Constantin Vasilescu, MD.,PhD. - Policliclinica Serban Voda- Dept. of Neurology - MD.,PhD. Head

Constantin Vasilescu

MD.,PhD.

Policliclinica Serban Voda- Dept. of Neurology

MD.,PhD. Head

Bucharest, Europe | Romania

Main Specialties: Neurology

Additional Specialties: Neurology-Neuromuscular Diseases-Neurotoxicology-Clinical Neurophysiology-Clinical Neuropharmacology

ORCID logohttps://orcid.org/0000-0003-2090-048X


Top Author

Constantin Vasilescu, MD.,PhD. - Policliclinica Serban Voda- Dept. of Neurology - MD.,PhD. Head

Constantin Vasilescu

MD.,PhD.

Introduction

From 1954 to 1960 I have been a student of the Iuliu Hatieganu University of Medicine and Pharmacy, Cluj-Napoca, Romania, which I graduated in 1960 obtaining the degree of MD.From Jan 1959 to Oct 1961, I worked as Intern MD. at the University Clinics- Iuliu Hatieganu University of Medicine and Pharmacy, Cluj-Napoca, Romania. Since 1963 I have been working as Researcher at the Institute of Neurology, Romanian Academy (Professor Oscar Sager). From 1970 to 1995 I was Senior Researcher at Institute of Neurology & Psychiatry, Bucharest(Professors: Arthur Kreindler, Vlad Voiculescu) and in 1971 I obtained the degree: PhD.-Neurology.
In 1973-1974 I had a Docentenstipendium of the Alexander von Humboldt-Stiftung, Bonn, Germany (Praesident: Professor Dr. W. Heisenberg-Nobel Laureat).I worked at the Universitaet des Saarlandes, Abteilung fuer Stereotaktische Neurochirurgie, Neurochirurgische Klinik der Universitaet des Saarlandes, Homburg/Saar, under the scientific direction of Professor Dr.Dr.h.c.Rolf Hassler, Direktor, Max-Planck-Institut fuer Hirnforschung, Frankfurt/Main; Professor Dr.med.Gert Dieckmann).
In 1985 I was Chairman of the Section: Neuromuscular Diseases of the XIII-th World Congress of Neurology, Hamburg, Germany.
In 1990 I was Invited as International Guest at the 42nd Annual Meeting of the American Academy of Neurology, Miami Beach, by Professor Lewis P. Rowland, President of the American Academy of Neurology.
From 1995 to 2008 I was Chief of the Division of Clinical Neurophysiology at the National Institute of Neurology and Neurovacular Diseases, Bucharest, Romania (Professor Constantin Popa).
From Sep 1978 to Sep 1985 I was Senior Lecturer in Neuromuscular Diseases, Clinical Neurophysiology at University Clinic of Neurology, Dr.G.Marinescu, University Hospital, Bucharest: Chairman: Professor Vlad Voiculescu.
In Nov 1977 I received the Premiul (Premium) Gheorghe Marinescu, Romanian Academy, for my book: Viteza de conducere in nervii periferici in conditii normale si patologice. Editura Academiei Romane,1975
Constantin Vasilescu, MD.,PhD.

Primary Affiliation: Policliclinica Serban Voda- Dept. of Neurology - Bucharest, Europe , Romania

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View Constantin Vasilescu’s Resume / CV

Education

Sep 1964
Policlinica Serban Voda
MD , PhD
Department of Neurology

Publications

54Publications

1885Reads

304Profile Views

546PubMed Central Citations

Nerve excitability threshold in periphereal neuropathies

Authors:
C.Vasilescu

EEG Clin Neurophysiol 1983 Sep 56;S192(3)S192

Electroencephalography and Clinical Neurophysiology

DOI:10.1016/0013-4694(83)92381-7

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March 2019
6 Reads

Study of nucleic acids of blood lymphocytes in myasthenia gravis

REV.ROUM.MED.-NEUROL.PSYCHIAT.1975 Jan 1327(1):32

REV.ROUM.MED.-NEUROL.PSYCHIAT.(1975)

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December 2018
20 Reads

Neuromyotonia in alcoholic patients with gastrectomy.

