Sichuan Xi

Sichuan Xi

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Sichuan Xi

Sichuan Xi

Publications by authors named "Sichuan Xi"

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42Publications

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Decrease in Lymphoid Specific Helicase and 5-hydroxymethylcytosine Is Associated with Metastasis and Genome Instability.

Theranostics 2017 5;7(16):3920-3932. Epub 2017 Sep 5.

Key Laboratory of Carcinogenesis and Cancer Invasion (Ministry of Education), Xiangya Hospital, Central South University, Changsha, Hunan, 410008 China.

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http://dx.doi.org/10.7150/thno.21389DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5667415PMC
June 2018

Deregulation of UBE2C-mediated autophagy repression aggravates NSCLC progression.

Oncogenesis 2018 Jun 13;7(6):49. Epub 2018 Jun 13.

Cancer Research Institute, Binzhou Medical University Hospital, 256603, Binzhou, P.R. China.

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http://www.nature.com/articles/s41389-018-0054-6
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http://dx.doi.org/10.1038/s41389-018-0054-6DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC6002383PMC
June 2018

Sodium selenite attenuates lung adenocarcinoma progression by repressing SOX2-mediated stemness.

Cancer Chemother Pharmacol 2018 05 15;81(5):885-895. Epub 2018 Mar 15.

Cancer Research Institute, Binzhou Medical University Hospital, No. 661 Huanghe Street, Binzhou, 256600, Shandong, People's Republic of China.

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http://dx.doi.org/10.1007/s00280-018-3561-4DOI Listing
May 2018

IKKα inactivation promotes Kras-initiated lung adenocarcinoma development through disrupting major redox regulatory pathways.

Proc Natl Acad Sci U S A 2018 01 8;115(4):E812-E821. Epub 2018 Jan 8.

Cancer and Inflammation Program, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Frederick, MD 21702;

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http://dx.doi.org/10.1073/pnas.1717520115DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5789942PMC
January 2018

Disruption of SHH signaling cascade by SBE attenuates lung cancer progression and sensitizes DDP treatment.

Sci Rep 2017 05 15;7(1):1899. Epub 2017 May 15.

Cancer Research Institute, Binzhou Medical University hospital, 256600, Binzhou, P.R. China.

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http://dx.doi.org/10.1038/s41598-017-02063-xDOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5432500PMC
May 2017

Repression of YAP by NCTD disrupts NSCLC progression.

Oncotarget 2017 Jan;8(2):2307-2319

Cancer Research Institute, Binzhou Medical University Hospital, Binzhou 256603, P.R. China.

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http://dx.doi.org/10.18632/oncotarget.13668DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5356801PMC
January 2017

As a novel p53 direct target, bidirectional gene HspB2/αB-crystallin regulates the ROS level and Warburg effect.

Biochim Biophys Acta 2014 Jul 22;1839(7):592-603. Epub 2014 May 22.

Cancer Research Institute, Central South University, Changsha, Hunan 410078, China; Center for Molecular Imaging, Central South University, Changsha, Hunan 410078, China; Key Laboratory of Carcinogenesis and Cancer Invasion, Ministry of Education, Hunan 410078, China; Key Laboratory of Carcinogenesis, Ministry of Health, Hunan 410078, China. Electronic address:

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http://dx.doi.org/10.1016/j.bbagrm.2014.05.017DOI Listing
July 2014

Cigarette smoke mediates epigenetic repression of miR-487b during pulmonary carcinogenesis.

J Clin Invest 2013 Mar 15;123(3):1241-61. Epub 2013 Feb 15.

Thoracic Oncology Section, Surgery Branch, Center for Cancer Research, National Cancer Institute, Bethesda, Maryland 20892, USA.

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http://dx.doi.org/10.1172/JCI61271DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3582115PMC
March 2013

Lymphoid-specific helicase (HELLS) is essential for meiotic progression in mouse spermatocytes.

Biol Reprod 2011 Jun 23;84(6):1235-41. Epub 2011 Feb 23.

Center for Animal Transgenesis and Germ Cell Research, School of Veterinary Medicine, University of Pennsylvania, Kennett Square, USA.

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http://dx.doi.org/10.1095/biolreprod.110.085720DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3099587PMC
June 2011

Lsh, chromatin remodeling family member, modulates genome-wide cytosine methylation patterns at nonrepeat sequences.

