in parkinsonian patients without apparent truncal dystonia or rigidity. Taking this "negative dystonia" into consideration, the author proposes a revised definition of dystonia as a symptom characterized by the central non-paretic loss of voluntary control of muscle activities, which may result in either excessive or deficient contractions of muscles, frequently causing twisting and repetitive movements, limitation of movements, or abnormal postures. Read More

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http://dx.doi.org/10.1016/j.mehy.2007.03.014DOI Listing
January 2008
10 Reads

Apraxia of eyelid opening: clinical features and therapy.

Authors:
E Kerty K Eidal

Eur J Ophthalmol 2006 Mar-Apr;16(2):204-8

Department of Neurology, Rikshospitalet, University Hospital, Oslo, Norway.

Purpose: Botulinum toxin injection is the treatment of choice in cases of benign essential blepharospasm. However, about 10% of the patients do not get sufficient effect from this treatment, and many of them have concomitant apraxia of lid opening.

Methods: Over a 3-year period we treated 12 patients. Read More

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July 2006
5 Reads

[A 54-year-old man with familial parkinsonism, gaze palsy, and dementia].

No To Shinkei 2005 Dec;57(12):1104-12

Department of Neurology, Juntendo University School of Medicine, 2-1-1 Hongo, Bunkyo, Tokyo, Japan.

We report a Japanese man with familial parkinsonism who died at age 54. His younger brother, his mother, the mother's 4 brothers, and their mother were also affected with similar parkinsonism. The patient had had nystagmus since adolescence. Read More

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December 2005
2 Reads

[A 65-year-old man with rigid-bradykinetic parkinsonism, vertical gaze palsy, difficulty of eye-lid opening, and marked pseudo-bulbar palsy].

No To Shinkei 2005 Jan;57(1):73-86

Department of Neurology, Juntendo University School of Medicine, Tokyo, Japan.

We report a 65-year-old man with rigid-bradykinetic parkinsonism, vertical gaze palsy, difficulty in eye-lid opening, and marked pseudo-bulbar palsy. He felt difficulty of it, hand movement at 59 years old. When he was 60 years old, monotonous speech and slowness of movement appeared. Read More

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January 2005
24 Reads

[Blepharospasm and blepharocolysis. Different sides of the same coin].

Rev Neurol 2005 Mar 1-15;40(5):298-302

Servicio de Neurofisiología, Hospital General Universitario Gregorio Marañón, Madrid, Spain.

Introduction: The eyelid movements are mediated mainly by the orbicularis oculi (OO) and the levator palpebrae superioris (LPS) muscles. Dissociated upper lid functions exhibit different counterbalanced action of these muscles, and in blinking they show a strictly reciprocal innervation. The disturbance of this close LPS-OO relationship likely leads to many of the central lid movement disorders. Read More

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December 2005
2 Reads

Improvement of apraxia of lid opening by STN-stimulation in a 70-year-old patient with Parkinson's disease. A case report.

Minim Invasive Neurosurg 2004 Feb;47(1):58-60

Department of Neurology, Saarland University, Homburg/Saar, Germany.

We report on a 70-year-old female patient with Parkinson's disease, who showed an improvement of a preexisting apraxia of lid opening on electrical impulses, so-called deep brain stimulation (DBS) delivered to the subthalamic nucleus (STN). This was not described by any other authors before. Up to now, the appearance of apraxia of lid opening was observed only as a side effect after deep brain stimulation in the nucleus subthalamicus. Read More

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http://dx.doi.org/10.1055/s-2003-812466DOI Listing
February 2004
2 Reads

Apraxia of lid opening: dose-dependent response to carbidopa-levodopa.

Pharmacotherapy 2004 Mar;24(3):401-3

School of Pharmacy, Loma Linda University, Loma Linda, California, USA.

A 67-year-old woman with an 8-year history of Parkinson's disease and Lewy body dementia experienced difficulty in opening her eyelids (apraxia of lid opening [ALO]); she could close them without difficulty. This problem emerged 2 weeks after the patient's dosage of carbidopa 50 mg-levodopa 200 mg 3 times/day was decreased to twice/day. Two weeks after the onset of ALO the patient visited her physician, who suspected carbidopa-levodopa of causing the problem; the drug was discontinued. Read More

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March 2004
1 Read

Eyelid movements in health and disease. The supranuclear impairment of the palpebral motility.

