Chenji Wang

Chenji Wang

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Chenji Wang

Chenji Wang

Publications by authors named "Chenji Wang"

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A novel androgen-reduced prostate-specific lncRNA, PSLNR, inhibits prostate-cancer progression in part by regulating the p53-dependent pathway.

Prostate 2019 Sep 3;79(12):1362-1377. Epub 2019 Jul 3.

Department of Urology, Shanghai Four People's Hospital Affiliated to Tongji University School of Medicine, Shanghai, PR China.

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http://dx.doi.org/10.1002/pros.23840DOI Listing
September 2019

HNRNPA1-mediated 3' UTR length changes of contributes to cancer- and senescence-associated phenotypes.

Aging (Albany NY) 2019 Jun;11(13):4407-4437

State Key Laboratory of Genetic Engineering, Collaborative Innovation Center of Genetics and Development, Human Phenome Institute, School of Life Sciences, Fudan University, Shanghai 200438, P.R. China.

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http://dx.doi.org/10.18632/aging.102060DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC6660030PMC
June 2019

A20-mediated deubiquitination of ERα in the microenvironment of CD163 macrophages sensitizes endometrial cancer cells to estrogen.

Cancer Lett 2019 02 27;442:137-147. Epub 2018 Oct 27.

Obstetrics and Gynecology Hospital of Fudan University, Shanghai, 200011, People's Republic of China; Shanghai Key Laboratory of Female Reproductive Endocrine Related Diseases, Fudan University, Shanghai, 200011, People's Republic of China. Electronic address:

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https://linkinghub.elsevier.com/retrieve/pii/S03043835183063
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http://dx.doi.org/10.1016/j.canlet.2018.10.019DOI Listing
February 2019

SPOP promotes ATF2 ubiquitination and degradation to suppress prostate cancer progression.

J Exp Clin Cancer Res 2018 Jul 11;37(1):145. Epub 2018 Jul 11.

State Key Laboratory of Genetic Engineering, Collaborative Innovation Center for Genetics and Development, School of Life Sciences, Fudan University, Shanghai, 200433, China.

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http://dx.doi.org/10.1186/s13046-018-0809-0DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC6042370PMC
July 2018

iASPP-PP1 complex is required for cytokinetic abscission by controlling CEP55 dephosphorylation.

Cell Death Dis 2018 05 1;9(5):528. Epub 2018 May 1.

State Key Laboratory of Genetic Engineering, School of Life Sciences, Institute of Biomedical Sciences, Fudan University, Shanghai, China.

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http://dx.doi.org/10.1038/s41419-018-0561-6DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5943338PMC
May 2018

MicroRNA-19a acts as a prognostic marker and promotes prostate cancer progression via inhibiting VPS37A expression.

Oncotarget 2018 Jan 6;9(2):1931-1943. Epub 2017 Dec 6.

Obstetrics and Gynecology Hospital, State Key Laboratory of Genetic Engineering, School of Life Science, Fudan University, Shanghai 200433, PR China.

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http://dx.doi.org/10.18632/oncotarget.23026DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5788610PMC
January 2018

ARRDC1 and ARRDC3 act as tumor suppressors in renal cell carcinoma by facilitating YAP1 degradation.

Am J Cancer Res 2018 1;8(1):132-143. Epub 2018 Jan 1.

Department of Urology, Shanghai General Hospital, School of Medicine, Shanghai Jiaotong UniversityShanghai, China.

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5794727PMC
January 2018

Ubiquitin-specific protease 11 (USP11) functions as a tumor suppressor through deubiquitinating and stabilizing VGLL4 protein.

Am J Cancer Res 2016 1;6(12):2901-2909. Epub 2016 Dec 1.

Department of Urology, Shanghai General Hospital, School of Medicine, Shanghai Jiaotong University Shanghai, P. R. China.

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5199763PMC
December 2016

High TXNDC5 expression predicts poor prognosis in renal cell carcinoma.

Tumour Biol 2016 Jul 26;37(7):9797-806. Epub 2016 Jan 26.

Department of Urology, Shanghai General Hospital, School of Medicine, Shanghai Jiaotong University, Shanghai, 200080, China.

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http://dx.doi.org/10.1007/s13277-016-4891-7DOI Listing
July 2016

TCP10L synergizes with MAD1 in transcriptional suppression and cell cycle arrest through mutual interaction.

BMB Rep 2016 Jun;49(6):325-30

State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Sciences, Fudan University, Shanghai 200433, P. R. China.

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC5070720PMC
http://dx.doi.org/10.5483/bmbrep.2016.49.6.248DOI Listing
June 2016

RNF12 promotes p53-dependent cell growth suppression and apoptosis by targeting MDM2 for destruction.

Cancer Lett 2016 May 27;375(1):133-141. Epub 2016 Feb 27.