Rom J Neurol Psychiatry 1995 Jan-Mar;33(1):3-6

Institute of Neurology and Psychiatry, Bucharest, Romania.

http://europepmc.org/abstract/med/7547369Four patients with severe muscle cramps were reported. In two of them the electrical signs of neuromyotonia were demonstrated. All the patients had undergone gastrectomy and all were immoderate consumers of alcohol. The neuromuscular disorder was reversible in all the patients.

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November 1995
16 Reads

Hereditary motor and sensory neuropathy. Clinical, genetic and electrodiagnostic studies.

Authors:
C Vasilescu

Rom J Neurol Psychiatry 1993 Jul-Dec;31(3-4):207-19

Institute of Neurology and Psychiatry, Bucharest, Romania.

http://europepmc.org/abstract/med/8011484Motor conduction velocity (MCV) alone cannot separate all the cases with types I and II of hereditary motor and sensory neuropathy (HMSN). However, sensory conduction velocity (SCV) in sural nerve distinctly separated types I and II of HMSN (8). As in most of our patients with HMSN sural nerve was unexcitable, we introduced SCV estimation in the distal segments of median and ulnar nerves. So, we studied 124 patients from families with typical and uncomplicated cases of Charcot-Marie-Tooth disease (CMTD): 68 patients had the "hypertrophic" form (type I) and 56 cases the "neuronal" form (type II). In this series, 16 patients had median MCV from 35 to 45 m/s, but SCV in the median and ulnar nerves separated 7 cases with type I and 9 patients with type II of HMSN. In conclusion, the type I and II of HMSN were delimited in most of the studied cases by MCV values in the median nerve. Nevertheless, in cases difficult to be classified either into type I or II of HMSN, i.e. patients with MCV from 35 to 45 m/s, only SCV measurements in distal median and ulnar nerves segments can distinctly separate type I (a slowing over 40% from control values), indicating that this type is underlain by a process of segmental demyelination (SD), which also was confirmed by sural nerve biopsy data. In our cases with HMSN type II MCV was either normal or slightly slowed. By contrast SCV was significantly slowed (a slowing of up to 30%). In addition, we have also a few cases of complicated HMSN forms (rare variants), associated with: 1. Isaacs' syndrome; 2. "denervation-reinnervation" muscle hypertrophy; and 3. Marinesco-Sjögren syndrome. 1. In 3 patients, the clinical features of "neuronal" form of CMTD were associated with fasciculation, cramps, impaired muscular relaxation, and percussion myotonia with respective electromyographic (EMG) accompaniments, which were responsive to valproic acid therapy. 2. On the other hand 3 patients developed in addition to the Isaacs' syndrome a significant "denervation-reinnervation" muscle hypertrophy, confirmed by both morphometric data on muscle biopsy and computed tomography. In these patients there was an increased proportion of type I and a decreased one either of type IIB or of type IIA fibres, without myotonic or dystrophic features.(ABSTRACT TRUNCATED AT 400 WORDS)

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July 1994
180 Reads
6 Citations

Chronic sensorimotor polyneuropathy associated with cerebello-extrapyramidal manifestations in a patient with tomaculous neuropathy.

Rom J Neurol Psychiatry 1993 Jul-Dec;31(3-4):279-81

Institute of Neurology and Psychiatry, Bucharest, Romania.

http://europepmc.org/abstract/med/8011490Clinical, electrophysiological and morphological (sural nerve and gastrocnemius muscle biopsies) data of a 57-year-old man with a chronic sensorimotor polyneuropathy of Charcot-Marie-Tooth type associated with a progressive cerebello-extrapyramidal syndrome are reported. Patient's family data were negative. Nerve structural and ultrastructural examinations revealed the morphological picture of a tomaculous neuropathy. The association of different clinical syndromes and the specificity of the tomaculous neuropathy are discussed.

