Masashi Idogawa

Masashi Idogawa

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Masashi Idogawa

Masashi Idogawa

Publications by authors named "Masashi Idogawa"

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Prognostic Effect of Long Noncoding RNA NEAT1 Expression Depends on p53 Mutation Status in Cancer.

J Oncol 2019 2;2019:4368068. Epub 2019 May 2.

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University School of Medicine, Japan.

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http://dx.doi.org/10.1155/2019/4368068DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC6521466PMC
May 2019

Targeted next-generation sequencing of 50 cancer-related genes in Japanese patients with oral squamous cell carcinoma.

Tumour Biol 2018 Sep;40(9):1010428318800180

1 Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University, Sapporo, Japan.

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http://dx.doi.org/10.1177/1010428318800180DOI Listing
September 2018

HSP72 functionally inhibits the anti-neoplastic effects of HDAC inhibitors.

J Dermatol Sci 2018 Apr 31;90(1):82-89. Epub 2018 Jan 31.

Department of Dermatology, Kagoshima University Graduate School of Medical and Dental Sciences, 8-35-1, Sakuragaoka, Kagoshima, Japan.

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http://dx.doi.org/10.1016/j.jdermsci.2018.01.002DOI Listing
April 2018

Identification and characterization of a metastatic suppressor BRMS1L as a target gene of p53.

Cancer Sci 2017 Dec 6;108(12):2413-2421. Epub 2017 Nov 6.

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University, Sapporo, Japan.

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http://dx.doi.org/10.1111/cas.13420DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5715288PMC
December 2017

Fledglings in p53 signaling network.

Oncotarget 2017 Aug 14;8(34):55768-55769. Epub 2017 Jul 14.

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University, Sapporo, Japan.

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http://dx.doi.org/10.18632/oncotarget.19229DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5593519PMC
August 2017

Profiling cancer-related gene mutations in oral squamous cell carcinoma from Japanese patients by targeted amplicon sequencing.

Oncotarget 2017 Aug 15;8(35):59113-59122. Epub 2017 Jul 15.

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University, Sapporo, Japan.

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http://dx.doi.org/10.18632/oncotarget.19262DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5601718PMC
August 2017

Long non-coding RNA NEAT1 is a transcriptional target of p53 and modulates p53-induced transactivation and tumor-suppressor function.

Int J Cancer 2017 06 27;140(12):2785-2791. Epub 2017 Mar 27.

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University School of Medicine, Sapporo, Japan.

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http://dx.doi.org/10.1002/ijc.30689DOI Listing
June 2017

p53 mediates the suppression of cancer cell invasion by inducing LIMA1/EPLIN.

Cancer Lett 2017 04 13;390:58-66. Epub 2017 Jan 13.

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University School of Medicine, Japan. Electronic address:

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http://dx.doi.org/10.1016/j.canlet.2016.12.034DOI Listing
April 2017

Identification and characterization of the intercellular adhesion molecule-2 gene as a novel p53 target.

Oncotarget 2016 Sep;7(38):61426-61437

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University, Sapporo, Japan.

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http://dx.doi.org/10.18632/oncotarget.11366DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5308662PMC
September 2016

CRKL oncogene is downregulated by p53 through miR-200s.

Cancer Sci 2015 Aug 14;106(8):1033-40. Epub 2015 Jul 14.

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University, Sapporo, Japan.

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http://dx.doi.org/10.1111/cas.12713DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4556393PMC
August 2015

Array-based genome-wide RNAi screening to identify shRNAs that enhance p53-related apoptosis in human cancer cells.

Oncotarget 2014 Sep;5(17):7540-8

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University School of Medicine, Sapporo, Japan.

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4202142PMC
http://dx.doi.org/10.18632/oncotarget.2272DOI Listing
September 2014

Conditioned mesenchymal stem cells produce pleiotropic gut trophic factors.

J Gastroenterol 2014 Feb 12;49(2):270-82. Epub 2013 Nov 12.

Department of Gastroenterology, Rheumatology, and Clinical Immunology, Sapporo Medical University, S-1, W-16, Chuo-ku, Sapporo, 060-8543, Japan.

