Renata Sano

Renata Sano

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Renata Sano

Renata Sano

Publications by authors named "Renata Sano"

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17Publications

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An antibody-drug conjugate directed to the ALK receptor demonstrates efficacy in preclinical models of neuroblastoma.

Sci Transl Med 2019 03;11(483)

Division of Oncology and Center for Childhood Cancer Research, Children's Hospital of Philadelphia, Philadelphia, PA 19104, USA.

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http://dx.doi.org/10.1126/scitranslmed.aau9732DOI Listing
March 2019

Mitochondria-associated ER membranes (MAMs) and lysosomal storage diseases.

Cell Death Dis 2018 02 28;9(3):328. Epub 2018 Feb 28.

Department of Genetics, St. Jude Children's Research Hospital, Memphis, TN, 38105, USA.

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http://dx.doi.org/10.1038/s41419-017-0025-4DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5832421PMC
February 2018

Crizotinib Synergizes with Chemotherapy in Preclinical Models of Neuroblastoma.

Clin Cancer Res 2016 Feb 5;22(4):948-60. Epub 2015 Oct 5.

Division of Oncology and Center for Childhood Cancer Research, The Children's Hospital of Philadelphia, Philadelphia, Pennsylvania.

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http://dx.doi.org/10.1158/1078-0432.CCR-15-0379DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4755925PMC
February 2016

The ALK/ROS1 Inhibitor PF-06463922 Overcomes Primary Resistance to Crizotinib in ALK-Driven Neuroblastoma.

Cancer Discov 2016 Jan 10;6(1):96-107. Epub 2015 Nov 10.

Division of Oncology and Center for Childhood Cancer Research, The Children's Hospital of Philadelphia, Philadelphia, Pennsylvania. Department of Pediatrics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, Pennsylvania.

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http://cancerdiscovery.aacrjournals.org/content/early/2015/1
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http://cancerdiscovery.aacrjournals.org/cgi/doi/10.1158/2159
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http://dx.doi.org/10.1158/2159-8290.CD-15-1056DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4707106PMC
January 2016

ER stress-induced cell death mechanisms.

Biochim Biophys Acta 2013 Dec 10;1833(12):3460-3470. Epub 2013 Jul 10.

Sanford-Burnham Medical Research Institute, La Jolla, CA, 92037, USA. Electronic address:

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http://dx.doi.org/10.1016/j.bbamcr.2013.06.028DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3834229PMC
December 2013

High-throughput fluorescence assay for small-molecule inhibitors of autophagins/Atg4.

J Biomol Screen 2011 Feb 18;16(2):174-82. Epub 2011 Jan 18.

Sanford-Burnham Medical Research Institute, Program on Apoptosis and Cell Death Research, and Conrad Prebys Center for Chemical Genomics, La Jolla, CA 92037, USA.

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http://dx.doi.org/10.1177/1087057110392996DOI Listing
February 2011

Population analysis of the GLB1 gene in South Brazil.

Genet Mol Biol 2011 Jan 1;34(1):45-8. Epub 2011 Mar 1.

Programa de Pós-Graduação em Genética e Biologia Molecular, Universidade Federal do Rio Grande do Sul, Porto Alegre, RS, Brasil.

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http://dx.doi.org/10.1590/S1415-47572011000100009DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3085372PMC
January 2011

Chemokine-induced recruitment of genetically modified bone marrow cells into the CNS of GM1-gangliosidosis mice corrects neuronal pathology.

Blood 2005 Oct 7;106(7):2259-68. Epub 2005 Jun 7.

Department of Genetics and Tumor Cell Biology, St Jude Children's Research Hospital, Memphis, TN 38105, USA.

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http://dx.doi.org/10.1182/blood-2005-03-1189DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1895262PMC
October 2005

G(M1)-ganglioside degradation and biosynthesis in human and murine G(M1)-gangliosidosis.

Clin Chim Acta 2005 Apr 26;354(1-2):131-9. Epub 2005 Jan 26.

Department of Biochemistry, ICBS, Federal University of Rio Grande do Sul, Porto Alegre, RS, Brazil.

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http://linkinghub.elsevier.com/retrieve/pii/S000989810400569
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http://dx.doi.org/10.1016/j.cccn.2004.11.035DOI Listing
April 2005

GM1-ganglioside-mediated activation of the unfolded protein response causes neuronal death in a neurodegenerative gangliosidosis.

Mol Cell 2004 Sep;15(5):753-66

Department of Genetics and Tumor Cell Biology, St. Jude Children's Research Hospital, 332 N. Lauderdale, Memphis, TN 38105, USA.

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http://dx.doi.org/10.1016/j.molcel.2004.08.029DOI Listing
September 2004