3,589 results match your criteria Adv Neurol[Journal]


Future and alternative therapies in Tourette syndrome.

Authors:
Roger Kurlan

Adv Neurol 2006 ;99:248-53

Department of Neurology, University of Rochester, Rochester, New York, USA.

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Behavioral neurosurgery.

Adv Neurol 2006 ;99:241-7

Department of Psychiatry, Cleveland Clinic Foundation, Cleveland, Ohio, USA.

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Behavior therapy: other interventions for tic disorders.

Adv Neurol 2006 ;99:234-40

Department of Psychology, University of Wisconsin-Milwaukee, Milwaukee, Wisconsin, USA.

This chapter reviewed other behavioral interventions for TS and discussed their efficacy. Clearly, the majority of behavioral interventions (habit/reversal excluded) have not been systematically evaluated enough to be deemed empirically supported monotherapies for TS. In addition to reviewing these interventions, recent advances in behavioral research on TS and a function-based model of treatment development and implementation were presented. Read More

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Behavioral treatments for tic suppression: habit reversal training.

Adv Neurol 2006 ;99:227-33

Department of Psychiatry and Biobehavioral Sciences, University of California-Los Angeles Los Angeles, California, USA.

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Treatment of aggression in Tourette syndrome.

Authors:
Cathy L Budman

Adv Neurol 2006 ;99:222-6

Department of Neurology, North Shore Hospital-Long Island Jewish Health System, Manhasset, New York, USA.

The largely non-specific and/or multiply-determined etiologies of aggressive symptoms in TS pose significant impediments to effective clinical management. At this time, treatment requires comprehensive neuropsychiatric assessment with a systematic prioritization of the different psychiatric co-morbidities that require intervention. Medication side effects, psychosocial stressors and environmental triggers must also be identified and addressed. Read More

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Treatment of co-morbid obsessive compulsive disorder, mood, and anxiety disorders.

Adv Neurol 2006 ;99:208-21

Department of Psychiatry, New York University School of Medicine, New York, New York, USA.

In Sumary, OCD, non-OCD anxiety disorders and mood disorders are common co-morbid psychiatric disorders are common co-morbid psychiatric disorders in clinically referred youth with TS. Emotional disorders such as anxiety and depression may be more problematic to the patient than the tics, with regard to overall illness severity and the potential for adverse outcomes, such as school and social failure. The emotional symptoms and co-morbid mood and anxiety disorders must be comprehensively identified because they will require specific intervention and treatment. Read More

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Attention deficit hyperactivity disorder, chronic tic disorder, and methylphenidate.

Adv Neurol 2006 ;99:197-207

Department of Psychiatry and Behavioral Science, State University of New York at Stony Brook, Stony Brook, New York 11794-8790, USA.

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Treatment of tics.

Adv Neurol 2006 ;99:191-6

Department of Pediatrics and Neurology, The University of Alabama at Birmingham, USA.

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Disruptive behavior problems in a community sample of children with tic disorders.

Adv Neurol 2006 ;99:184-90

Yale Child Study Center, Yale University School of Medicine, New Haven, Connecticut, USA.

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PANDAS: to treat or not to treat?

Authors:
Robert A King

Adv Neurol 2006 ;99:179-83

Tourette's/OCD Clinic, Yale Child Study Center, New Haven, Connecticut, USA.

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Autoimmunity and pediatric movement disorders.

Adv Neurol 2006 ;99:166-78

Division of Pediatric Neurology, Johns Hopkins Hospital, Baltimore, Maryland, USA.

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PANDAS: overview of the hypothesis.

Authors:
Gavin Giovannoni

Adv Neurol 2006 ;99:159-65

Department of Neuroinflammnation, Institute of Neurology, University College London, London, United Kingdom.

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Genes and Tourette syndrome: scientific, ethical, and social implications.

Authors:
Nancy M P King

Adv Neurol 2006 ;99:144-7

School of Medicine, University of North Carolina-Chapel Hill, Chapel Hill, NC, USA.

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Web-based consensus diagnosis for genetics studies of Gilles de la Tourette syndrome.

Adv Neurol 2006 ;99:136-43

Tourette Research Program, University of Utah, Division of Child and Adolescent Psychiatry, Salt Lake City, Utah, USA.

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A genome-wide scan and fine mapping in Tourette syndrome families.

