Louisa M Gerhardt

Louisa M S Gerhardt

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Louisa M Gerhardt

Louisa M S Gerhardt

Publications by authors named "Louisa M S Gerhardt"

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1PubMed Central Citations

[Hyperkalemia - Pathophysiology, prognostic significance and treatment options].

Dtsch Med Wochenschr 2019 Nov 28;144(22):1576-1584. Epub 2019 Oct 28.

Medizinische Klinik I und Deutsches Zentrum für Herzinsuffizienz, Universität und Universitätsklinikum Würzburg, Würzburg, Deutschland.

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http://dx.doi.org/10.1055/a-0762-8244DOI Listing
November 2019

Ly-6Chigh monocytes depend on Nr4a1 to balance both inflammatory and reparative phases in the infarcted myocardium.

Circ Res 2014 May 13;114(10):1611-22. Epub 2014 Mar 13.

From the Center for Systems Biology (I.H., L.M.S.G., C.W., B.G.C., Y.I., M.N., R.W., F.K.S.) and Department of Cardiology (T.C.T., M.S.-C.), Massachusetts General Hospital, Boston; Department of Gastroenterology, Hepatology, and Infectious Diseases, University of Duesseldorf, Duesseldorf, Germany (T.A.W.H.); Department of Medicine (R.L.) and Cardiovascular Division, Department of Medicine (P.L.), Brigham and Women's Hospital, Boston, MA; Department of Cardiology and Angiology I, University Heart Center Freiburg, Freiburg, Germany (A.Z.); Division of Inflammation Biology, La Jolla Institute for Allergy and Immunology, CA (C.C.H.); and Department of Systems Biology, Harvard Medical School, Boston, MA (R.W.).

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http://circres.ahajournals.org/content/early/2014/03/13/CIRC
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http://circres.ahajournals.org/cgi/doi/10.1161/CIRCRESAHA.11
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http://dx.doi.org/10.1161/CIRCRESAHA.114.303204DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4017349PMC
May 2014

Innate response activator B cells aggravate atherosclerosis by stimulating T helper-1 adaptive immunity.

Circulation 2014 Apr 31;129(16):1677-87. Epub 2014 Jan 31.

Center for Systems Biology, Massachusetts General Hospital, Boston (I.H., I.T., L.M.S.G., C.S.R., G.F.W., Y.I., C.W., H.Y.L., M.N., R.W., F.K.S.); Department of Internal Medicine VI, Infectious Diseases, Immunology Rheumatology, Pneumology, University Hospital of Innsbruck, Innsbruck, Austria (I.T.); Toronto General Research Institute, University Health Network, Toronto, ON, Canada (C.S.R., N.D.); Department of Medicine, University of California, San Diego, La Jolla (A.G., J.L.W.); Department of Gastroenterology, Hepatology and Infectious Diseases, University of Duesseldorf, Duesseldorf, Germany (T.A.W.H.); Department of Cardiology and Angiology I, University Heart Center Freiburg, Freiburg, Germany (C.W., A.Z.); Cardiovascular Division, Department of Medicine, Brigham and Women's Hospital, Boston, MA (G.K.S., P.L.); Department of Pathology (J.B.) and Division of Vascular Surgery (B.B.R.), Peter Munk Cardiac Centre, Toronto General Hospital, Toronto, ON, Canada; and Department of Systems Biology, Harvard Medical School, Boston, MA (R.W.).

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https://www.ahajournals.org/doi/10.1161/CIRCULATIONAHA.113.0
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http://dx.doi.org/10.1161/CIRCULATIONAHA.113.006381DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3997655PMC
April 2014

Local proliferation dominates lesional macrophage accumulation in atherosclerosis.

Nat Med 2013 Sep 11;19(9):1166-72. Epub 2013 Aug 11.

1] Center for Systems Biology, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA. [2] Toronto General Research Institute, University Health Network, Toronto, Ontario, Canada. [3] Department of Laboratory Medicine and Pathobiology, University of Toronto, Toronto, Ontario, Canada. [4] Department of Immunology, University of Toronto, Toronto, Ontario, Canada. [5].

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http://dx.doi.org/10.1038/nm.3258DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3769444PMC
September 2013