Publications



High Vitamin D-Binding Protein Concentration, Low Albumin, and Mode of Remission Predict Relapse in Crohn's Disease.
Inflamm Bowel Dis 2016 Oct;22(10):2456-64
*Harry Perkins Institute of Medical Research, School of Medicine and Pharmacology, University of Western Australia, Murdoch, Western Australia, Australia;†Telethon Kids Institute, University of Western Australia, Subiaco, Western Australia, Australia;‡UWA Centre for Applied Statistics, University of Western Australia, Western Australia, Crawley, Australia;§Department of Gastroenterology and Hepatology, Flinders Medical Centre, South Australia, Adelaide, Australia;‖Department of Gastroenterology and Hepatology, Royal Brisbane and Women's Hospital, Queensland, Brisbane, Australia; and¶Centre for Inflammatory Bowel Diseases, Saint John of God Hospital, Subiaco, Western Australia, Australia.

Genome-wide meta-analysis identifies five new susceptibility loci for cutaneous malignant melanoma.
Nat Genet 2015 Sep 3;47(9):987-995. Epub 2015 Aug 3.
Section of Epidemiology and Biostatistics, Leeds Institute of Cancer and Pathology, University of Leeds, Leeds, UK.

Smoking behaviour modifies IL23r-associated disease risk in patients with Crohn's disease.
J Gastroenterol Hepatol 2015 Feb;30(2):299-307
CSIRO Computational Informatics/Australian E-Health Research Centre, Royal Brisbane and Women's Hospital, Brisbane, Victoria, Australia; Preventative Health Flagship, Melbourne, Victoria, Australia.



Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease.
Nature 2012 Nov;491(7422):119-24
Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Hinxton, Cambridge CB10 1HH, UK.

Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47.
Nat Genet 2011 Mar 6;43(3):246-52. Epub 2011 Feb 6.
Wellcome Trust Sanger Institute, Wellcome Trust Genome Campus, Hinxton, Cambridge, UK.

Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci.
Nat Genet 2010 Dec;42(12):1118-25
Institute of Clinical Molecular Biology, Christian-Albrechts-University Kiel, Kiel, Germany.



Bone morphogenic protein 3 inactivation is an early and frequent event in colorectal cancer development.
Genes Chromosomes Cancer 2008 Jun;47(6):449-60
Conjoint Gastroenterology Laboratory, Royal Brisbane and Women's Hospital Research Foundation Clinical Research Centre and Queensland Institute of Medical Research, Brisbane 4029, Australia.


SnoN expression is differently regulated in microsatellite unstable compared with microsatellite stable colorectal cancers.
BMC Cancer 2006 Oct 24;6:252. Epub 2006 Oct 24.
The Conjoint Gastroenterology Laboratory, Royal Brisbane and Women's Hospital Foundation Clinical Research Centre and the Queensland Institute of Medical Research, Brisbane, 4029, Australia.

The PCNA-associated factor KIAA0101/p15(PAF) binds the potential tumor suppressor product p33ING1b.
Exp Cell Res 2006 Jan 8;312(1):73-85. Epub 2005 Nov 8.
Institute for Molecular Bioscience, The University of Queensland, St Lucia, Brisbane, Queensland 4072, Australia.


Identification of genes uniquely involved in frequent microsatellite instability colon carcinogenesis by expression profiling combined with epigenetic scanning.
Cancer Res 2004 Apr;64(7):2434-8
Department of Medicine, Division of Gastroenterology, University of Maryland School of Medicine and Greenebaum Cancer Center and Baltimore Veterans Affairs Hospital, Baltimore, Maryland, USA.

The impact of microsatellite instability on the molecular phenotype of colorectal tumors.
Cancer Res 2003 Aug;63(15):4577-82
Department of Medicine, Division of Gastroenterology, University of Maryland School of Medicine, 655 West Baltimore Street, Baltimore, MD 21201, USA.

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