State Key Laboratory of Genetic Engineering, Collaborative Innovation Center for Genetics and Development, School of Life Sciences, Fudan University, 2005 Songhu Road, Shanghai 200433, China; Shanghai Cancer Center, Institute of Biomedical Sciences, Shanghai Medical College of Fudan University, Shanghai 200032, China. Electronic address:

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http://dx.doi.org/10.1016/j.canlet.2016.02.013DOI Listing
May 2016

Molecular chaperone CCT3 supports proper mitotic progression and cell proliferation in hepatocellular carcinoma cells.

Cancer Lett 2016 Mar 29;372(1):101-9. Epub 2015 Dec 29.

State Key Laboratory of Genetic Engineering, Collaborative Innovation Center for Genetics and Development, School of Life Sciences, Fudan University, 2005 Songhu Road, Shanghai 200433, China. Electronic address:

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http://dx.doi.org/10.1016/j.canlet.2015.12.029DOI Listing
March 2016

Stabilization of MCRS1 by BAP1 prevents chromosome instability in renal cell carcinoma.

Cancer Lett 2015 Dec 20;369(1):167-74. Epub 2015 Aug 20.

Department of Urology, Shanghai General Hospital, School of Medicine, Shanghai Jiaotong University, 100 Haining Road, Shanghai 200080, China. Electronic address:

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http://dx.doi.org/10.1016/j.canlet.2015.08.013DOI Listing
December 2015

ASPP1/2-PP1 complexes are required for chromosome segregation and kinetochore-microtubule attachments.

Oncotarget 2015 Dec;6(39):41550-65

State Key Laboratory of Genetic Engineering, Collaborative Innovation Center for Genetics and Development, School of Life Sciences, Fudan University, Shanghai, P.R. China.

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http://dx.doi.org/10.18632/oncotarget.6355DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4747173PMC
December 2015

Tumor suppressor SPOP mediates the proteasomal degradation of progesterone receptors (PRs) in breast cancer cells.

Am J Cancer Res 2015 15;5(10):3210-20. Epub 2015 Sep 15.

State Key Laboratory of Genetic Engineering, Collaborative Innovation Center for Genetics and Development, School of Life Sciences, Fudan University Shanghai, P. R. China.

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http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4656742PMC
December 2015

Truncated ERG Oncoproteins from TMPRSS2-ERG Fusions Are Resistant to SPOP-Mediated Proteasome Degradation.

Mol Cell 2015 Sep 3;59(6):904-16. Epub 2015 Sep 3.

Department of Biochemistry and Molecular Biology, Mayo Clinic College of Medicine, Rochester, MN 55905, USA; Department of Urology, Mayo Clinic College of Medicine, Rochester, MN 55905, USA; Mayo Clinic Cancer Center, Mayo Clinic College of Medicine, Rochester, MN 55905, USA. Electronic address:

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http://www.pcf.org/atf/cf/%7B7c77d6a2-5859-4d60-af47-132fd0f
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http://linkinghub.elsevier.com/retrieve/pii/S109727651500583
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http://dx.doi.org/10.1016/j.molcel.2015.07.025DOI Listing
September 2015

PLK1 and β-TrCP-dependent ubiquitination and degradation of Rap1GAP controls cell proliferation.

PLoS One 2014 17;9(10):e110296. Epub 2014 Oct 17.

State Key Laboratory of Genetic Engineering, School of Life Sciences, Fudan University, Shanghai, P.R. China; Institutes of Biomedical Sciences, Fudan University, Shanghai, P.R. China.

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http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0110296PLOS
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4201484PMC
July 2015

Front-signal-dependent accumulation of the RHOA inhibitor FAM65B at leading edges polarizes neutrophils.

J Cell Sci 2015 Mar 14;128(5):992-1000. Epub 2015 Jan 14.

Vascular Biology and Therapeutic Program, Department of Pharmacology, Yale University School of Medicine, 10 Amistad Street, New Haven, CT 06410, USA

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http://dx.doi.org/10.1242/jcs.161497DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4342581PMC
March 2015

The E3 ubiquitin ligase Itch and Yap1 have antagonistic roles in the regulation of ASPP2 protein stability.

FEBS Lett 2015 Jan 29;589(1):94-101. Epub 2014 Nov 29.

State Key Laboratory of Genetic Engineering, School of Life Sciences, Fudan University, Shanghai 200433, PR China. Electronic address:

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http://dx.doi.org/10.1016/j.febslet.2014.11.030DOI Listing
January 2015

Destruction of DDIT3/CHOP protein by wild-type SPOP but not prostate cancer-associated mutants.

Hum Mutat 2014 Sep 23;35(9):1142-51. Epub 2014 Jul 23.

State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Sciences, Fudan University, Shanghai, 200433, P.R. China; Institutes of Biomedical Sciences, Fudan University, Shanghai, 200032, P. R. China.

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http://dx.doi.org/10.1002/humu.22614DOI Listing
September 2014

Destruction of full-length androgen receptor by wild-type SPOP, but not prostate-cancer-associated mutants.

