Dmitry Terentyev

Dmitry Terentyev

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Dmitry Terentyev

Dmitry Terentyev

Publications by authors named "Dmitry Terentyev"

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53Publications

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Proarrhythmic Remodeling of Calcium Homeostasis in Cardiac Disease; Implications for Diabetes and Obesity.

Front Physiol 2018 30;9:1517. Epub 2018 Oct 30.

Department of Medicine, The Warren Alpert Medical School of Brown University, Providence, RI, United States.

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October 2018

NCX-Mediated Subcellular Ca Dynamics Underlying Early Afterdepolarizations in LQT2 Cardiomyocytes.

Biophys J 2018 Sep 9;115(6):1019-1032. Epub 2018 Aug 9.

Physics Department and Center for Interdisciplinary Research in Complex Systems, Northeastern University, Boston, Massachusetts. Electronic address:

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September 2018

Transient Outward K Current (I) Underlies the Right Ventricular Initiation of Polymorphic Ventricular Tachycardia in a Transgenic Rabbit Model of Long-QT Syndrome Type 1.

Circ Arrhythm Electrophysiol 2018 Jun;11(6):e005414

Cardiovascular Research Center, Division of Cardiology, Rhode Island Hospital, Warren Alpert Medical School of Brown University, Providence (B.-R.C., W.L., D.T., A.Y.K., R.T., T.Y.K., G.K.).

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June 2018

Long-Term Exposure to Imatinib Mesylate Downregulates Hippo Pathway and Activates YAP in a Model of Chronic Myelogenous Leukemia.

Stem Cells Dev 2017 05 27;26(9):656-677. Epub 2017 Feb 27.

1 Signal Transduction Laboratory, Division of Hematology/Oncology, Department of Medicine, Rhode Island Hospital, Warren Alpert Medical School, Brown University , Providence, Rhode Island.

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May 2017

SK channel enhancers attenuate Ca2+-dependent arrhythmia in hypertrophic hearts by regulating mito-ROS-dependent oxidation and activity of RyR.

Cardiovasc Res 2017 03;113(3):343-353

Department of Medicine, Division of Cardiology, Cardiovascular Research Center, Rhode Island Hospital, The Warren Alpert Medical School of Brown University, 1 Hoppin Street, Providence, RI, 02903-4141, USA.

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March 2017

Regulation of sarcoplasmic reticulum Ca release by serine-threonine phosphatases in the heart.

J Mol Cell Cardiol 2016 Dec 29;101:156-164. Epub 2016 Aug 29.

Cardiff University, School of Medicine, Wales Heart Research Institute, United Kingdom.

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December 2016

Ca(2+)-activated K(+) channels as therapeutic targets for myocardial and vascular protection.

Circ J 2015 30;79(3):455-62. Epub 2015 Jan 30.

Department of Surgery, Cardiovascular Research Center, Cardiovascular Institute, Rhode Island Hospital and Alpert Medical School of Brown University.

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November 2015

BKCa channel activation increases cardiac contractile recovery following hypothermic ischemia/reperfusion.

Am J Physiol Heart Circ Physiol 2015 Aug 12;309(4):H625-33. Epub 2015 Jun 12.

Cardiovascular Research Center and Department of Surgery, Rhode Island Hospital and Alpert Medical School of Brown University, Providence, Rhode Island

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August 2015

Dietary omega-3 fatty acids promote arrhythmogenic remodeling of cellular Ca2+ handling in a postinfarction model of sudden cardiac death.

PLoS One 2013 18;8(10):e78414. Epub 2013 Oct 18.

Dorothy M. Davis Heart and Lung Research Institute, the Ohio State University, Columbus, Ohio, United States of America ; Department of Physiology and Cell Biology, College of Medicine, the Ohio State University, Columbus, Ohio, United States of America.

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February 2015

Progesterone modulates SERCA2a expression and function in rabbit cardiomyocytes.

Am J Physiol Cell Physiol 2014 Dec 24;307(11):C1050-7. Epub 2014 Sep 24.

Cardiovascular Research Center, Division of Cardiology, Department of Medicine, Rhode Island Hospital, Alpert Medical School of Brown University, Providence, Rhode Island

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December 2014

Hyperphosphorylation of RyRs underlies triggered activity in transgenic rabbit model of LQT2 syndrome.

Circ Res 2014 Nov 23;115(11):919-28. Epub 2014 Sep 23.

From the Department of Medicine, Division of Cardiology, Cardiovascular Research Center, Rhode Island Hospital, Alpert Medical School of Brown University, Providence (D.T., W.L., L.L.C., H.K.J., Y.L., R.T., J.D., K.B., B.-R.C., G.K.); Physics Department, Center for Interdisciplinary Research on Complex Systems, Northeastern University, Boston, MA (C.M.R., A.K.); Department of Comparative Medicine, Pennsylvania State University College of Medicine, Hershey (X.P.); and Department of Cardiology and Angiology I, Heart Center Freiburg University, Freiburg, Germany (K.E.O.).

