Patrick Viatour

Patrick Viatour

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Patrick Viatour

Patrick Viatour

Publications by authors named "Patrick Viatour"

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Promotion of growth factor signaling as a critical function of β-catenin during HCC progression.

Nat Commun 2019 04 23;10(1):1909. Epub 2019 Apr 23.

Children's Hospital of Philadelphia, Center for Childhood Cancer Research, Philadelphia, PA, 19104, USA.

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http://dx.doi.org/10.1038/s41467-019-09780-zDOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC6478918PMC
April 2019

Recruitment of Pontin/Reptin by E2f1 amplifies E2f transcriptional response during cancer progression.

Nat Commun 2015 Dec 7;6:10028. Epub 2015 Dec 7.

Division of Cancer Pathobiology, Department of Pathology and Laboratory Medicine, Center for Childhood Cancer Research, Children's Hospital of Philadelphia, Philadelphia, Pennsylvania 19104, USA.

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http://dx.doi.org/10.1038/ncomms10028DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4686657PMC
December 2015

Bridges between Cell Cycle Regulation and Self-Renewal Maintenance.

Authors:
Patrick Viatour

Genes Cancer 2012 Nov;3(11-12):670-7

Center for Childhood Cancer Research, The Children's Hospital of Philadelphia, Philadelphia, PA, USA ; Department of Pathology and Laboratory Medicine, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.

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http://dx.doi.org/10.1177/1947601913481355DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3636744PMC
November 2012

Notch signaling inhibits hepatocellular carcinoma following inactivation of the RB pathway.

J Exp Med 2011 Sep 29;208(10):1963-76. Epub 2011 Aug 29.

Department of Genetics, Department of Pediatrics, Stanford University, Stanford, CA, USA; Department of Medical Chemistry, University of Liège, B-4000 Liège, Belgium.

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http://dx.doi.org/10.1084/jem.20110198DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3182062PMC
September 2011

Newly identified aspects of tumor suppression by RB.

Dis Model Mech 2011 Sep;4(5):581-5

Department of Genetics, Stanford University, Stanford, CA 94305, USA.

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http://dx.doi.org/10.1242/dmm.008060DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3180221PMC
September 2011

BCL-3 degradation involves its polyubiquitination through a FBW7-independent pathway and its binding to the proteasome subunit PSMB1.

J Biol Chem 2010 Aug 17;285(33):25831-40. Epub 2010 Jun 17.

Interdisciplinary Cluster for Applied Genoproteomics, GIGA-Research, Unit of Medical Chemistry, Department of Infectious and Parasitic Diseases, Faculty of Veterinary Medicine, University of Liège, Sart-Tilman, 4000 Liège, Belgium.

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http://www.jbc.org/content/285/33/25831.full.pdf
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http://www.jbc.org/cgi/doi/10.1074/jbc.M110.112128
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http://dx.doi.org/10.1074/jbc.M110.112128DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2919145PMC
August 2010

Regulation of RB transcription in vivo by RB family members.

Mol Cell Biol 2010 Apr 25;30(7):1729-45. Epub 2010 Jan 25.

Department of Pediatrics, Stanford Medical School, 269 Campus Drive, CCSR1215, Stanford, CA 94305, USA.

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http://dx.doi.org/10.1128/MCB.00952-09DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2838078PMC
April 2010

GFP reporter mice for the retinoblastoma-related cell cycle regulator p107.

Cell Cycle 2008 Aug 12;7(16):2544-52. Epub 2008 Aug 12.

Department of Pediatrics and Genetics, Cancer Biology Program, Stanford Medical School, Stanford, California, USA.

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2730771PMC
http://dx.doi.org/10.4161/cc.7.16.6441DOI Listing
August 2008

pRB family proteins are required for H3K27 trimethylation and Polycomb repression complexes binding to and silencing p16INK4alpha tumor suppressor gene.

Genes Dev 2007 Jan;21(1):49-54

Department of Biochemistry and Biophysics, Lineberger Comprehensive Cancer Center, Program in Molecular Biology and Biotechnology, University of North Carolina at Chapel Hill, Chapel Hill, North Carolina 27599, USA.

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http://dx.doi.org/10.1101/gad.1499407DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1759899PMC
January 2007

Deregulated NF-kappaB activity in haematological malignancies.

Biochem Pharmacol 2006 Oct 18;72(9):1069-80. Epub 2006 Jul 18.

Laboratory of Medical Chemistry, Center for Cellular and Molecular Therapy, Center for Biomedical Integrative Genoproteomics, University of Liege, Tour de Pathologie, +3 B23, CHU Sart-Tilman, 4000 Liège, Belgium.

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http://dx.doi.org/10.1016/j.bcp.2006.06.011DOI Listing
October 2006

Phosphorylation of NF-kappaB and IkappaB proteins: implications in cancer and inflammation.

Trends Biochem Sci 2005 Jan;30(1):43-52

Laboratory of Medical Chemistry and Human Genetics, CHU, Sart-Tilman, Center for Biomedical Integrated Genoproteomics, University of Liege, Belgium.

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http://linkinghub.elsevier.com/retrieve/pii/S096800040400299
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http://dx.doi.org/10.1016/j.tibs.2004.11.009DOI Listing
January 2005

Protein phosphorylation as a key mechanism for the regulation of BCL-3 activity.

Cell Cycle 2004 Dec 22;3(12):1498-501. Epub 2004 Dec 22.

Laboratory of Medical Chemistry and Human Genetics, Center for Biomedical Integrated Genoproteomics, University of Liège, Sart-Tilman, Liège, Belgium.

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http://dx.doi.org/10.4161/cc.3.12.1328DOI Listing
December 2004

Cytoplasmic IkappaBalpha increases NF-kappaB-independent transcription through binding to histone deacetylase (HDAC) 1 and HDAC3.

J Biol Chem 2003 Nov 12;278(47):46541-8. Epub 2003 Sep 12.

Laboratory of Medical Chemistry and Human Genetics, Center for Cellular and Molecular Therapy, University of Liège, Sart-Tillman, 4000 Liège, Belgium.

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http://www.jbc.org/lookup/doi/10.1074/jbc.M306381200
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http://dx.doi.org/10.1074/jbc.M306381200DOI Listing
November 2003