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http://link.springer.com/10.1007/s00535-013-0901-3
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http://dx.doi.org/10.1007/s00535-013-0901-3DOI Listing
February 2014

AKR1B10, a transcriptional target of p53, is downregulated in colorectal cancers associated with poor prognosis.

Mol Cancer Res 2013 Dec 18;11(12):1554-63. Epub 2013 Oct 18.

S1W17, Chuo-Ku, Sapporo 060-8556, Japan.

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http://dx.doi.org/10.1158/1541-7786.MCR-13-0330-TDOI Listing
December 2013

CLCA2, a target of the p53 family, negatively regulates cancer cell migration and invasion.

Cancer Biol Ther 2012 Dec 18;13(14):1512-21. Epub 2012 Sep 18.

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University, Sapporo, Japan.

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http://www.tandfonline.com/doi/abs/10.4161/cbt.22280
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http://dx.doi.org/10.4161/cbt.22280DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3542243PMC
December 2012

CHFR protein regulates mitotic checkpoint by targeting PARP-1 protein for ubiquitination and degradation.

J Biol Chem 2012 Apr 15;287(16):12975-84. Epub 2012 Feb 15.

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University, Sapporo 060-8556, Japan.

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http://dx.doi.org/10.1074/jbc.M111.321828DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3339944PMC
April 2012

p53 negatively regulates the hepatoma growth factor HDGF.

Cancer Res 2011 Nov 17;71(22):7038-47. Epub 2011 Oct 17.

Department of Medical Genome Sciences, Research Institute for Frontier Medicine, Sapporo Medical University, Sapporo, Japan.

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http://dx.doi.org/10.1158/0008-5472.CAN-11-1053DOI Listing
November 2011

Identification and characterization of early growth response 2, a zinc-finger transcription factor, as a p53-regulated proapoptotic gene.

Int J Oncol 2010 Dec;37(6):1407-16

Department of Molecular Biology, Cancer Research Institute, Sapporo Medical University School of Medicine, Chuo-ku, Sapporo 060-8556, Japan.

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http://dx.doi.org/10.3892/ijo_00000792DOI Listing
December 2010

A single recombinant adenovirus expressing p53 and p21-targeting artificial microRNAs efficiently induces apoptosis in human cancer cells.

Clin Cancer Res 2009 Jun 19;15(11):3725-32. Epub 2009 May 19.

Department of Molecular Biology, Cancer Research Institute, Sapporo Medical University, Sapporo, Japan.

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http://dx.doi.org/10.1158/1078-0432.CCR-08-2396DOI Listing
June 2009

p53 family members regulate the expression of the apolipoprotein D gene.

J Biol Chem 2009 Jan 11;284(2):872-83. Epub 2008 Nov 11.

Department of Molecular Biology, Cancer Research Institute, Sapporo Medical University, S-1, W-17, Chuo-ku, Sapporo, 060-8556 Japan.

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http://dx.doi.org/10.1074/jbc.M807185200DOI Listing
January 2009

Histone deacetylase inhibitor FK228 enhances adenovirus-mediated p53 family gene therapy in cancer models.

Mol Cancer Ther 2008 Apr;7(4):779-87

Department of Molecular Biology, Cancer Research Institute, Sapporo Medical University School of Medicine, S-1, W-17, Chuo-ku, Sapporo, 060-8556 Japan.

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http://dx.doi.org/10.1158/1535-7163.MCT-07-0395DOI Listing
April 2008

Frequent epigenetic inactivation of DICKKOPF family genes in human gastrointestinal tumors.

Carcinogenesis 2007 Dec 3;28(12):2459-66. Epub 2007 Aug 3.

First Department of Internal Medicine, Sapporo Medical University, Sapporo 060-8543, Japan.

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http://dx.doi.org/10.1093/carcin/bgm178DOI Listing
December 2007

Antitumor effect of adenovirus-mediated p53 family gene transfer on osteosarcoma cell lines.

