Publications by authors named "Enza Piccolella"

21Publications

The antimicrobial peptide LL37 is a T-cell autoantigen in psoriasis.

Nat Commun 2014 Dec 3;5:5621. Epub 2014 Dec 3.

1] Department of Infectious, Parasitic and Immunomediated Diseases, Istituto Superiore di Sanità, 00100 Rome, Italy [2] Department of Dermatology, University Hospital CHUV, 1011 Lausanne, Switzerland.

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http://dx.doi.org/10.1038/ncomms6621DOI Listing
December 2014

CD28 ligation in the absence of TCR stimulation up-regulates IL-17A and pro-inflammatory cytokines in relapsing-remitting multiple sclerosis T lymphocytes.

Immunol Lett 2014 Mar-Apr;158(1-2):134-42. Epub 2014 Jan 8.

Istituto Pasteur-Fondazione Cenci Bolognetti, Department of Biology and Biotechnology Charles Darwin, Sapienza University, Via dei sardi 70, 00185 Rome, Italy. Electronic address:

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http://dx.doi.org/10.1016/j.imlet.2013.12.020DOI Listing
November 2014

Phosphatidylinositol 4-phosphate 5-kinase α activation critically contributes to CD28-dependent signaling responses.

J Immunol 2013 May 15;190(10):5279-86. Epub 2013 Apr 15.

Department of Biology and Biotechnology Charles Darwin, Fondazione Cenci Bolognetti Pasteur Institute, Sapienza University, 00185 Rome, Italy.

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http://www.jimmunol.org/cgi/doi/10.4049/jimmunol.1203157
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http://dx.doi.org/10.4049/jimmunol.1203157DOI Listing
May 2013

CD28 costimulation regulates FOXP3 in a RelA/NF-κB-dependent mechanism.

Eur J Immunol 2011 Feb 11;41(2):503-13. Epub 2011 Jan 11.

Department of Biology and Biotechnology C. Darwin, University Sapienza of Rome, Italy.

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http://doi.wiley.com/10.1002/eji.201040712
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http://dx.doi.org/10.1002/eji.201040712DOI Listing
February 2011

FOXP3 induced by CD28/B7 interaction regulates CD25 and anergic phenotype in human CD4+CD25- T lymphocytes.

J Immunol 2008 Jul;181(2):1025-33

Department of Cellular and Developmental Biology, University Sapienza of Rome, Rome, Italy.

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http://dx.doi.org/10.4049/jimmunol.181.2.1025DOI Listing
July 2008

Hypervariable region 1 variant acting as TCR antagonist affects hepatitis C virus-specific CD4+ T cell repertoire by favoring CD95-mediated apoptosis.

J Leukoc Biol 2005 Aug 27;78(2):372-82. Epub 2005 May 27.

Department of Cellular and Developmental Biology, La Sapienza University, Rome, 00185, Italy.

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http://dx.doi.org/10.1189/jlb.0804456DOI Listing
August 2005

CD28 delivers a unique signal leading to the selective recruitment of RelA and p52 NF-kappaB subunits on IL-8 and Bcl-xL gene promoters.

Proc Natl Acad Sci U S A 2004 Apr 12;101(16):6098-103. Epub 2004 Apr 12.

Department of Cellular and Developmental Biology, University of Rome La Sapienza, 00185 Rome, Italy.

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http://dx.doi.org/10.1073/pnas.0308688101DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC395929PMC
April 2004

The activation of Csk by CD4 interferes with TCR-mediated activatory signaling.

Eur J Immunol 2003 Sep;33(9):2609-18

Department of Cellular and Developmental Biology, La Sapienza University, I-00185 Rome, Italy.

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http://dx.doi.org/10.1002/eji.200324064DOI Listing
September 2003

Vav exchange factor counteracts the HIV-1 Nef-mediated decrease of plasma membrane GM1 and NF-AT activity in T cells.

Eur J Immunol 2003 Aug;33(8):2186-96

Department of Cellular and Developmental Biology, La Sapienza University, Rome, Italy.

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http://dx.doi.org/10.1002/eji.200323682DOI Listing
August 2003

CD4-Lck through TCR and in the absence of Vav exchange factor induces Bax increase and mitochondrial damage.

J Immunol 2002 Jun;168(12):6106-12

Department of Cellular and Developmental Biology, La Sapienza University, Rome, Italy.

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http://dx.doi.org/10.4049/jimmunol.168.12.6106DOI Listing
June 2002

Vav cooperates with CD28 to induce NF-kappaB activation via a pathway involving Rac-1 and mitogen-activated kinase kinase 1.

Eur J Immunol 2002 02;32(2):447-56

Department of Cellular and Developmental Biology, University of Rome La Sapienza, Via del Sardi 70, I-00185 Rome, Italy.

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http://dx.doi.org/10.1002/1521-4141(200202)32:2<447::AID-IMMU447>3.0.CO;2-5DOI Listing
February 2002

Human anergic CD4+ T cells can act as suppressor cells by affecting autologous dendritic cell conditioning and survival.

J Immunol 2002 Feb;168(3):1060-8

Department of Cellular and Developmental Biology, La Sapienza University, Via dei Sardi, 70-00185 Rome, Italy.

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http://dx.doi.org/10.4049/jimmunol.168.3.1060DOI Listing
February 2002