Proc Natl Acad Sci U S A 2011 Apr 22;108(14):5626-31. Epub 2011 Mar 22.

Basic Science Program, Laboratory of Cancer Prevention, Advanced Biomedical Computing Center, and Data Management Services, Science Applications International Corporation Frederick, Inc, National Cancer Institute Frederick, Frederick, MD 21702, USA.

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http://dx.doi.org/10.1073/pnas.1017000108DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3078362PMC
April 2011

Cigarette smoke induces C/EBP-β-mediated activation of miR-31 in normal human respiratory epithelia and lung cancer cells.

PLoS One 2010 Oct 29;5(10):e13764. Epub 2010 Oct 29.

Thoracic Oncology Section, Surgery Branch, Center for Cancer Research, National Cancer Institute, Bethesda, Maryland, United States of America.

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http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0013764PLOS
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2966442PMC
October 2010

Lsh mediated RNA polymerase II stalling at HoxC6 and HoxC8 involves DNA methylation.

PLoS One 2010 Feb 11;5(2):e9163. Epub 2010 Feb 11.

Laboratory of Cancer Prevention, SAIC-Frederick, National Cancer Institute, Frederick, Maryland, United States of America.

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http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0009163PLOS
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2820093PMC
February 2010

Lsh participates in DNA methylation and silencing of stem cell genes.

Stem Cells 2009 Nov;27(11):2691-702

Laboratory of Cancer Prevention, SAIC-Frederick, National Cancer Institute, Frederick, MD 21701, USA.

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http://dx.doi.org/10.1002/stem.183DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3327128PMC
November 2009

DNA hypomethylation caused by Lsh deletion promotes erythroleukemia development.

Epigenetics 2008 May-Jun;3(3):134-42. Epub 2008 May 9.

Laboratory of Cancer Prevention, SAIC-FCRDC, Basic Research Program, National Cancer Institute, Frederick, Maryland, USA.

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3113485PMC
http://dx.doi.org/10.4161/epi.3.3.6252DOI Listing
October 2008

The see-saw of differentiation: tipping the chromatin balance.

Mol Interv 2008 Feb;8(1):15-8, 2

Laboratory of Cancer Prevention, SAIC-FCRDC, National Cancer Institute, Frederick, MD 21701, USA.

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http://dx.doi.org/10.1124/mi.8.1.4DOI Listing
February 2008

Lsh controls Hox gene silencing during development.

Proc Natl Acad Sci U S A 2007 Sep 28;104(36):14366-71. Epub 2007 Aug 28.

Laboratory of Cancer Prevention, SAIC-Frederick, National Cancer Institute, Frederick, MD 21702-1201, USA.

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http://dx.doi.org/10.1073/pnas.0703669104DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1955459PMC
September 2007

Mutant epidermal growth factor receptor (EGFRvIII) contributes to head and neck cancer growth and resistance to EGFR targeting.

Clin Cancer Res 2006 Sep;12(17):5064-73

Department of Otolaryngology, and University of Pittsburgh Cancer Institute Biostatistics, University of Pittsburgh, Pennsylvania 15213, USA.

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http://dx.doi.org/10.1158/1078-0432.CCR-06-0913DOI Listing
September 2006

Decreased STAT1 expression by promoter methylation in squamous cell carcinogenesis.

J Natl Cancer Inst 2006 Feb;98(3):181-9

Department of Otolaryngology, University of Pittsburgh School of Medicine, Cancer Institute, Pittsburgh, PA, USA.

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http://dx.doi.org/10.1093/jnci/djj020DOI Listing
February 2006

Lsh is involved in de novo methylation of DNA.

EMBO J 2006 Jan 5;25(2):335-45. Epub 2006 Jan 5.

Laboratory of Cancer Prevention, SAIC-FCRDC, Basic Research Program, National Cancer Institute, Frederick, MD 21701-1201, USA.

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http://dx.doi.org/10.1038/sj.emboj.7600925DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1383509PMC
January 2006

Erythropoietin-mediated activation of JAK-STAT signaling contributes to cellular invasion in head and neck squamous cell carcinoma.

