Debdeep Banerjee

Dr. Debdeep Banerjee

MD

University of Florida

Resident Physician

Gainesville, FL | United States

Specialties: Internal Medicine

Debdeep Banerjee

Dr. Debdeep Banerjee

MD
Introduction

Rising PGY-2 at University of Florida Internal Medicine. Aspiring gastroenterology/hepatology fellow. Strong passion for gastrointestinal oncology and the diagnostic & therapeutic utility of endoscopy therein.

Primary Affiliation: University of Florida - Gainesville, FL , United States

Specialties:

Research Interests:

Metrics

10

Publications

638

Profile Views

10

Reads

3

PubMed Central Citations

Education
Jul 2020
University of Florida
PGY
May 2017
Texas Tech University Health Sciences Center
MD
May 2013
Washington University in St. Louis
AB
Experience
Sep 2016
Baylor Scott and White
Visiting Student
Aug 2016
University of Arkansas for Medical Sciences
Visiting Student
Top co-authors
Benjamin Tharian
Benjamin Tharian

Ulster Hospital

3
James C Wang
James C Wang

Harvard University

2
Saikiran Raghavapuram
Saikiran Raghavapuram

University of Arkansas for Medical Sciences

2
Matthew Myntti
Matthew Myntti

Centre Hospitalier de l'Université de Montréal and McGill University Health Center

1
Joehassin Cordero
Joehassin Cordero

Texas Tech University Health Sciences Center

1
Joshua C Demke
Joshua C Demke

School of Medicine

1
Abdul N Hamood
Abdul N Hamood

Texas Tech University Health Sciences Center

1
Faysal Fedda
Faysal Fedda

University of Arkansas for Medical Sciences

1

Publications

10Publications

10Reads

3PubMed Central Citations

Endoscopic Resection of Esophageal Mucosal Bridge.

Gastroenterology 2018 Jun 9;154(8):2033-2034. Epub 2017 Sep 9.

Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, Arkansas.

View Article
June 2018
1 PubMed Central Citation(source)
16.72 Impact Factor

Metastatic duodenal germ cell tumor diagnosed with endoscopic ultrasound.

Endoscopy 2018 Jan 10;50(1):E27-E28. Epub 2017 Nov 10.

Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, Arkansas, United States.

View Article
January 2018
5.05 Impact Factor

Ectopic Hepatocellular Carcinoma within a Choledochal Cyst Diagnosed Using Single-Operator Digital Cholangioscopy.

Am J Gastroenterol 2017 Aug;112(8):1347-1348

Division of Gastroenterology and Hepatology, Department of Internal Medicine, University of Arkansas for Medical Sciences, Little Rock, Arkansas, USA.

View Article
August 2017
2 Reads
1 PubMed Central Citation(source)
10.76 Impact Factor

The antimicrobial agent, Next-Science, inhibits the development of Staphylococcus aureus and Pseudomonas aeruginosa biofilms on tympanostomy tubes.

Int J Pediatr Otorhinolaryngol 2015 Nov 7;79(11):1909-14. Epub 2015 Sep 7.

Department of Immunology and Molecular Microbiology, Texas Tech University Health Sciences Center, Lubbock, TX, USA; Department of Surgery/Division of Otolaryngology, Texas Tech University Health Sciences Center, Lubbock, TX, USA. Electronic address:

View Article
November 2015
7 Reads
1.32 Impact Factor

Novel use of tissue expander for dilation of oropharyngeal stenosis.

Int J Pediatr Otorhinolaryngol 2014 Nov 8;78(11):2018-20. Epub 2014 Sep 8.

Facial Plastic and Reconstructive Surgery, Division of Otolaryngology - Head and Neck Surgery, Department of Surgery, Texas Tech University Health Sciences Center, Lubbock, TX, United States; West Texas Craniofacial Center of Excellence, Lubbock, TX, United States. Electronic address:

View Article
November 2014
1 Read
1 PubMed Central Citation(source)
1.32 Impact Factor

Adipose tissue dysfunction in a model of developmental programming of metabolic syndrome

DOI: http://dx.doi.org/10.1016/j.ajog.2011.10.210

American Journal of Obstetrics & Gynecology Supplement

Obesity is characterized by adipose tissue accumulation, which in turn is associated with metabolic consequences. Distribution, type, and function of adipose tissue accumulation are believed to be more important than overall change in body or fat mass. The objective of this study was to investigate whether visceral adipose tissue dysfunction is present in a previously validated mouse model of developmental programming of metabolic syndrome.

View Article
January 2012
18 Reads

The role of cell signaling mechanisms in the downregulation of antioxidant enzymes in the fetal brain in a mouse model of fetal alcohol syndrome

DOI: http://dx.doi.org/10.1016/j.ajog.2011.10.069

American Journal of Obstetrics & Gynecology Supplement

Fetal alcohol syndrome (FAS) is the most common non genetic cause of mental retardation. Prenatal alcohol exposure decreases antioxidant enzymes in the fetal brain in a mouse model of fetal alcohol syndrome. Exposure to alcohol is known to decrease expression of Glycogen Synthase Kinase-3β (GSK-3β) a serine threonine kinase that regulates glycogen synthesis through phosphorylation and inactivation of glycogen synthase. In addition to regulation of glycogen, GSK-3β is positively correlated with expression of nuclear transcription factors, NF-κβ and Nrf2, which are inactive in the cytosol and active within nucleus.

View Article
January 2012
9 Reads

Protein expression and enzymatic activity of superoxide dismutase in the fetal brain in a mouse model of fetal alcohol syndrome

DOI: http://dx.doi.org/10.1016/j.ajog.2011.10.449

American Journal of Obstetrics & Gynecology Supplement

Fetal alcohol syndrome (FAS) is the most common non genetic cause of mental retardation. Prenatal alcohol exposure decreases the fetal brain mRNA expression of the antioxidant enzyme, superoxide dismutase (SOD). Our objective was to elucidate the role of oxidant/antioxidant mechanisms in the etiology of FAS by determining whether decrease in SOD mRNA expression is associated with reduced protein expression and/or enzymatic activity.

View Article
January 2012
10 Reads

Hypoxic and cellular differentiation mediators in offspring in a mouse model of prepregnancy obesity and preeclampsia

DOI: http://dx.doi.org/10.1016/j.ajog.2010.10.219

American Journal of Obstetrics & Gynecology Supplement

Our studies have shown that maternal prepregnancy obesity and exposure during pregnancy to soluble fms tyrosine-like kinase (sFlt1) exposure lead to development of metabolic syndrome in the offspring later in life. The aim of this study was to investigate the role of cellular differentiation (transforming growth factor-beta3, TGF-β3) and hypoxic-pathway mediators (hypoxia-inducible factor-1 alpha, HIF-1α) in this altered programming.

View Article
January 2011
10 Reads
Top co-authors
Benjamin Tharian
Benjamin Tharian

Ulster Hospital

3
James C Wang
James C Wang

Harvard University

2
Saikiran Raghavapuram
Saikiran Raghavapuram

University of Arkansas for Medical Sciences

2
Matthew Myntti
Matthew Myntti

Centre Hospitalier de l'Université de Montréal and McGill University Health Center

1
Joehassin Cordero
Joehassin Cordero

Texas Tech University Health Sciences Center

1
Joshua C Demke
Joshua C Demke

School of Medicine

1
Abdul N Hamood
Abdul N Hamood

Texas Tech University Health Sciences Center

1
Faysal Fedda
Faysal Fedda

University of Arkansas for Medical Sciences

1