Neurophysiol Clin 2004 Feb;34(1):3-15

Service of Clinical Neurophysiology, Hospital General Universitario Gregorio Marañón, c/ Dr. Esquerdo, 46, 28007 Madrid, Spain.

The eyelid movements are mediated mainly by the orbicularis oculi (OO) and the levator palpebrae superioris (LPS) muscles. Dissociated upper lid functions exhibit different counterbalanced action of these muscles, and in blinking they show a strictly reciprocal innervation. The disturbance of this close LPS-OO relationship likely leads to many of the central lid movement disorders. Read More

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http://dx.doi.org/10.1016/j.neucli.2004.01.002DOI Listing
February 2004
4 Reads

The effects of levodopa on apraxia of lid opening: A case report.

Neurology 2004 Mar;62(5):830-1

Department of Neurology, Nagoya University Graduate School of Medicine, Japan.

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March 2004
5 Reads

Glucose hypometabolism in medial frontal cortex of patients with apraxia of lid opening.

Graefes Arch Clin Exp Ophthalmol 2003 Jul 18;241(7):529-534. Epub 2003 Jun 18.

Positron Medical Center, Tokyo Metropolitan Institute of Gerontology, Tokyo, Japan.

Purpose: To determine whether regional cerebral functional abnormalities exist in patients with apraxia of lid opening (ALO).

Methods: Cerebral glucose metabolism was examined by positron emission tomography (PET) in 11 patients (8 women and 3 men, age 48-69 years); 10 with ALO accompanied by blepharospasm and 1 patient with pure ALO. Eleven normal volunteers (6 women and 5 men, age 45-66 years) were examined as controls. Read More

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http://dx.doi.org/10.1007/s00417-003-0682-0DOI Listing
July 2003
1 Read

Meige syndrome with apraxia of lid opening after the discontinuation of sulpiride treatment.

Pharmacopsychiatry 2002 Jul;35(4):155-6

Department of Psychiatry and Neurosciences, Hiroshima University School of Medicine, 1-2-3 Kasumi, Minami-ku, Hiroshima 734-8551, Japan.

We report a case of Meige syndrome with apraxia of lid opening that lasted for about seven months after discontinuation of sulpiride treatment. To our knowledge, this is the first report demonstrating that Meige syndrome with apraxia of lid opening is induced by sulpiride, and that the condition persists. Read More

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http://dx.doi.org/10.1055/s-2002-33198DOI Listing
July 2002
1 Read

Botulinum toxin improves lid opening delays in blepharospasm-associated apraxia of lid opening.

Neurology 2002 Jun;58(12):1843-6

Centre de recherche interdisciplinaire en réadaptation du Montréal Métropolitain, Institut de Réadaptation de Montréal, Qué H3S 2J4, Canada.

Lid movement and EMG of the orbicularis oculi (OOc) were analyzed in 10 patients with apraxia of lid opening associated with blepharospasm before and after botulinum toxin treatment. The latencies to onset and to complete the eye opening and the time during which eye opening was sustained were studied in relation to OOc activity and compared with control values obtained in 12 healthy subjects. Following treatment there was an improvement of all lid opening measurements, a decrease of the abnormally prolonged OOc activity, and a reduction of the functional disability. Read More

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June 2002
1 Read

Botulinum toxin therapy for apraxia of lid opening.

Ann N Y Acad Sci 2002 Apr;956:482-3

Neuro-ophthalmology Section, Centre Hospitalier de l'Université de Montréal (Hôtel-Dieu), Montréal, Québec, Canada.

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April 2002
3 Reads

Prolonged orbicularis oculi activity: a major factor in apraxia of lid opening.

Neurology 2001 Sep;57(6):1013-8

Centre de Recherche Interdisciplinaire en Réadaptation, Institut de Réadaptation de Montréal, Canada.

Background: Apraxia of lid opening (ALO) is a nonparalytic inability to open the eyes at will in the absence of visible contraction of the orbicularis oculi (OOc) muscle.

Objective: To test the hypothesis that sustained activity of the OOc is a major factor in the difficulty in opening the eyes in this condition.