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July 1994
34 Reads

CHARCOT-MARIE-TOOTH DISEASE: CLINICA,ELECTROPHYSIOLOGICAL AND NERVE/MUSCLE BIOPSY STUDIES

Edition: NEUROLOGY AND NEUROBIOLOGY, Volume 53Publisher: Alan R.Liss,Inc.,New York,USAEditor: Robert E.Lovelace, Howard K.Shapiro

NEUROLOGY AND NEUROBIOLOGY, Volume 53

CHARCOT-MARIE-TOOTH DISEASE: CLINICAL, ELECTROPHYSIOLOGICAL AND NERVE/MUSCLE BIOPSY STUDIES By Constantin Vasilescu, Marilena Alexianu and Andrei Dan Department of Neuromuscular Diseases, Institute of Neurology and Psychiatry, Bucharest, Romania Abstract Taking into account the electrophysiological findings correlated with sural nerve biopsy data, we can conclude that sensory conduction velocity (SCV) measurements in distal segments of the median and ulnar nerves may be validate as a fine discriminator (1,2) which clearly separate the cases difficult to classify as either type I or type II of Charcot-Marie-Tooth (CMT) disease. In our patients with type I of hereditary motor and sensory neuropathy (HMSN), SCV in the distal segments of the median and ulnar nerves slowed by 60% (2) from control values, indicating that this type of disease is underlain by a process of segmental demylination (SD), which was confirmed by sural nerve biopsy data. In the patients with type II of the HMSN, MCV was either normal or slightly slowed. By contrast SCV was significantly slowed by 29% (1,2) in the median and by 19% (1,2) in the ulnar nerves, which could be explain by sural nerve biopsy findings, namely secondary SD. References 1.Vasilescu C, Florescu A (1977) Motor and sensory conduction velocity in Charcot-Marie-Tooth disease. J Neurol 214:305-312 2.Vasilescu C (1993) Hereditary motor and sensory neuropathy. Clinical,genetic and electrodiagnostic studies.Rom J Neurol Psychiatry 31:207-219

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January 1990
116 Reads

Recent advances in the electrodiagnosis of peripheral neuropathies.

Neurol Psychiatr (Bucur) 1988 Jul-Sep;26(3):123-34

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December 1988
47 Reads

Valproic acid in Isaacs-Mertens syndrome.

Clin Neuropharmacol 1987 Jun;10(3):215-24

Department of Neuromuscular Diseases, Institute of Neurology and Psychiatry, Bucharest, Romania.

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June 1987
140 Reads
10 Citations
1.836 Impact Factor

Pseudomyopathic changes in peripheral neuropathies.

Neurol Psychiatr (Bucur) 1986 Oct-Dec;24(4):241-8

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February 1987
10 Reads

Muscle hypertrophy and a syndrome of continuous motor unit activity in prednisone-responsive Guillain-Barré polyneuropathy.

J Neurol 1984 ;231(5):276-9

http://europepmc.org/abstract/med/6520623The clinical, electrophysiological and morphological findings (light and electron microscopy of the sural nerve and gastrocnemius muscle) are reported in an unusual case of Guillain-Barré polyneuropathy with an association of muscle hypertrophy and a syndrome of continuous motor unit activity. Fasciculation, muscle stiffness, cramps, myokymia, impaired muscle relaxation and percussion myotonia, with their electromyographic accompaniments, were abolished by peripheral nerve blocking, carbamazepine, valproic acid or prednisone therapy. Muscle hypertrophy, which was confirmed by morphometric data, diminished 2 months after the beginning of prednisone therapy. Electrophysiological and nerve biopsy findings revealed a mixed process of axonal degeneration and segmental demyelination. Muscle biopsy specimen showed a marked predominance and hypertrophy of type-I fibres and atrophy, especially of type-II fibres.

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March 1985
127 Reads
49 Citations
3.377 Impact Factor

Delayed neuropathy after organophosphorus insecticide (Dipterex) poisoning: a clinical, electrophysiological and nerve biopsy study.

Journal of neurology, neurosurgery, and psychiatry, 1 May 1984, 47(5):543-548

http://europepmc.org/abstract/med/6736986Clinical, electrophysiological and histological findings in four patients accidentally poisoned with the organophosphorus insecticide Dipterex are reported. Three to five weeks after insecticide ingestion signs of a distal sensorimotor (preponderantly motor) neuropathy occurred. The patients complained of paraesthesia in the lower limbs, and two of them of very disagreeable pricking sensation in the soles of the feet, responsive to carbamazepine. They showed distal weakness mainly of the legs, footdrop , difficult gait and muscle hypotonia. Ankle jerk was abolished while other tendon reflexes persisted. Two months or even later after poisoning, knee jerks in all the patients were very brisk and more and less accompanied by other pyramidal signs (patellar clonus, abolishment of abdominal cutaneous reflexes, Babinski's sign). Clinical, electrophysiological and nerve biopsy data revealed a "dying-back" neuropathy in our patients. Distal muscle fatigue was confirmed by failure of neuromuscular transmission on repetitive nerve stimulation.

