Maxim V Frolov

Maxim V Frolov

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Maxim V Frolov

Maxim V Frolov

Publications by authors named "Maxim V Frolov"

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27Publications

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E2F/DP Prevents Cell-Cycle Progression in Endocycling Fat Body Cells by Suppressing dATM Expression.

Dev Cell 2017 12 7;43(6):689-703.e5. Epub 2017 Dec 7.

Massachusetts General Hospital Cancer Center and Harvard Medical School, Building 149 13(th) Street, Charlestown, MA 02129, USA. Electronic address:

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December 2017

Microprocessor Recruitment to Elongating RNA Polymerase II Is Required for Differential Expression of MicroRNAs.

Cell Rep 2017 Sep;20(13):3123-3134

Department of Molecular Biosciences, Northwestern University, Evanston, IL 60208, USA. Electronic address:

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September 2017

Novel regulation and functional interaction of polycistronic miRNAs.

RNA 2016 Jan 9;22(1):129-38. Epub 2015 Nov 9.

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Chicago, Illinois 60607, USA.

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January 2016

E2F function in muscle growth is necessary and sufficient for viability in Drosophila.

Nat Commun 2016 Jan 29;7:10509. Epub 2016 Jan 29.

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, 900 S Ashland Avenue, Chicago, Illinois 60607, USA.

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January 2016

Emerging links between E2F control and mitochondrial function.

Cancer Res 2015 Feb 29;75(4):619-23. Epub 2015 Jan 29.

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Chicago, Illinois.

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February 2015

An intronic microRNA links Rb/E2F and EGFR signaling.

PLoS Genet 2014 Jul 24;10(7):e1004493. Epub 2014 Jul 24.

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Chicago, Illinois, United States of America.

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July 2014

Loss of dE2F compromises mitochondrial function.

Dev Cell 2013 Nov;27(4):438-51

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Chicago, IL 60607, USA.

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November 2013

Drosophila GAGA factor is required for full activation of the dE2f1-Yki/Sd transcriptional program.

Cell Cycle 2012 Nov 15;11(22):4191-202. Epub 2012 Oct 15.

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Chicago, IL, USA.

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November 2012

mir-11 limits the proapoptotic function of its host gene, dE2f1.

Genes Dev 2011 Sep 19;25(17):1820-34. Epub 2011 Aug 19.

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Chicago, Illinois 60607, USA.

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September 2011

Cooperation between dE2F1 and Yki/Sd defines a distinct transcriptional program necessary to bypass cell cycle exit.

Genes Dev 2011 Feb;25(4):323-35

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Chicago, Illinois 60607, USA.

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February 2011

Notch-dependent expression of the archipelago ubiquitin ligase subunit in the Drosophila eye.

Development 2011 Jan 9;138(2):251-60. Epub 2010 Dec 9.

Department of Cell Biology, Emory University School of Medicine, Atlanta, GA 30322, USA.

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January 2011

Tuberous sclerosis complex 1 regulates dE2F1 expression during development and cooperates with RBF1 to control proliferation and survival.

PLoS Genet 2010 Aug 19;6(8):e1001071. Epub 2010 Aug 19.

Department of Biology, Developmental Biology Research Initiative, McGill University, Montreal, Canada.

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August 2010

Combined inactivation of pRB and hippo pathways induces dedifferentiation in the Drosophila retina.

PLoS Genet 2010 Apr 22;6(4):e1000918. Epub 2010 Apr 22.

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Chicago, Illinois, United States of America.

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April 2010

Mutation of the DEAD-box helicase belle downregulates the cyclin-dependent kinase inhibitor Dacapo.

Cell Cycle 2010 Mar 15;9(5):1016-20. Epub 2010 Mar 15.

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Chicago, IL, USA.

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March 2010

The diverse roles of RNA helicases in RNAi.

Cell Cycle 2009 Nov 24;8(21):3500-5. Epub 2009 Nov 24.

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Chicago, IL, USA.

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November 2009

Mosaic genetic screen for suppressors of the de2f1 mutant phenotype in Drosophila.

Genetics 2009 Sep 22;183(1):79-92. Epub 2009 Jun 22.

Department of Biochemistry and Molecular Genetics, University of Illinois, Chicago, Illinois 60607, USA.

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September 2009

The archipelago tumor suppressor gene limits rb/e2f-regulated apoptosis in developing Drosophila tissues.

Curr Biol 2009 Sep 3;19(18):1503-10. Epub 2009 Sep 3.

Department of Cell Biology, Emory University School of Medicine, Atlanta, GA 30322, USA.

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September 2009

Context-dependent requirement for dE2F during oncogenic proliferation.

PLoS Genet 2008 Oct 3;4(10):e1000205. Epub 2008 Oct 3.

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Illinois, United States of America.

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October 2008

dE2F2-independent rescue of proliferation in cells lacking an activator dE2F1.

Mol Cell Biol 2007 Dec 8;27(24):8561-70. Epub 2007 Oct 8.

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, Chicago, IL 60607, USA.

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December 2007

Specific role of the SR protein splicing factor B52 in cell cycle control in Drosophila.

Mol Cell Biol 2006 May;26(9):3468-77

Department of Biochemistry and Molecular Genetics, University of Illinois at Chicago, MBRB 2352, MC 669, 900 S. Ashland Ave., Chicago, IL 60607, USA.

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May 2006

Retinoblastoma family 2 is required in vivo for the tissue-specific repression of dE2F2 target genes.

Cell Cycle 2005 Sep 29;4(9):1272-80. Epub 2005 Sep 29.

Massachusetts General Hospital Cancer Center, Charlestown, Massachusetts 02129, USA.

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September 2005

dDP is needed for normal cell proliferation.

Mol Cell Biol 2005 Apr;25(8):3027-39

Massachusetts General Hospital Cancer Center, Bldg. 149, 13th St., Charlestown, MA 02129, USA.

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April 2005

Molecular mechanisms of E2F-dependent activation and pRB-mediated repression.

J Cell Sci 2004 May;117(Pt 11):2173-81

Massachusetts General Hospital Cancer Center, Bldg 149, 13th Street, Charlestown, MA 02129, USA.

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May 2004

Cell cycle-dependent and cell cycle-independent control of transcription by the Drosophila E2F/RB pathway.

Genes Dev 2003 Sep;17(18):2308-20

Massachusetts General Hospital Cancer Center, Charlestown, Massachusetts, 02129 USA.

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September 2003

G1 cyclin-dependent kinases are insufficient to reverse dE2F2-mediated repression.

Genes Dev 2003 Mar;17(6):723-8

Massachusetts General Hospital Cancer Center, Charlestown, MA 02129, USA.

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March 2003

Distinct mechanisms of E2F regulation by Drosophila RBF1 and RBF2.

EMBO J 2002 Sep;21(18):4927-37

Massachusetts General Hospital Cancer Center, Laboratory of Molecular Oncology, Charlestown, MA 02129, USA.

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September 2002