Oncogene 2005 Jun;24(27):4442-9

Department of Otolaryngology--Head and Neck Surgery, The Eye & Ear Institute, University of Pittsburgh Cancer Institute, Pittsburgh, PA 15213, USA.

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http://dx.doi.org/10.1038/sj.onc.1208635DOI Listing
June 2005

In vivo antitumor efficacy of STAT3 blockade using a transcription factor decoy approach: implications for cancer therapy.

Oncogene 2005 Feb;24(6):970-9

Department of Otolaryngology, University of Pittsburgh School of Medicine, Pittsburgh, PA, USA.

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http://dx.doi.org/10.1038/sj.onc.1208316DOI Listing
February 2005

Mutation of p53 in head and neck squamous cell carcinoma correlates with Bcl-2 expression and increased susceptibility to cisplatin-induced apoptosis.

Head Neck 2004 Oct;26(10):870-7

Department of Otolaryngology, University of Pittsburgh School of Medicine and the University of Pittsburgh Cancer Institute, Pittsburgh, Pennsylvania 15213, USA.

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http://dx.doi.org/10.1002/hed.20029DOI Listing
October 2004

SRC family kinases mediate epidermal growth factor receptor ligand cleavage, proliferation, and invasion of head and neck cancer cells.

Cancer Res 2004 Sep;64(17):6166-73

Department of Otolaryngology, University of Pittsburgh School of Medicine and University of Pittsburgh Cancer Institute, Pittsburgh, Pennsylvania 15213, USA.

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http://dx.doi.org/10.1158/0008-5472.CAN-04-0504DOI Listing
September 2004

Constitutive activation of Stat5b contributes to carcinogenesis in vivo.

Cancer Res 2003 Oct;63(20):6763-71

Departments of Otolaryngology, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania 15213, USA.

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October 2003

Responsiveness to the retinoic acid receptor-selective retinoid LGD1550 correlates with abrogation of transforming growth factor alpha/epidermal growth factor receptor autocrine signaling in head and neck squamous carcinoma cells.

Clin Cancer Res 2003 Sep;9(11):4205-13

Department of Otolaryngology, University of Pittsburgh School of Medicine and Cancer Institute, 200 Lothrop Street, Pittsburgh, PA 15213, USA.

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September 2003

Src kinases mediate STAT growth pathways in squamous cell carcinoma of the head and neck.

J Biol Chem 2003 Aug 27;278(34):31574-83. Epub 2003 May 27.

Department of Otolaryngology, University of Pittsburgh School of MedicinePennsylvania 15213, USA.

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http://dx.doi.org/10.1074/jbc.M303499200DOI Listing
August 2003

Targeted inhibition of Stat3 with a decoy oligonucleotide abrogates head and neck cancer cell growth.

Proc Natl Acad Sci U S A 2003 Apr 14;100(7):4138-43. Epub 2003 Mar 14.

Department of Otolaryngology, University of Pittsburgh School of Medicine and University of Pittsburgh Cancer Institute, Pittsburgh, PA 15260, USA.

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http://dx.doi.org/10.1073/pnas.0534764100DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC153061PMC
April 2003

STAT3 activation abrogates growth factor dependence and contributes to head and neck squamous cell carcinoma tumor growth in vivo.

Cell Growth Differ 2002 Aug;13(8):355-62

Departments of Otolaryngology, University of Pittsburgh School of Medicine and University of Pittsburgh Cancer Institute, Pittsburgh, Pennsylvania 15213, USA.

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August 2002

Mitogen activated protein kinase (MAPK) pathway regulates heme oxygenase-1 gene expression by hypoxia in vascular cells.

Antioxid Redox Signal 2002 Aug;4(4):587-92

Department of Medicine, Division of Pulmonary, Allergy, and Critical Care Medicine, University of Pittsburgh Medical Center, Pittsburgh, PA 15213, USA.

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http://dx.doi.org/10.1089/15230860260220085DOI Listing
August 2002

Differential function of STAT5 isoforms in head and neck cancer growth control.

Oncogene 2002 Apr;21(18):2846-53

Department of Otolaryngology, The University of Pittsburgh School of Medicine and The University of Pittsburgh Cancer Institute, Pittsburgh, Pennsylvania, USA.

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http://dx.doi.org/10.1038/sj.onc.1205385DOI Listing
April 2002