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May 1984

50 Citations

57 Reads

Multiminicore disease. Report of a case with histological and histoenzymatic peculiarities.

Neurol Psychiatr (Bucur) 1983 Oct-Dec;21(4):441-4

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February 1984
4 Reads

Sensorimotor neuropathy in a patient with Marinesco-Sjögren syndrome.

Eur Neurol 1983 ;22(3):222-6

http://europepmc.org/abstract/med/6305664A case of Marinesco-Sjögren syndrome, displaying the characteristic signs (ataxia, congenital cataract and mental retardation) is presented. Electrophysiological examination pointed to the presence of a sensorimotor peripheral neuropathy with an underlying mixed process of segmental demyelination and axonal degeneration (probably secondary). The sural nerve and gastrocnemius biopsy confirmed these data, showing that in this case the segmental demyelination process was accompanied by axonal degeneration.

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http://dx.doi.org/10.1159/000115563DOI Listing
August 1983
48 Reads
28 Citations
1.362 Impact Factor

Neuropathy after organophosphorus compounds poisoning.

Authors:
C Vasilescu
October 1982
36 Reads
6.807 Impact Factor

Electromyographic activity of neck muscles in patients affected by retrocollis under the influence of stimulation and coagulation of the prestitial nucleus of the midbrain.

Appl Neurophysiol 1981 ;44(5-6):291-301

www.karger.com/Article/Full Text/102211In 2 patients with retrocollis, the positive effects obtained by stereotactic high frequency coagulation of the prestitial nucleus on the tonic activity of the neck muscle were studied electromyographically and clinically. Before the stereotactic therapy, electromyographic recordings at rest were recorded from the musculi splenii. During stimulation of the prestitial nucleus at frequencies of 8, 25 and 50 Hz, the EMG activity at rest diminished in frequency and amplitude and ultimately disappeared. Passage from the EMG recordings at rest to electrical silence took place, in one case, though a rhythmic activity of potentials grouped at 11/s. After coagulation of the prestitial nucleus, the involuntary contractions of neck muscles disappeared and electrical silence occurred. On reexamination 1 month later, the positive results were still present.

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October 1982
71 Reads
9 Citations

Peripheral neuropathy with a syndrome of continuous motor unit activity.

J Neurol 1982 ;226(4):275-82

http://europepmc.org/abstract/med/6174709A chronic alcoholic who had also been submitted to partial gastrectomy developed a syndrome of continuous motor unit activity responsive to phenytoin therapy. There were signs of minimal distal sensorimotor polyneuropathy. Symptoms of the syndrome of continuous motor unit activity were fasciculation, muscle stiffness, myokymia, impaired muscular relaxation and percussion myotonia. Electromyography at rest showed fasciculation, doublets, triplets, multiplets, trains of repetitive discharges and myotonic discharges. Trousseau's and Chvostek's signs were absent. No abnormality of serum potassium, calcium, magnesium, creatine kinase, alkaline phosphatase, arterial blood gases and pH were demonstrated, but the serum Vitamin B12 level was reduced. The electrophysiological findings and muscle biopsy were compatible with a mixed sensorimotor polyneuropathy. Tests of neuromuscular transmission showed a significant decrement in the amplitude of the evoked muscle action potential in the abductor digiti minimi on repetitive nerve stimulation. These findings suggest that hyperexcitability and hyperactivity of the peripheral motor axons underlie the syndrome of continuous motor unit activity in the present case.

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May 1982
325 Reads
17 Citations
3.377 Impact Factor

Clinical and electrophysiological studies of carbon disulphide polyneuropathy.