Cell Rep 2014 Feb 6;6(4):657-69. Epub 2014 Feb 6.

Department of Biochemistry and Molecular Biology, Mayo Clinic College of Medicine, Rochester, MN 55905, USA; Department of Urology, Mayo Clinic College of Medicine, Rochester, MN 55905, USA; Mayo Clinic Cancer Center, Mayo Clinic College of Medicine, Rochester, MN 55905, USA. Electronic address:

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https://linkinghub.elsevier.com/retrieve/pii/S22111247140003
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http://dx.doi.org/10.1016/j.celrep.2014.01.013DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4361392PMC
February 2014

T-LAK cell-originated protein kinase is essential for the proliferation of hepatocellular carcinoma SMMC-7721 cells.

Cell Biochem Funct 2013 Dec 22;31(8):736-42. Epub 2013 Mar 22.

State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Science, Fudan University, Shanghai, 200433, China.

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http://dx.doi.org/10.1002/cbf.2964DOI Listing
December 2013

p53 Represses transcription of RING finger LIM domain-binding protein RLIM through Sp1.

PLoS One 2013 1;8(5):e62832. Epub 2013 May 1.

State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Sciences, Fudan University, Shanghai, PR China.

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http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0062832PLOS
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3641103PMC
November 2013

HDGF-related protein-3 is required for anchorage-independent survival and chemoresistance in hepatocellular carcinomas.

Gut 2013 Mar 5;62(3):440-51. Epub 2012 Apr 5.

The State Key Laboratory of Genetic Engineering, Fudan University, 220 Handan Road, Shanghai 200433, People's Republic of China.

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http://dx.doi.org/10.1136/gutjnl-2011-300781DOI Listing
March 2013

Polycomb group protein PHF1 regulates p53-dependent cell growth arrest and apoptosis.

J Biol Chem 2013 Jan 13;288(1):529-39. Epub 2012 Nov 13.

State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Sciences, Fudan University, 220 Handan Road, Shanghai 200433, China.

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http://www.jbc.org/lookup/doi/10.1074/jbc.M111.338996
Publisher Site
http://dx.doi.org/10.1074/jbc.M111.338996DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3537050PMC
January 2013

The Nedd4-like ubiquitin E3 ligases target angiomotin/p130 to ubiquitin-dependent degradation.

Biochem J 2012 Jun;444(2):279-89

State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Sciences, Fudan University, Shanghai, PR China.

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http://dx.doi.org/10.1042/BJ20111983DOI Listing
June 2012

SNIP1: a new activator of HSE signaling pathway.

Mol Cell Biochem 2012 Mar 22;362(1-2):1-6. Epub 2011 Oct 22.

State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Sciences, Fudan University, Shanghai, People's Republic of China.

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http://link.springer.com/10.1007/s11010-011-1120-y
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http://dx.doi.org/10.1007/s11010-011-1120-yDOI Listing
March 2012

E3 ubiquitin ligase Siah-1 facilitates poly-ubiquitylation and proteasomal degradation of the hepatitis B viral X protein.

FEBS Lett 2011 Oct 26;585(19):2943-50. Epub 2011 Aug 26.

State Key Laboratory of Genetic Engineering, Institute of Genetics, Shanghai, PR China.

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http://dx.doi.org/10.1016/j.febslet.2011.08.015DOI Listing
October 2011

RLIM interacts with Smurf2 and promotes TGF-β induced U2OS cell migration.

Biochem Biophys Res Commun 2011 Oct 17;414(1):181-5. Epub 2011 Sep 17.

State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Science, Fudan University, Shanghai 200433, China.

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http://dx.doi.org/10.1016/j.bbrc.2011.09.053DOI Listing
October 2011

The ubiquitin ligase itch regulates apoptosis by targeting thioredoxin-interacting protein for ubiquitin-dependent degradation.

J Biol Chem 2010 Mar 12;285(12):8869-79. Epub 2010 Jan 12.

State Key Laboratory of Genetic Engineering, Institute of Genetics, School of Life Sciences, Fudan University, Shanghai 200433, China.

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http://dx.doi.org/10.1074/jbc.M109.063321DOI Listing
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2838308PMC
March 2010

Evaluation of Cu in hepatocellular carcinoma by particle induced X-ray emission.

J Trace Elem Med Biol 2007 31;21(4):255-60. Epub 2007 Aug 31.

Institute of Modern Physic, Applied Ion Beam Physics Lab, Fudan University, 220 Handan Road, Shanghai 200433, PR China.

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http://dx.doi.org/10.1016/j.jtemb.2007.06.004DOI Listing
January 2008

[Molecular cloning and analysis of a monocot mannose-binding agglutinin from Zephyranthes grandiflora (family Amaryllidaceae)].

Sheng Wu Yi Xue Gong Cheng Xue Za Zhi 2004 Oct;21(5):812-8

College of Life Science, Sichuan University, Chengdu 610064, China.

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October 2004