Diana C Haines

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Diana C Haines

Publications by authors named "Diana C Haines"

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40Publications

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RB inactivation in keratin 18 positive thymic epithelial cells promotes non-cell autonomous T cell hyperproliferation in genetically engineered mice.

PLoS One 2017 3;12(2):e0171510. Epub 2017 Feb 3.

Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute, Frederick, Maryland, United States of America.

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August 2017

Interaction with PALB2 Is Essential for Maintenance of Genomic Integrity by BRCA2.

PLoS Genet 2016 Aug 4;12(8):e1006236. Epub 2016 Aug 4.

Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute, Frederick, Maryland, United States of America.

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August 2016

Loss of oncogenic miR-155 in tumor cells promotes tumor growth by enhancing C/EBP-β-mediated MDSC infiltration.

Oncotarget 2016 Mar;7(10):11094-112

Department of Biomedical Sciences, Department of Physiology, University of Ulsan School of Medicine, Seoul, South Korea.

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March 2016

C/EBPγ Is a Critical Regulator of Cellular Stress Response Networks through Heterodimerization with ATF4.

Mol Cell Biol 2015 Dec 14;36(5):693-713. Epub 2015 Dec 14.

Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute, Frederick, Maryland, USA

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December 2015

Folliculin-interacting proteins Fnip1 and Fnip2 play critical roles in kidney tumor suppression in cooperation with Flcn.

Proc Natl Acad Sci U S A 2015 Mar 16;112(13):E1624-31. Epub 2015 Mar 16.

Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892; Basic Science Program, Leidos Biomedical Research, Inc., Frederick National Laboratory for Cancer Research, Frederick, MD 21702

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March 2015

Folliculin (Flcn) inactivation leads to murine cardiac hypertrophy through mTORC1 deregulation.

Hum Mol Genet 2014 Nov 6;23(21):5706-19. Epub 2014 Jun 6.

Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA,

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November 2014

Carcinoma initiation via RB tumor suppressor inactivation: a versatile approach to epithelial subtype-dependent cancer initiation in diverse tissues.

PLoS One 2013 2;8(12):e80459. Epub 2013 Dec 2.

Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute, Frederick, Maryland, United States of America.

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August 2014

COX-2 inhibition potentiates antiangiogenic cancer therapy and prevents metastasis in preclinical models.

Sci Transl Med 2014 Jun;6(242):242ra84

Tumor Angiogenesis Section, Mouse Cancer Genetics Program (MCGP), National Cancer Institute (NCI) at Frederick, National Institutes of Health, Frederick, MD 21702, USA.

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June 2014

Longitudinal imaging of cancer cell metastases in two preclinical models: a correlation of noninvasive imaging to histopathology.

Int J Mol Imaging 2014 3;2014:102702. Epub 2014 Mar 3.

Nanotechnology Characterization Laboratory, Cancer Research Technology Program, Leidos Biomedical Research, Inc., Frederick National Laboratory for Cancer Research, Frederick, MD 21702, USA.

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April 2014

Essential regulation of lung surfactant homeostasis by the orphan G protein-coupled receptor GPR116.

Cell Rep 2013 May 16;3(5):1457-64. Epub 2013 May 16.

Tumor Angiogenesis Section, Mouse Cancer Genetics Program (MCGP), Center for Cancer Research (CCR), National Cancer Institute (NCI), Frederick, MD 21702, USA.

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May 2013

Activation of TLR4 is required for the synergistic induction of dual oxidase 2 and dual oxidase A2 by IFN-γ and lipopolysaccharide in human pancreatic cancer cell lines.

J Immunol 2013 Feb 7;190(4):1859-72. Epub 2013 Jan 7.

Laboratory of Molecular Pharmacology, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.

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February 2013

The counter regulatory response induced by CpG oligonucleotides prevents bleomycin induced pneumopathy.

Respir Res 2012 Jun 18;13:47. Epub 2012 Jun 18.

Cancer and Inflammation Program, National Cancer Institute, Frederick, MD 21702, USA.

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June 2012

TEM8/ANTXR1 blockade inhibits pathological angiogenesis and potentiates tumoricidal responses against multiple cancer types.

Cancer Cell 2012 Feb;21(2):212-26

Tumor Angiogenesis Section, Mouse Cancer Genetics Program, National Cancer Institute (NCI), National Institutes of Health (NIH), Frederick, MD 21702, USA.

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February 2012

Tumor suppressor BRCA1 epigenetically controls oncogenic microRNA-155.

Nat Med 2011 Sep 25;17(10):1275-82. Epub 2011 Sep 25.

Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute at Frederick, Frederick, Maryland, USA.

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September 2011

Homozygous loss of BHD causes early embryonic lethality and kidney tumor development with activation of mTORC1 and mTORC2.

Proc Natl Acad Sci U S A 2009 Nov 22;106(44):18722-7. Epub 2009 Oct 22.

Urologic Oncology Branch, Center for Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD 20892, USA.

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November 2009

Host-derived tumor endothelial marker 8 promotes the growth of melanoma.

Cancer Res 2009 Aug 21;69(15):6021-6. Epub 2009 Jul 21.

Tumor Angiogenesis Section, Mouse Cancer Genetics Program, Science Applications International Corporation, National Cancer Institute-Frederick, Frederick, Maryland 21702-1201, USA.

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August 2009

Loss of Rad51c leads to embryonic lethality and modulation of Trp53-dependent tumorigenesis in mice.

Cancer Res 2009 Feb 20;69(3):863-72. Epub 2009 Jan 20.

Mouse Cancer Genetics Program, Center for Cancer Research, Science Applications International Corporation-Frederick, National Cancer Institute at Frederick, Frederick, Maryland 21702, USA.

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February 2009

Degradation of BRCA2 in alkyltransferase-mediated DNA repair and its clinical implications.

Cancer Res 2008 Dec;68(23):9973-81

Mouse Cancer Genetics Program, Center for Cancer Research, and Pathology Histotechnology Laboratory, Science Applications International Corporation-Frederick, Inc., National Cancer Institute at Frederick, Frederick, Maryland 21702, USA.

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December 2008

Itch genetically interacts with Notch1 in a mouse autoimmune disease model.

Hum Mol Genet 2006 Dec 9;15(24):3485-97. Epub 2006 Nov 9.

Mouse Cancer Genetics Program, Center for Cancer Research, National Cancer Institute, Frederick, MD 21702, USA.

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December 2006

Increased tumorigenicity and sensitivity to ionizing radiation upon loss of chromosomal protein HMGN1.

Cancer Res 2005 Aug;65(15):6711-8

Protein Section, Laboratory of Metabolism, Center for Cancer Research, National Cancer Institute, NIH, Bethesda, Maryland 20892, USA.

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August 2005

Calorie restriction and diet composition modulate spontaneous intestinal tumorigenesis in Apc(Min) mice through different mechanisms.

Cancer Res 2003 Apr;63(8):1752-5

Cancer Prevention Fellowship Program, Division of Cancer Prevention, National Cancer Institute, Bethesda, Maryland 20892-7105, USA.

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April 2003

Bethesda proposals for classification of lymphoid neoplasms in mice.

Blood 2002 Jul;100(1):246-58

Laboratory of Immunopathology, National Institute of Allergy and Infectious Diseases, National Institutes of Health, Bethesda, MD 20892-0760, USA.

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July 2002

Adult-onset calorie restriction and fasting delay spontaneous tumorigenesis in p53-deficient mice.

Carcinogenesis 2002 May;23(5):817-22

Division of Cancer Prevention, National Cancer Institute, Bethesda, MD 20892-7105, USA.

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May 2002