Methods: Lid movement detected in an electromagnetic field and electromyogram activity of the septal and pretarsal portions of the OOc were recorded in 12 healthy control subjects and 12 patients with a clinical diagnosis of ALO. Read More

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September 2001
1 Read

Apraxia of lid opening associated with hydrocephalus.

Eur Neurol 2001 ;45(1):53-4

Department of Neurology, College of Medicine, Seoul National University, Seoul, Korea.

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http://dx.doi.org/10.1159/000052091DOI Listing
March 2001
3 Reads

Apraxia of lid opening is alleviated by pallidal stimulation in a patient with Parkinson's disease.

Eur J Neurol 2000 May;7(3):337-40

Department of Neurosurgery, Kumamoto University Medical School, Japan.

Apraxia of lid opening (ALO) is a syndrome characterized by a non-paralytic inability to open the eyes at will in the absence of visible contraction of the orbicularis oculi muscle. Here we report that globus pallidus internus deep brain stimulation on the right side markedly alleviates ALO as well as gait freezing in a patient with Parkinson's disease. Read More

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May 2000
1 Read

Isolated so-called apraxia of eyelid opening: report of 10 cases and a review of the literature.

Eur Neurol 1998 ;39(4):204-10

Institute of Neurology of the University of Bari, Italy.

So-called apraxia of eyelid opening (scAEO) has been described chiefly in the context of extrapyramidal disorders. We described 10 new patients with scAEO developing in the absence of any other CNS sign and reviewed the 11 cases with isolated scAEO reported in the literature. Combining our patients and those from the literature, peak age at onset was in the 6th decade and there was a female preponderance of 2:1. Read More

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http://dx.doi.org/10.1159/000007935DOI Listing
August 1998
4 Reads

Apraxia of lid opening: a review.

Authors:
D Boghen

Neurology 1997 Jun;48(6):1491-4

Neuro-ophthalmology section, Hôtel-Dieu de Montréal Hospital, Québec, Canada.

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June 1997
3 Reads

Apraxia of eyelid opening after bilateral stereotaxic subthalamotomy.

J Neuroophthalmol 1997 Jun;17(2):122-3

Department of Neurology, Medical University Lübeck, Germany.

The case of a patient with apraxia of eyelid opening and blepharospasm occurring during the course of idiopathic torsion dystonia and previously treated with stereotaxic subthalamotomy is presented. The anatomic basis of this lid movement disorder is suggested to be located in the rostral brain stem. There was a considerable amelioration after treatment with trihexyphenidyl. Read More

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June 1997
2 Reads

[Progressive supranuclear palsy with macular degeneration--report of three cases].

Rinsho Shinkeigaku 1997 May;37(5):397-401

Hokuyukai Neurology Hospital.

Three cases of progressive supranuclear palsy (PSP) with decreased visual acuity were reported. Case 1 was a 50-year-old man, case 2 a 60-year old woman, and case 3 a 66-year-old woman. They all had vertical gaze palsy and unexplained falls, and were diagnosed as PSP by NINDS-SPSP clinical criteria. Read More

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May 1997
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Botulinum toxin treatment of apraxia of eyelid opening in progressive supranuclear palsy: report of two cases.

Arch Phys Med Rehabil 1997 May;78(5):525-9

Department of Neurorehabilitation, Ospedale S. Camillo, Alberoni-Venezia, Italy.

We report two patients, with postural instability and dystonic parkinsonism whose adjunctive disabling feature was blindness due to an inability to reopen the eyes after voluntary closure of the eyelids, as in apraxia of lid opening (ALO). Supranuclear downgaze paresis permitted the diagnosis of progressive supranuclear palsy (PSP) in one case. Electromyographic studies showed a loss of normal reciprocal inhibition between the levator palpebrae and the pretarsal portion of the orbicularis oculi, with a cocontraction of these two antagonist muscles. Read More

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May 1997
2 Reads

Disorders of facial motor function.

Authors:
D R Boghen

Curr Opin Ophthalmol 1996 Dec;7(6):48-52

Neurology Unit, Hôtel-Dieu de Montréal Hospital, Québec, Canada.

