Publications

Two Cases of Occupational Contact Urticaria Caused by Percutaneous Sensitization to Parvalbumin.
Case Rep Dermatol 2015 May-Aug;7(2):227-32. Epub 2015 Aug 29.
Department of Integrative Medical Science for Allergic Disease, Toyoake, Japan ; Department of Dermatology, Fujita Health University School of Medicine, Toyoake, Japan.

Accelerated differentiation of melanocyte stem cells contributes to the formation of hyperpigmented maculae.
Exp Dermatol 2014 Sep;23(9):652-8
Research Laboratories, Nippon Menard Cosmetic Co. Ltd., Nagoya, Aichi, Japan; Department of Applied Cell and Regenerative Medicine, Fujita Health University School of Medicine, Toyoake, Aichi, Japan; Department of Dermatology, Fujita Health University School of Medicine, Toyoake, Aichi, Japan.

Comprehensive analysis of melanogenesis and proliferation potential of melanocyte lineage in solar lentigines.
J Dermatol Sci 2014 Mar 14;73(3):251-7. Epub 2013 Nov 14.
Department of Applied Cell and Regenerative Medicine, Fujita Health University School of Medicine, 1-98 Kutsukakecho, Toyoake, Aichi Japan.

Allergic contact dermatitis caused by phenylethyl resorcinol [4-(1-phenylethyl)-1,3-benzenediol], a skin-lightening agent in cosmetics.
Contact Dermatitis 2013 Nov;69(5):319-20
Department of Dermatology, Fujita Health University School of Medicine, Aichi, 470-1192, Japan; Department of Dermatology, Kurume University School of Medicine, Fukuoka, 830-0011, Japan.


The roles of P- and E-selectins and P-selectin glycoprotein ligand-1 in primary and metastatic mouse melanomas.
J Dermatol Sci 2011 Nov 5;64(2):99-107. Epub 2011 Aug 5.
Department of Dermatology, Nagasaki University Graduate School of Biomedical Sciences, Nagasaki, Japan.






CD19, a response regulator of B lymphocytes, regulates wound healing through hyaluronan-induced TLR4 signaling.
Am J Pathol 2009 Aug 2;175(2):649-60. Epub 2009 Jul 2.
Department of Dermatology, Nagasaki University Graduate School of Biomedical Sciences, 1-7-1 Sakamoto, Nagasaki, 852-8501, Japan.


CD19 regulates skin and lung fibrosis via Toll-like receptor signaling in a model of bleomycin-induced scleroderma.
Am J Pathol 2008 Jun 8;172(6):1650-63. Epub 2008 May 8.
Department of Dermatology, Nagasaki University Graduate School of Biomedical Sciences, 1-7-1 Sakamoto, Nagasaki, 852-8501, Japan.

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