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November 2014

Ca handling during excitation-contraction coupling in heart failure.

Pflugers Arch 2014 Jun 11;466(6):1129-37. Epub 2014 Feb 11.

Department of Cell and Molecular Physiology, Loyola University Chicago, Stritch School of Medicine, 2160 South First Avenue, Maywood, IL, 60153, USA,

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June 2014

Sarcoplasmic reticulum Ca²⁺ release is both necessary and sufficient for SK channel activation in ventricular myocytes.

Am J Physiol Heart Circ Physiol 2014 Mar 31;306(5):H738-46. Epub 2013 Dec 31.

Cardiovascular Research Center, Division of Cardiology, Rhode Island Hospital, Alpert Medical School of Brown University, Providence, Rhode Island.

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March 2014

Redox modification of ryanodine receptors by mitochondria-derived reactive oxygen species contributes to aberrant Ca2+ handling in ageing rabbit hearts.

J Physiol 2013 Dec 16;591(23):5895-911. Epub 2013 Sep 16.

D. Terentyev: Cardiovascular Research Center, Rhode Island Hospital, Brown University School of Medicine, 1 Hoppin Street, West Coro-5, Providence, RI 02903, USA.

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December 2013

Solving the puzzle of <100> interstitial loop formation in bcc Iron.

Phys Rev Lett 2013 Jun 26;110(26):265503. Epub 2013 Jun 26.

Materials Science and Technology Division, Oak Ridge National Laboratory, Oak Ridge, Tennessee 37831-6138, USA.

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June 2013

Interaction of dislocations with carbon-decorated dislocation loops in bcc Fe: an atomistic study.

J Phys Condens Matter 2012 Nov 19;24(45):455402. Epub 2012 Oct 19.

SCK⋅CEN, Nuclear Material Science Institute, Boeretang 200, B-2400 Mol, Belgium.

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November 2012

Shortened Ca2+ signaling refractoriness underlies cellular arrhythmogenesis in a postinfarction model of sudden cardiac death.

Circ Res 2012 Feb 5;110(4):569-77. Epub 2012 Jan 5.

Davis Heart and Lung Research Institute, The Ohio State University Medical Center, 473 W 12th Ave., Columbus, OH 43210, USA..

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February 2012

How to stop the fire? Control of Ca²⁺-induced Ca²⁺ release in cardiac muscle.

J Physiol 2011 Dec;589(Pt 24):5899-900

Department of Medicine, The Warren Alpert Medical School of Brown University and Rhode Island Hospital, Providence, RI 02903, USA.

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December 2011

Arrhythmogenic adverse effects of cardiac glycosides are mediated by redox modification of ryanodine receptors.

J Physiol 2011 Oct 1;589(Pt 19):4697-708. Epub 2011 Aug 1.

Davis Heart and Lung Research Institute, The Ohio State University Medical Centre, 473 W. 12th Avenue, Columbus, OH 43210, USA.

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October 2011

MicroRNAs in cardiovascular disease.

F1000 Med Rep 2011 3;3:10. Epub 2011 May 3.

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July 2011

The relationship between arrhythmogenesis and impaired contractility in heart failure: role of altered ryanodine receptor function.

Cardiovasc Res 2011 Jun 27;90(3):493-502. Epub 2011 Jan 27.

Department of Physiology and Cell Biology, Davis Heart and Lung Research Institute, College of Medicine, The Ohio State University, Medical Center, 473 W. 12th Avenue Columbus, OH 43210, USA.

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June 2011

miRNAs got rhythm.

Life Sci 2011 Feb 3;88(9-10):373-83. Epub 2010 Dec 3.

Davis Heart and Lung Research Institute, The Ohio State University, Columbus, OH 43210, USA.

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February 2011

Redox modification of ryanodine receptors underlies calcium alternans in a canine model of sudden cardiac death.

Cardiovasc Res 2009 Dec 17;84(3):387-95. Epub 2009 Jul 17.

Department of Physiology and Cell Biology, Davis Heart and Lung Research Institute, The Ohio State University, College of Medicine, Columbus, OH 43210, USA.

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December 2009

Chronic heart failure and the substrate for atrial fibrillation.

Cardiovasc Res 2009 Nov 30;84(2):227-36. Epub 2009 Jun 30.

Davis Heart and Lung Research Institute, The Ohio State University, Columbus, OH 43210, USA.

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November 2009

Intra-sarcoplasmic reticulum Ca2+ oscillations are driven by dynamic regulation of ryanodine receptor function by luminal Ca2+ in cardiomyocytes.

J Physiol 2009 Oct 24;587(Pt 20):4863-72. Epub 2009 Aug 24.

Department of Physiology and Cell Biology, Ohio State University, Columbus, OH 43214, USA.

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October 2009

Cardiac calsequestrin: quest inside the SR.

J Physiol 2009 Jul;587(Pt 13):3091-4

Department of Physiology and Cell Biology, The Ohio State University, Columbus, USA.