Cancer Biol Ther 2007 Jul;6(7):1058-66

Department of Molecular Biology, Cancer Research Institute Sapporo Medical University School of Medicine, Chuo-ku, Sapporo, Japan.

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https://fly-bay.com/journals/cbt/OshimaCBT6-7.pdf
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http://dx.doi.org/10.4161/cbt.6.7.4320DOI Listing
July 2007

Involvement of splicing factor-1 in beta-catenin/T-cell factor-4-mediated gene transactivation and pre-mRNA splicing.

Gastroenterology 2007 Mar 5;132(3):1039-54. Epub 2007 Jan 5.

Chemotherapy Division and Cancer Proteomics Project, National Cancer Center Research Institute, Tokyo, Japan.

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http://linkinghub.elsevier.com/retrieve/pii/S001650850700009
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http://dx.doi.org/10.1053/j.gastro.2007.01.007DOI Listing
March 2007

Ku70 and poly(ADP-ribose) polymerase-1 competitively regulate beta-catenin and T-cell factor-4-mediated gene transactivation: possible linkage of DNA damage recognition and Wnt signaling.

Cancer Res 2007 Feb;67(3):911-8

Chemotherapy Division and ADP-Ribosylation in Oncology Project, National Cancer Center Research Institute, 5-1-1 Tsukiji, Chuo-ku, Tokyo 104-0045, Japan.

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http://dx.doi.org/10.1158/0008-5472.CAN-06-2360DOI Listing
February 2007

E-cadherin regulates the association between beta-catenin and actinin-4.

Cancer Res 2005 Oct;65(19):8836-45

Chemotherapy Division and Cancer Proteomics Project, National Cancer Center Research Institute, Tokyo, Japan.

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http://dx.doi.org/10.1158/0008-5472.CAN-05-0718DOI Listing
October 2005

Beta-catenin interacts with the FUS proto-oncogene product and regulates pre-mRNA splicing.

Gastroenterology 2005 Oct;129(4):1225-36

Chemotherapy Division and Cancer Proteomics Project, National Cancer Center Research Institute, Tokyo, Japan.

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http://dx.doi.org/10.1053/j.gastro.2005.07.025DOI Listing
October 2005

Poly(ADP-ribose) polymerase-1 is a component of the oncogenic T-cell factor-4/beta-catenin complex.

Gastroenterology 2005 Jun;128(7):1919-36

Chemotherapy Division, National Cancer Center Research Institute, Tokyo, Japan.

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http://dx.doi.org/10.1053/j.gastro.2005.03.007DOI Listing
June 2005

Morphological and transcriptional responses of untransformed intestinal epithelial cells to an oncogenic beta-catenin protein.

Oncogene 2005 Apr;24(19):3141-53

Chemotherapy Division and Cancer Proteomics Project, National Cancer Center Research Institute, 5-1-1 Tsukiji, Chuo-ku, Tokyo 104-0045, Japan.

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http://dx.doi.org/10.1038/sj.onc.1208517DOI Listing
April 2005

Actinin-4 increases cell motility and promotes lymph node metastasis of colorectal cancer.

Gastroenterology 2005 Jan;128(1):51-62

Chemotherapy Division and Cancer Proteomics Project, National Cancer Center Research Institute, 5-1-1 Tsukiji, Chuoh-ku, Tokyo 104-0045, Japan.

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http://dx.doi.org/10.1053/j.gastro.2004.10.004DOI Listing
January 2005

Alternative splice variant of actinin-4 in small cell lung cancer.

Oncogene 2004 Jul;23(30):5257-62

Cancer Proteomics Project, National Cancer Center Research Institute, 5-1-1 Tsukiji, Chuo-ku, Tokyo 104-0045, Japan.

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http://www.nature.com/articles/1207652
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http://dx.doi.org/10.1038/sj.onc.1207652DOI Listing
July 2004

Overexpression of BAD preferentially augments anoikis.

Int J Cancer 2003 Nov;107(2):215-23

First Department of Internal Medicine, Sapporo Medical University, Sapporo, Japan.

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http://dx.doi.org/10.1002/ijc.11399DOI Listing
November 2003