J Neurol 1980 ;224(1):59-70

http://europepmc.org/abstract/med/6157800A clinical and electrophological study was performed on 30 patients with chronic carbon disulphide poisoning. Although the measurements of motor conduction velocity and of terminal latency were within the normal range in the subclinical stage, estimation of nerve excitability threshold showed distal motor hypoexcitability, thus proving a very effective means for the early detection of carbon disulphide polyneuropathy. The distal muscle fatigue found in 35% of patients was confirmed by the decrement (more than 10%) in the amplitude of muscle evoked potentials in the abductor digiti minimi muscle in response to repetitive stimulation of the ulnar nerve. Association of the above findings with the significant electrophysiological changes, viz., decrease in the amplitude of sensory evoked potentials on stimulation of the digital fibres, mild slowing of sensory conduction velocity in the peripheral nerves, and decrease in the amplitude of evoked potentials in the distal muscles, suggest that the carbon disulphide polyneuropathy would be underlain by a primary distal axonopathy.

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November 1980
98 Reads
37 Citations
3.377 Impact Factor

Clinical and electrophysiological study of neuropathy after organophosphorus compounds poisoning.

Arch Toxicol 1980 Feb;43(4):305-15

http://europepmc.org/abstract/med/7387390Clinical and electrophysiological examinations were performed on 12 patients with toxic neuropathy following accidental ingestion of alcohol polluted by triorthocresyl phosphate (TOCP). Concurrent PNS and CNS lesions were found in all patients. Two to three months after ingestion, five of them showed prevalent signs of mixed, sensorimotor polyneuropathy, especially motor and distal, and the electrophysiological data pointed to the mixed process of axonal degeneration and secondary demyelination. In two of these five patients in whom examinations were repeated 13 years after TOCP ingestion, there was a marked clinical and electrophysiological improvement of signs of PNS lesions. Improvement of signs of CNS lesions was very poor even after 13 years. Signs of CNS lesions prevailed in the remaining seven patients. The clinical picture resembled that in amyotrophic lateral sclerosis and the electrophysiological data suggested a neuronal and axonal degeneration. Apart from the 12 cases of TOCP neuropathy, we also studied two cases of poisoning with organophosphorus insecticides, Dipterex and Divipan, in which a pure motor form of neuropathy was found.

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February 1980
64 Reads
49 Citations
5.980 Impact Factor

Clinical value of early electrophysiological changes in sensory and motor fibres.

Neurol Psychiatr (Bucur) 1979 Jul-Sep;17(3):159-68

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January 1980
23 Reads

Triorthocresyl phosphate neuropathy.

Authors:
C Vasilescu

Arch Neurol 1979 Jul;36(7):455

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July 1979
22 Reads
7.419 Impact Factor

Polyneuropathy in Dupuytren's disease. (An electroneurographic study).

Neurol Psychiatr (Bucur) 1978 Jul-Sep;16(3):191-202

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December 1978
39 Reads

Ultrastructural study of the muscle in case of atypical hypertrophic myotonia.

Neurol Psychiatr (Bucur) 1977 Oct-Dec;15(4):281-6

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March 1978
3 Reads

Electroneurographic evidence of polyneuropathy in chronic liver disease.

Arch Psychiatr Nervenkr (1970) 1978 Mar;225(1):87-96

http://europepmc.org/abstract/med/206228An electroneurographic study performed on the peripheral nerves of 25 patients with severe cirrhosis following viral hepatitis showed slight slowing (P greater than 0.05) of motor conduction velocity (CV) and significant diminution (P less than 0.001) of sensory CV and mixed sensorimotor-evoked potentials, associated with a significant decrease in the amplitude of sensory evoked potentials. The slowing was about equal in the distal (digital) and in the proximal segments of the same nerve. A mixed axonal degeneration and segmental demyelination is presumed to explain these findings. The CV measurements proved helpful for an early diagnosis of hepatic polyneuropathy showing subjective symptoms in the subclinical stage.

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March 1978
35 Reads

[Entrapment neuropathy of the terminal branch of the ulnar nerve].

Rev Med Interna Neurol Psihiatr Neurochir Dermatovenerol Neurol Psihiatr Neurochir 1977 Jul-Sep;22(3):209-13

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January 1978
9 Reads

Motor and sensory conduction velocity in Charcot-Marie-Tooth disease.

J Neurol 1977 Mar;214(4):305-12

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March 1977
27 Reads
5 Citations
3.377 Impact Factor

Sensory and motor coduction in chronic carbon disulphide poisoning.