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July 2009

Repolarization abnormalities and afterdepolarizations in a canine model of sudden cardiac death.

Am J Physiol Regul Integr Comp Physiol 2008 Nov 3;295(5):R1463-72. Epub 2008 Sep 3.

Davis Heart and Lung Research Institute, Ohio State University, Columbus, Ohio 43210, USA.

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November 2008

Modulation of SR Ca release by luminal Ca and calsequestrin in cardiac myocytes: effects of CASQ2 mutations linked to sudden cardiac death.

Biophys J 2008 Aug 9;95(4):2037-48. Epub 2008 May 9.

Department of Physiology, Dorothy M. Davis Heart and Lung Research Institute, The Ohio State University Medical Center, Columbus, Ohio 43210-1252, USA.

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August 2008

Modulation of ryanodine receptor by luminal calcium and accessory proteins in health and cardiac disease.

Cardiovasc Res 2008 Jan 15;77(2):245-55. Epub 2007 Oct 15.

Department of Physiology and Cell Biology, 505 Davis Heart and Lung Research Institute, The Ohio State University, 473 West 12th Avenue, Columbus, OH 43210, USA.

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January 2008

Enhanced ryanodine receptor-mediated calcium leak determines reduced sarcoplasmic reticulum calcium content in chronic canine heart failure.

Biophys J 2007 Dec 7;93(11):4083-92. Epub 2007 Sep 7.

Davis Heart and Lung Research Institute, Department of Physiology and Cell Biology, Ohio State University Medical Center, Columbus, Ohio 43210, USA.

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December 2007

Mechanisms of impaired calcium handling underlying subclinical diastolic dysfunction in diabetes.

Am J Physiol Regul Integr Comp Physiol 2007 Nov 29;293(5):R1787-97. Epub 2007 Aug 29.

College of Pharmacy, The Ohio State University, Columbus, OH 43210, USA.

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November 2007

Protein protein interactions between triadin and calsequestrin are involved in modulation of sarcoplasmic reticulum calcium release in cardiac myocytes.

J Physiol 2007 Aug 14;583(Pt 1):71-80. Epub 2007 Jun 14.

Department of Physiology and Cell Biology, 505 Davis Heart and Lung Research Institute, The Ohio State University, 473 W 12th Ave, Columbus, OH 43210, USA.

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August 2007

Chain-reaction Ca(2+) signaling in the heart.

J Clin Invest 2007 Jul;117(7):1758-62

Department of Physiology and Cell Biology and OSU Dorothy M Davis Heart and Lung Research Institute, The Ohio State University, Columbus, OH, USA.

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July 2007

Modulation of sarcoplasmic reticulum calcium release by calsequestrin in cardiac myocytes.

Biol Res 2004 ;37(4):603-7

Department of Physiology, Texas Tech University Health Sciences Center, Lubbock, Texas 79430, USA.

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October 2005

Synergistic interactions between Ca2+ entries through L-type Ca2+ channels and Na+-Ca2+ exchanger in normal and failing rat heart.

J Physiol 2005 Sep 23;567(Pt 2):493-504. Epub 2005 Jun 23.

Department of Physiology and Cell Biology, Davis Heart and Lung Research Institute, Ohio State University Medical Center, 473 West 12th Avenue, Columbus, OH 43210-1252, USA.

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September 2005

Abnormal intrastore calcium signaling in chronic heart failure.

Proc Natl Acad Sci U S A 2005 Sep 19;102(39):14104-9. Epub 2005 Sep 19.

Department of Physiology and Cell Biology, Davis Heart and Lung Research Institute, Ohio State University, Columbus, OH 43210, USA.

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September 2005

Triadin overexpression stimulates excitation-contraction coupling and increases predisposition to cellular arrhythmia in cardiac myocytes.

Circ Res 2005 Apr 24;96(6):651-8. Epub 2005 Feb 24.

Department of Physiology and Cell Biology, Heart and Lung Research Institute, Ohio State University, Columbus, Ohio 43210, USA.

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April 2005

Abnormal calcium signaling and sudden cardiac death associated with mutation of calsequestrin.

Circ Res 2004 Mar 8;94(4):471-7. Epub 2004 Jan 8.

Department of Physiology, Texas Tech University Health Sciences Center, Lubbock, Tex 79430-6551, USA.

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March 2004

Protein phosphatases decrease sarcoplasmic reticulum calcium content by stimulating calcium release in cardiac myocytes.

J Physiol 2003 Oct 1;552(Pt 1):109-18. Epub 2003 Aug 1.

Texas Tech University Health Sciences Center, 3601 4th Street, Lubbock, TX 79430-6551, USA.

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October 2003

Calsequestrin determines the functional size and stability of cardiac intracellular calcium stores: Mechanism for hereditary arrhythmia.

Proc Natl Acad Sci U S A 2003 Sep 16;100(20):11759-64. Epub 2003 Sep 16.

Department of Physiology and Cell Biology, Texas Tech University Health Sciences Center, Lubbock, TX 79430-6551, USA.

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September 2003