Authors:
David L Pauls

Adv Neurol 2006 ;99:130-5

Psychiatric and Neurodevelopmental Genetics Unit, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts, USA.

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Functional neuroimaging of Tourette syndrome: advances and future directions.

Adv Neurol 2006 ;99:115-29

Division of Neuropsychiatry, New York Presbyterian Hospital, Department of Psychiatry, Weill Medical College of Cornell University, USA.

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Neurobiology of basal ganglia and Tourette syndrome: striatal and dopamine function.

Authors:
Roger L Albin

Adv Neurol 2006 ;99:99-106

Department of Neurology, University of Michigan School of Medicine, Ann Arbor, Michigan, USA.

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Neurobiology of basal ganglia and Tourette syndrome: basal ganglia circuits and thalamocortical outputs.

Authors:
Jonathan W Mink

Adv Neurol 2006 ;99:89-98

Department of Neurology, Neurobiology and Anatomy, Pediatrics, University of Rochester, New York, USA.

In summary, the scheme of basal ganglia function presented here, in conjunction with known features of anatomical organization and dopamine neurotransmission provides a hypothesis for the pathophysiology of tics. According to the hypothesis, clusters of striatal neurons (matrisomes) become abnormally active in inappropriate contexts leading to inhibition of GPi or SNpr neurons that would normally be active to supress unwanted movements. The inhibition of htese GPi or SNpr neurons would then disinhibit thalamocortical circuits. Read More

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Preclinical models relevant to Tourette syndrome.

Adv Neurol 2006 ;99:69-88

Department of Psychiatry, UCSD School of Medicine, La Jolla, California, USA.

Preclinical models, if used appropriately, can greatly accelerate the understanding of neuropsychiatric disorders. A number of animal models have predictive validity for antidopaminergic compounds that have traditionally been used to suppress motor and vocal tics in TS. Other models have been proposed that may have construct validity for specific hypotheses of infectious/immune and neural circuit etiologies of TS. Read More

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Tics associated with other disorders.

Adv Neurol 2006 ;99:61-8

Parkinson's Disease Center and Movement Disorders Clinic, Department of Neurology, Baylor College of Medicine, Houston, Texas, USA.

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Behavioral and affective disorders in Tourette syndrome.

Adv Neurol 2006 ;99:39-60

Department of Mental Health Sciences, St. Georges Hospital, London, United Kingdom.

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Attention deficit hyperactivity disorder: the childhood co-morbidity that most influences the disability burden in Tourette syndrome.

Adv Neurol 2006 ;99:17-21

Developmental Cognitive Neurology, Kenneldy Krieger Institute, Baltinore, Mnryland, USA.

ADHD is a complex co-morbidity, as it is heteregeneous in terms of the clinical subtypes, but also in terms of the circuits involved and the level of involvement within those circuits. Specially focusing on the relationship of ADHD to TS, this author's studies have added some neurobehavioral and some anatomical magnetic resonance imaging evidence suggesting the ADHD occurring with TS, appears like "garden-variety" ADHD, at least in the matched research sample. The similarities of neuroanatomical findings in the TS plus ADHD and ADHD groups and their distinctness from neuroanatomical findings in childdren with "pure TS provide some parallels to the observed similarity of functional deficit in TS plus ADHD and ADHD alone groups and the relative lack of functional deficits in children with TS only. Read More

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Phenomenology of tics and natural history of tic disorders.

Adv Neurol 2006 ;99:1-16

Child Study Center, Sterling Hall of Medicine, Yale University School of Medicine, New Haven, Connecticut, USA.

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Other demyelinating diseases.

Adv Neurol 2006 ;98:335-49

Department of Neurology, Wayne State University School of Medicine, Detroit, Michigan, USA.

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February 2006

Neuromyelitis optica.

Adv Neurol 2006 ;98:319-33

Department of Neurology, Mayo Clinic, Scottsdale, Arizona, USA.

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February 2006

Acute disseminated encephalomyelitis: distinction from multiple sclerosis and treatment issues.

Adv Neurol 2006 ;98:303-18

Department of Neurology, Mayo Clinic, Scottsdale, Arizona, USA.

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February 2006

Mitoxantrone in multiple sclerosis.

Adv Neurol 2006 ;98:293-302

Department of Neurology, Heinrich Heine University, Düsseldorf, Germany.

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February 2006