Authors:
C Vasilescu

Eur Neurol 1976 ;14(6):447-57

http://europepmc.org/abstract/med/991882The greater decrease of conduction velocity in sensory than in motor fibres of the peroneal, median and ulnar nerves (particularly in the digital segments) found in patients with chronic carbon disulphide poisoning, permitted the diagnosis of polyneuropathy to be made in the subclinical stage, even while the conduction in motor fibres was still within normal limits. A process of axonal degeneration is presumed to underlie occurrence of neuropathy consequent to carbon disulphide poisoning.

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https://www.karger.com/Article/FullText/114772
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http://dx.doi.org/10.1159/000114772DOI Listing
January 1977
127 Reads
38 Citations
1.362 Impact Factor

Electromyographic investigations in torticollis.

Appl Neurophysiol 1975 ;38(3):153-60

www.karger.com/Article/Full Text/102657In 13 patients with torticollis, the electromyographic (EMG) activity in the neck muscles at rest was investigated before, during and after stimulation of the H1 bundle (in horizontal torticollis) and inner part of oral ventral nucleus (in the rotatory form), as well as after coagulation. Three EMG types of torticollis-spasmodic, myoclonic and mixed - could be differentiated. During the 8-, 25- and 50-Hz stimulations, a diminution in the amplitude and frequency of muscle potentials, followed by electric silence, was most often obtained; in general, the electric silence was preceded by a rhythmic activity of grouped potentials. Following coagulation, no spontaneous EMG activity was any longer recorded in the neck muscles, in the majority of the cases.

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October 1976
83 Reads
5 Citations

Study of nucleic acids on blood lymphocytes in myasthenia gravis.

Rev Roum Neurol Psychiatr 1975 ;13(1):27-32

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August 1975
38 Reads

Motorische Leitgeschwindigkeit bei toxischen peripheren Neuropathien

Authors:
C. Vasilescu

Z. BEG-SMG 5 (1974) 177•181 © Georg Thieme-Verl., Stuttprt

Z. BEG-SMG 5 (1974) 177•181 © Georg Thieme-Verl

Motorische Leitgeschwindigkeit bei toxischen peripheren Neuropathien C. Vasilescu" Institut fUr Neurologie und Psychiatrie, Bukarest Motor conduction velocity in toxic peripheiai neuropathies The conduction velocity (c.v.) of the fastest conducting motor fibres in the peripheral nerves was studied in three groups of patients with toxic neuropathies. 1. Toxic polyneuritis by carbon disulphide: The c.v. in the median, ulnar and peroneal nerves in the patients of sub¬group I (with objective signs of mixed sensory-motor poly¬neuritis) was 43.0, 43.5 and 36.3 m/sec, while in those of subgroup n (only with subjective clinical symptoms) 49.1; 49.7 and 42.1 m/sec, respectively. 2. Toxic neuropathy by triorthocresyl-phosphate: The c.v. in the motor fibres of the peroneal, median and ulnar nerves, measured 1-3 month after ingestion was 33.6; 42.2 and 51.7 m/ sec, respectively. 3. Toxic neuropathy by lead poisoning: The c.v. in the motor fibres of the median, ulnar, peroneal and radial nerves in 50 patients with clinically and biochemically evi¬denced chronic lead poisoning was 55.6; 56.5; 46.1 and 44.8 m/sec, respectively. Damage of peripheral motoneurons was identified exclusively by c.v. measurements and EMG recordings. Key-Words: Motor conduction velocity - Toxic neuro¬pathies - Carbon disulphide intoxication - Triorthocresyl¬phosphate intoxication - Lead poisoning. Toxische periphere Neuropathien kommen in den modemen Industriezentren immer haufiger vor. Die klinische Untersuchung gestattet die Diagnose einer Neuropathie leider jedoch erst in einem ziemlich fortgeschrittenen Stadium. Daraus ergeben sich mit- Zusammenfassung Die Leitgeschwindigkeit in den schnellsten mctcnschen Fa~ sem der peripheren Nerven wurde bel drtti Patientengruppen mit chronischen Neuropathien untersucht, 1. Toxische Schwefelkohlenstoff-Polyneuritis: Patienten mit objektiven Symptomen einer gemischten sensorisch-metori .. schen Polyneuritis zeigten Verlangsamungen der NLG, tm N. medianus auf 43,0, im N. ulnaris auf 43,5 und lm N. fi¬bularis auf 36,3 m/sec. Patienten mit ausschlieBlich subjek~ tiven Beschwerden zeigten Verlangsamungen fill den N. me¬dianus auf 49,1, fur den N. ulnaris auf 49,7 und ftir den N. fibularis auf 42,1 m/sec. 2. Toxische Triorthokresylphosphat-Neuropathie: Die NLG in den motorischen Fasem lag 1-3 Monate nach der Ver:¬giftung fiir den N. fibularis bel 33,6, fUr den Medianus bed 42,2 und fiir den N. ulnaris bei 51,7 mJsec. 3. Toxische Blei-Neuropathie: Die NLG in den motorischen. Fasem lag bei chronischen Bleivergiftungen fUr den N. me¬dianus bei 55,6, ftiI: den N. ulnaris bei 56,5, flir den N. fibu¬laris bei 46,1 und den N. radialis bei 44,8 m/sec. Die Scha¬digung des peripheren motorischen Neurons wurde aus-schliefslich mit Hilfe der NLG und dem EMG festgestellt. unter erhebliche Nachteile fur die Behandlung, Mit modemen Untersuchungsmethoden, wie der Bestim .. mung der Nervenleitgeschwindigkeit (NLG) und der Elektromyographie (EMG) sind periphere Neuro¬pathien dagegen frillier aufzudecken. * Hnmboldt-Stipendiat an der Stereotaktischen Abteilung der Neurochirurgischen Universitatsklir'.k, Homburg/Saar