This review deals with a variety of disorders of facial movement. Recent publications on blepharospasm, facial spasm, facial myokymia, apraxia of lid opening and facial paralysis are referenced and discussed. In blepharospasm, carefully performed electromyographic studies reveal a variety of abnormal patterns of contraction of the oribicularis oculi and the levator palpebrae muscles confirming the clinical impression that blepharospasm is not a homogeneous disease. Read More

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December 1996
5 Reads

Ocular motility in Parkinson's disease.

J Pediatr Ophthalmol Strabismus 1996 May-Jun;33(3):144-7

Wilmer Ophthalmological Institute, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA.

Introduction: Parkinson's disease is associated with multiple abnormalities of both the afferent and efferent visual systems. Blepharospasm, paucity of blinking, apraxia of lid opening, visual neglect, reduced vergence, reduced upgaze, and blurred vision are reported findings in these patients. The association of these findings with the disease, and their duration, severity, and treatment have not been systematically investigated. Read More

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November 1996
2 Reads

[Difficulty in eye opening following left hemispheric infarction-- causative lesion and pathophysiology of abnormalities of the eye and eyelids movements].

Rinsho Shinkeigaku 1996 Apr;36(4):577-83

Department of Neurology, School of Medicine, Chiba University.

We reported a patient suffering from difficulty in eye opening following left hemispheric infarction. A 78-year-old left-handed woman with atrial fibrillation had an acute onset of right hemiparesis and difficulty in eye opening. On admission, mild disorientation, vertical gaze palsy, right homonymous hemianopia, anosognosia of right hemiparesis, agnosia of right side of the body and forced grasping reflex of the left hand were seen. Read More

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April 1996
4 Reads

Apraxia of lid opening.

Authors:
J Jankovic

Mov Disord 1995 Sep;10(5):686-7

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September 1995
4 Reads

Lid-opening apraxia.

Neurology 1995 Sep;45(9):1788-9; author reply 1789-90

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September 1995
4 Reads

Lid-opening apraxia.

Authors:
G J Gilbert

Neurology 1995 Sep;45(9):1788; author reply 1789-90

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September 1995
4 Reads

Botulinum A toxin for the so-called apraxia of lid opening.

Mov Disord 1995 Jul;10(4):525-6

Institutes of Neurology and Ophthalmology, University of Bari, Italy.

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http://dx.doi.org/10.1002/mds.870100425DOI Listing
July 1995
4 Reads

Lid opening apraxia is associated with medial frontal hypometabolism.

Mov Disord 1995 May;10(3):341-4

Department of Neurology, New York Hospital, New York, USA.

We used [18F]fluorodeoxyglucose with positron emission tomography to explore the functional basis of lid opening apraxia (LOA). We studied four patients with LOA without associated pyramidal or extrapyramidal motor dysfunction (mean age 65.5 +/- 4. Read More

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http://dx.doi.org/10.1002/mds.870100319DOI Listing
May 1995
1 Read

"Apraxia of lid opening," a focal eyelid dystonia: clinical study of 32 patients.

Authors:
P Krack M H Marion

Mov Disord 1994 Nov;9(6):610-5

Department of Neurology, University of Giessen, Germany.

We have seen 32 patients with "apraxia of lid opening" (ALO) in the following clinical settings: as an isolated condition (3 patients), idiopathic blepharospasm (BSP, 20 patients, including 4 familial cases), progressive supranuclear palsy (PSP, 7 patients), and dystonic parkinsonian syndrome (2 patients). Twenty-nine patients treated with botulinum toxin into the orbicularis oculi muscle were rated before and after treatment and 83% of the patients improved on a clinical scale. Best results were obtained with injections directed toward the junction of the preseptal and pretarsal parts of the palpebral orbicularis oculi. Read More

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http://dx.doi.org/10.1002/mds.870090605DOI Listing
November 1994
1 Read

[Asynchronous Bell's phenomenon in progressive supranuclear palsy].

Authors:
M Konagaya M Iida

No To Shinkei 1993 Aug;45(8):765-8

Department of Neurology, Suzuka National Hospital, Mie, Japan.

A 60-year-old man suffering from progressive supranuclear palsy showed asynchronous Bell's phenomenon and apraxia of eye-lid opening. He showed synchronous upward rotation of the eyeball at closing eyes. Although apraxia of eye-lid opening was observed in both sides, eye opening of the right side took longer duration than that of the opposite side. Read More

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August 1993
3 Reads