Publications

Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells.
J Proteome Res 2017 Oct 30;16(10):3536-3546. Epub 2017 Aug 30.
Department of Chemistry, and ‡Nebraska Center for Integrated Biomolecular Communication, University of Nebraska-Lincoln , Lincoln, Nebraska 68588, United States.


Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.
Cancer Metab 2014 1;2:18. Epub 2014 Sep 1.
The Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, NE 68198, USA ; Department of Pathology and Microbiology, University of Nebraska Medical Center, Omaha, NE 68198, USA ; Department of Biochemistry and Molecular Biology, University of Nebraska Medical Center, Omaha, NE 68198, USA ; Department of Genetic Cell Biology and Anatomy, University of Nebraska Medical Center, Omaha, NE 68198, USA.


MUC1 mucin stabilizes and activates hypoxia-inducible factor 1 alpha to regulate metabolism in pancreatic cancer.
Proc Natl Acad Sci U S A 2012 Aug 6;109(34):13787-92. Epub 2012 Aug 6.
Eppley Institute for Research in Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, NE 68198, USA.

Differential expression of metabolic genes in tumor and stromal components of primary and metastatic loci in pancreatic adenocarcinoma.
PLoS One 2012 7;7(3):e32996. Epub 2012 Mar 7.
The Eppley Institute for Cancer and Allied Diseases, University of Nebraska Medical Center, Omaha, Nebraska, United States of America.


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