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January 1974

5 Citations

22 Reads

Motor nerve conduction velocity and electromyogram in chronic lead poisoning.

Authors:
C Vasilescu

Rev Roum Neurol 1973 ;10(3):221-6

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October 1973
24 Reads
16 Citations

Motor nerve conduction velocity and electromyogram in triorthocresyl-phosphate poisoning.

Authors:
C Vasilescu

Rev Roum Neurol 1972 ;9(5):345-50

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September 1973
15 Reads
7 Citations

Motor nerve conduction velocity and electromyogram in carbon disulphide poisoning.

Authors:
C Vasilescu

Rev Roum Neurol 1972 ;9(2):63-71

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November 1972
29 Reads
17 Citations

Clinical and EEG investigations in latent tetany.

Electroencephalogr Clin Neurophysiol 1971 Apr;30(4):364

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April 1971
17 Reads

Electroneurographic Evidence of Polyneuropathy in Chronic Liver Disease

Arch.Psychiat.Nervenkr.1978 Mar 225;87(1)96

Archiv fuer Psychiatrie und Nervenkrankheiten

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9 Reads

Study of conduction velocity in motor fibres of peripheral nerves in toxic polyneuritis produced by carbon disulphide.

Authors:
C Vasilescu

Electroencephalogr Clin Neurophysiol 1969 Dec;27(6):637

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10 Reads

[Study of the conduction velocity in motor fibers of the peripheral nerves in man in normal conditions].

Authors:
C Vasilescu

Stud Cercet Neurol 1968 ;13(6):447-54

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August 1969
6 Reads

Study of the conduction velocity of peripheral motor nerve fibers in some neurological diseases.

Authors:
C Vasilescu

Electroencephalogr Clin Neurophysiol 1967 Jun;22(6):578

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June 1967
11 Reads

[Paralysis with dyskalemia].

Authors:
C Vasilescu

Stud Cercet Neurol 1966 Jun;11(3):257-72

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June 1966
5 Reads

[Late scapuloperoneal form of progressive muscular dystrophy].

Neurol Psihiatr Neurochir 1965 Jan-Feb;10(1):21-7

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November 1965
3 Reads

Neuronal type of Charcot-Marie-Tooth Disease with a syndrome of continuous motor unit activity

J Neurol Sci. 1984 Jan 63;11(1):25

Journal of the Neurological Sciences

www.ncbi.nlm.nih.gov/pubmed/6699650

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November 0001

52 Citations

11 Reads